Mechanistic study of endogenous skin lesions in diabetic rats

Please cite this paper as: Mechanistic study of endogenous skin lesions in diabetic rats. Experimental Dermatology 2010; 19: 1088–1095.

[1]  A. Harel,et al.  Sensitivity of HaCat keratinocytes to diabetogenic toxins. , 2002, Biochemical pharmacology.

[2]  V. Hwa,et al.  Connective tissue growth factor/IGF-binding protein-related protein-2 is a mediator in the induction of fibronectin by advanced glycosylation end-products in human dermal fibroblasts. , 2002, Endocrinology.

[3]  P. D. de Groot,et al.  Effect of Extracellular Matrix Glycation on Endothelial Cell Adhesion and Spreading: Involvement of Vitronectin , 1997, Diabetes.

[4]  B. Sadr,et al.  Biophysical characteristics of skin in diabetes: a controlled study , 2009, Journal of the European Academy of Dermatology and Venereology : JEADV.

[5]  A. Nègre-Salvayre,et al.  Advanced glycation end product precursors impair epidermal growth factor receptor signaling. , 2002, Diabetes.

[6]  Shuliang Lu,et al.  Effects of Extracellular Matrix Glycosylation on Proliferation and Apoptosis of Human Dermal Fibroblasts Via the Receptor for Advanced Glycosylated End Products , 2008, The American Journal of dermatopathology.

[7]  D. Accili,et al.  Glucose effects on skin keratinocytes: implications for diabetes skin complications. , 2001, Diabetes.

[8]  P. Blumberg,et al.  Protein kinase C isoenzymes differentially regulate the differentiation‐dependent expression of adhesion molecules in human epidermal keratinocytes , 2009, Experimental dermatology.

[9]  Su-Yen Goh,et al.  The role of advanced glycation end products in progression and complications of diabetes , 2008 .

[10]  A. Schmidt,et al.  RAGE is a multiligand receptor of the immunoglobulin superfamily: implications for homeostasis and chronic disease , 2002, Cellular and Molecular Life Sciences CMLS.

[11]  Danielle T. Loughlin,et al.  3‐Deoxyglucosone‐collagen alters human dermal fibroblast migration and adhesion: Implications for impaired wound healing in patients with diabetes , 2009, Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society.

[12]  D. Accili,et al.  The regulation of skin proliferation and differentiation in the IR null mouse: implications for skin complications of diabetes. , 2001, Endocrinology.

[13]  L. Segars,et al.  Assessing prescriptions for statins in ambulatory diabetic patients in the United States: a national, cross-sectional study. , 2008, Clinical therapeutics.

[14]  J. Zou,et al.  Insulin Prevents Latent Skin Lesions by Inhibiting the Generation of Advanced Glycation End Products in Streptozotocin-Induced Diabetic Rats , 2009, Endocrine pathology.

[15]  P. Xie,et al.  Diabetic Nephropathy: Mechanisms of Renal Disease Progression , 2008, Experimental biology and medicine.

[16]  R. Schwartz,et al.  Diabetic dermopathy: A subtle sign with grave implications. , 2008, Journal of the American Academy of Dermatology.

[17]  S. Shaikh,et al.  Advanced glycation end products induce in vitro cross‐linking of α‐synuclein and accelerate the process of intracellular inclusion body formation , 2008, Journal of neuroscience research.

[18]  J. Cook,et al.  Abnormal Connexin Expression Underlies Delayed Wound Healing in Diabetic Skin , 2007, Diabetes.

[19]  M. McCarthy,et al.  Polymorphisms in the Glucokinase-Associated, Dual-Specificity Phosphatase 12 (DUSP12) Gene Under Chromosome 1q21 Linkage Peak Are Associated With Type 2 Diabetes , 2006, Diabetes.

[20]  A. Kitabchi,et al.  Proinflammatory cytokines, markers of cardiovascular risks, oxidative stress, and lipid peroxidation in patients with hyperglycemic crises. , 2004, Diabetes.

[21]  E. Fuchs,et al.  Defining the Epithelial Stem Cell Niche in Skin , 2004, Science.

[22]  V. Monnier,et al.  Prevention and repair of protein damage by the Maillard reaction in vivo. , 2006, Rejuvenation research.

[23]  Z. Guo,et al.  Advanced oxidation protein products activate vascular endothelial cells via a RAGE-mediated signaling pathway. , 2008, Antioxidants & redox signaling.

[24]  Plantar skin in type II diabetes: an investigation of protein glycation and biomechanical properties of plantar epidermis. , 2006, European journal of dermatology : EJD.

[25]  E. Sage,et al.  SPARC, a matricellular protein that functions in cellular differentiation and tissue response to injury. , 2001, The Journal of clinical investigation.

[26]  S. Sampson,et al.  Characterization of glucose transport system in keratinocytes: insulin and IGF-1 differentially affect specific transporters. , 2000, The Journal of investigative dermatology.

[27]  V. D’Agati,et al.  Blockade of receptor for advanced glycation endproducts: a new target for therapeutic intervention in diabetic complications and inflammatory disorders. , 2003, Archives of biochemistry and biophysics.

[28]  S. G. Laychock,et al.  Efficient production of bioactive insulin from human epidermal keratinocytes and tissue-engineered skin substitutes: implications for treatment of diabetes. , 2007, Tissue engineering.

[29]  A J Bailey,et al.  The effects of the Maillard reaction on the physical properties and cell interactions of collagen. , 2006, Pathologie-biologie.

[30]  S. Bernard,et al.  Adipocyte Turnover: Relevance to Human Adipose Tissue Morphology , 2009, Diabetes.

[31]  A. Schmidt,et al.  Receptor for advanced glycation endproducts (RAGE) and the complications of diabetes , 2002, Ageing Research Reviews.

[32]  S. Yamagishi,et al.  Advanced glycation end products and insulin resistance. , 2008, Current pharmaceutical design.

[33]  T. Tennenbaum,et al.  Differential roles of insulin receptor and insulin-like growth factor-1 receptor in differentiation of murine skin keratinocytes. , 2000, The Journal of investigative dermatology.

[34]  S. G. Laychock,et al.  Regulated insulin delivery from human epidermal cells reverses hyperglycemia. , 2008, Molecular therapy : the journal of the American Society of Gene Therapy.

[35]  I. Maatouk,et al.  Microcystin-LR and nodularin induce intracellular glutathione alteration, reactive oxygen species production and lipid peroxidation in primary cultured rat hepatocytes. , 2004, Toxicology letters.

[36]  In Sup Kil,et al.  Glycation-induced inactivation of NADP(+)-dependent isocitrate dehydrogenase: implications for diabetes and aging. , 2004, Free radical biology & medicine.

[37]  A. Schmidt,et al.  Receptor for advanced glycation endproducts: a multiligand receptor magnifying cell stress in diverse pathologic settings. , 2002, Advanced drug delivery reviews.

[38]  H. Vlassara,et al.  Receptor-Mediated Interactions of Advanced Glycosylation End Products With Cellular Components Within Diabetic Tissues , 1992, Diabetes.

[39]  Dae Joong Kim,et al.  Thickness of Korean Upper Eyelid Skin at Different Levels , 2006, The Journal of craniofacial surgery.

[40]  J. Drukała,et al.  Effect of heme and heme oxygenase-1 on vascular endothelial growth factor synthesis and angiogenic potency of human keratinocytes. , 2006, Free radical biology & medicine.

[41]  G. Sajithlal,et al.  Effect of curcumin on the advanced glycation and cross-linking of collagen in diabetic rats. , 1998, Biochemical pharmacology.

[42]  Paul J Thornalley,et al.  Advanced Glycation End Products in Extracellular Matrix Proteins Contribute to the Failure of Sensory Nerve Regeneration in Diabetes , 2009, Diabetes.

[43]  S. E. James,et al.  Purinergic receptor expression in the regenerating epidermis in a rat model of normal and delayed wound healing , 2003, Experimental dermatology.

[44]  Weiliam Chen,et al.  Cells and tissue interactions with glycated collagen and their relevance to delayed diabetic wound healing. , 2009, Biomaterials.

[45]  R. Hill.  Extracellular matrix remodelling in human diabetic neuropathy , 2009, Journal of anatomy.

[46]  Y. Kaneda,et al.  Normalizing mitochondrial superoxide production blocks three pathways of hyperglycaemic damage , 2000, Nature.