Prostaglandin E1 does not accelerate rTPA-induced thrombolysis in acute myocardial infarction.
暂无分享,去创建一个
N. Kleiman | R. Tracy | R. Roberts | S. Harris | P. Puleo | R. Hill | L. J. Schaaff
[1] R. Califf,et al. Profound inhibition of platelet aggregation with monoclonal antibody 7E3 Fab after thrombolytic therapy. Results of the Thrombolysis and Angioplasty in Myocardial Infarction (TAMI) 8 Pilot Study. , 1993, Journal of the American College of Cardiology.
[2] Eric J. Topol,et al. A Randomized Trial of Late Reperfusion Therapy for Acute Myocardial Infarction , 1992, Circulation.
[3] G. FitzGerald,et al. Pharmacokinetics of Tissue‐Type Plasminogen Activator During Acute Myocardial Infarction in Men: Effect of a Prostacyclin Analogue , 1992, Circulation.
[4] W. Cawello,et al. On the metabolism of prostaglandin E1 administered intravenously to human volunteers. , 1991, Journal of physiology and pharmacology : an official journal of the Polish Physiological Society.
[5] J. Schneider. Taprostene, a stable prostacyclin analogue, enhances the thrombolytic efficacy of saruplase (recombinant single-chain urokinase-type plasminogen activator) in rabbits with pulmonary embolized thrombi. , 1991, Prostaglandins.
[6] R. Haslam,et al. Effects of Nitrovasodilators on Platelet Cyclic Nucleotide Levels in Rabbit Blood; Role for Cyclic AMP in Synergistic Inhibition of Platelet Function by SIN‐1 and Prostaglandin E1 , 1991, Journal of cardiovascular pharmacology.
[7] E. Braunwald,et al. Comparison of coronary and myocardial morphologic findings in patients with and without thrombolytic therapy during fatal first acute myocardial infarction. The TIMI Investigators. , 1990, The American journal of cardiology.
[8] J. Gore,et al. Comparison of immediate invasive, delayed invasive, and conservative strategies after tissue-type plasminogen activator. Results of the Thrombolysis in Myocardial Infarction (TIMI) Phase II-A trial. , 1990, Circulation.
[9] J. Vane,et al. Synergistic effect of prostaglandin E1 and isosorbide dinitrate in peripheral vascular disease , 1990, The Lancet.
[10] J. Mehta,et al. Prostacyclin analogue iloprost decreases thrombolytic potential of tissue-type plasminogen activator in canine coronary thrombosis. , 1990, Circulation.
[11] G. FitzGerald,et al. Platelet and vascular function during coronary thrombolysis with tissue-type plasminogen activator. , 1989, Circulation.
[12] K. Lee,et al. Combined tissue-type plasminogen activator and prostacyclin therapy for acute myocardial infarction. Thrombolysis and Angioplasty in Myocardial Infarction (TAMI) 4 Study Group. , 1989, Journal of the American College of Cardiology.
[13] M. Kroll,et al. Biochemical mechanisms of platelet activation. , 1989, Blood.
[14] G. FitzGerald,et al. Increased thromboxane biosynthesis during coronary thrombolysis. Evidence that platelet activation and thromboxane A2 modulate the response to tissue-type plasminogen activator in vivo. , 1989, Circulation research.
[15] W. Schumacher,et al. Effect of Thromboxane Antagonism on Recanalization During Streptokinase-Induced Thrombolysis in Anesthetized Monkeys , 1989, Journal of cardiovascular pharmacology.
[16] W. Terres,et al. Effects of aspirin and prostaglandin E1 on in vitro thrombolysis with urokinase. Evidence for a possible role of inhibiting platelet activity in thrombolysis. , 1989, Circulation.
[17] J. Loscalzo,et al. PGE1 accelerates thrombolysis by tissue plasminogen activator. , 1989, Blood.
[18] R. O'brien,et al. Simultaneous administration of thromboxane A2- and serotonin S2-receptor antagonists markedly enhances thrombolysis and prevents or delays reocclusion after tissue-type plasminogen activator in a canine model of coronary thrombosis. , 1989, Circulation.
[19] R. Feldman,et al. Hemodynamic and angiographic effects of prostaglandin E1 in coronary artery disease. , 1988, The American journal of cardiology.
[20] H. Sinzinger,et al. Comparable effect of prostaglandin E1 in decreasing in vivo platelet deposition on human lesion sites after intravenous and intraarterial application. , 1988, Thrombosis research.
[21] P. J. Simpson,et al. Reduction of experimental canine myocardial infarct size with prostaglandin E1: inhibition of neutrophil migration and activation. , 1988, The Journal of pharmacology and experimental therapeutics.
[22] Gundu H.R. Pao. Influence of anti-platelet drugs on platelet-vessel waif interacticnks , 1987 .
[23] R Roberts,et al. Thrombolysis in Myocardial Infarction (TIMI) Trial, Phase I: A comparison between intravenous tissue plasminogen activator and intravenous streptokinase. Clinical findings through hospital discharge. , 1987, Circulation.
[24] K. Swedberg,et al. Central haemodynamic and antiplatelet effects of iloprost--a new prostacyclin analogue--in acute myocardial infarction in man. , 1987, European heart journal.
[25] C. Fisher,et al. Comparison of equimolar concentrations of iloprost, prostacyclin, and prostaglandin E1 on human platelet function. , 1987, The Journal of laboratory and clinical medicine.
[26] Harold T. Dodge,et al. Thrombolysis inMyocardial Infarction (TIMI) Trial, Phase I:acomparison between intravenous tissue plasminogen activator andintravenous streptokinase* , 1987 .
[27] R. Wyeth,et al. Intracoronary prostaglandin E1 plus streptokinase in acute myocardial infarction. , 1986, The American journal of cardiology.
[28] D. Collen,et al. Assay of Human Tissue-Type Plasminogen Activator (t-PA) with an Enzyme-Linked Immunosorbent Assay (ELISA) Based on Three Murine Monoclonal Antibodies to t-PA , 1985, Thrombosis and Haemostasis.
[29] M J Davies,et al. Thrombosis and acute coronary-artery lesions in sudden cardiac ischemic death. , 1984, The New England journal of medicine.
[30] H. Pickles,et al. Epoprostenol (prostacyclin,PGI2) increases apparent liver blood flow in man. , 1983, Prostaglandins, leukotrienes, and medicine.
[31] J. Mustard,et al. Inhibitors of ADP-induced platelet aggregation prevent fibrinogen binding to rabbit platelets and cause rapid deaggregation and dissociation of bound fibrinogen. , 1981, The Journal of laboratory and clinical medicine.
[32] E. D. Jacobson,et al. Superior mesenteric angiography and blood flow measurement following intra-arterial injection of prostaglandin E1. , 1980, Radiology.
[33] J. Westwick. The effect of pulmonary metabolites of prostaglandins E1, E2 and F2alpha on ADP-induced aggregation of human and rabbit platelets [proceedings]. , 1976, British journal of pharmacology.
[34] R. Horton,et al. Metabolism of prostaglandins A1 and E1 in man. , 1975, The Journal of clinical investigation.
[35] E. Veys,et al. HL-A AND INFECTIVE SACROILEITIS , 1974 .
[36] J. Smith,et al. The influence on platelet aggregation of drugs that affect the accumulation of adenosine 3':5'-cyclic monophosphate in platelets. , 1971, The Biochemical journal.
[37] G. Ball,et al. Effect of prostaglandin E1 alone and in combination with theophylline or aspirin on collagen-induced platelet aggregation and on platelet nucleotides including adenosine 3′:5′-cyclic monophosphate , 1970 .
[38] A. Fletcher,et al. Enzymic lysis of plasma clots: The influence of fibrin stabilization on lysis rates , 1967 .
[39] A. J. Honour,et al. Effect of prostaglandin E1 on platelet behaviour in vitro and in vivo. , 1967, British medical journal.
[40] M. Friedman,et al. The pathogenesis of a coronary thrombus. , 1966, The American journal of pathology.
[41] K. Lindahl-Kiessling,et al. USE AND ABUSE OF ELECTROENCEPHALOGRAPHY. , 1964, Lancet.