From Ionic Currents to Molecular Mechanisms The Structure and Function of Voltage-Gated Sodium Channels
暂无分享,去创建一个
[1] M. Orfanopoulos,et al. Mechanism of the , 2000, The Journal of organic chemistry.
[2] J. Dixon,et al. A sodium channel signaling complex: modulation by associated receptor protein tyrosine phosphatase β , 2000, Nature Neuroscience.
[3] E. Isacoff,et al. Spectroscopic mapping of voltage sensor movement in the Shaker potassium channel , 1999, Nature.
[4] Francisco Bezanilla,et al. Atomic scale movement of the voltage-sensing region in a potassium channel measured via spectroscopy , 1999, Nature.
[5] John C. Rogers,et al. Functional Roles of the Extracellular Segments of the Sodium Channel α Subunit in Voltage-dependent Gating and Modulation by β1 Subunits* , 1999, The Journal of Biological Chemistry.
[6] William A. Catterall,et al. The Extracellular Domain of the β1 Subunit Is Both Necessary and Sufficient for β1-like Modulation of Sodium Channel Gating* , 1999, The Journal of Biological Chemistry.
[7] M. Baulac,et al. A second locus for familial generalized epilepsy with febrile seizures plus maps to chromosome 2q21-q33. , 1999, American journal of human genetics.
[8] M. Schachner,et al. Tenascin-R Is a Functional Modulator of Sodium Channel β Subunits* , 1999, The Journal of Biological Chemistry.
[9] W. Catterall,et al. Dopaminergic Modulation of Voltage-Gated Na+ Current in Rat Hippocampal Neurons Requires Anchoring of cAMP-Dependent Protein Kinase , 1999, The Journal of Neuroscience.
[10] D. Hanck,et al. The Na channel voltage sensor associated with inactivation is localized to the external charged residues of domain IV, S4. , 1999, Biophysical journal.
[11] W. Catterall,et al. Voltage-Dependent Neuromodulation of Na+ Channels by D1-Like Dopamine Receptors in Rat Hippocampal Neurons , 1999, The Journal of Neuroscience.
[12] F Elinder,et al. The screw–helical voltage gating of ion channels , 1999, Proceedings of the Royal Society of London. Series B: Biological Sciences.
[13] A. Goldin. Diversity of Mammalian Voltage‐Gated Sodium Channels , 1999, Annals of the New York Academy of Sciences.
[14] J. Balser,et al. Molecular Dynamics of the Sodium Channel Pore Vary with Gating: Interactions between P-Segment Motions and Inactivation , 1999, The Journal of Neuroscience.
[15] Francisco Bezanilla,et al. Voltage Sensors in Domains III and IV, but Not I and II, Are Immobilized by Na+ Channel Fast Inactivation , 1999, Neuron.
[16] C. Rohl,et al. Solution structure of the sodium channel inactivation gate. , 1999, Biochemistry.
[17] W. Catterall,et al. Interaction of voltage-gated sodium channels with the extracellular matrix molecules tenascin-C and tenascin-R. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[18] W. Catterall,et al. Voltage Sensor–Trapping Enhanced Activation of Sodium Channels by β-Scorpion Toxin Bound to the S3–S4 Loop in Domain II , 1998, Neuron.
[19] Samuel F. Berkovic,et al. Febrile seizures and generalized epilepsy associated with a mutation in the Na+-channel ß1 subunit gene SCN1B , 1998, Nature Genetics.
[20] Douglas C. Wallace,et al. Radicals r'aging , 1998, Nature Genetics.
[21] Robert L. Barchi,et al. Inactivation and Secondary Structure in the D4/S4-5 Region of the SkM1 Sodium Channel , 1998, The Journal of general physiology.
[22] N. Chehab,et al. Glutamine Substitution at Alanine1649 in the S4–S5 Cytoplasmic Loop of Domain 4 Removes the Voltage Sensitivity of Fast Inactivation in the Human Heart Sodium Channel , 1998, The Journal of general physiology.
[23] B. Chait,et al. The structure of the potassium channel: molecular basis of K+ conduction and selectivity. , 1998, Science.
[24] G. Breithardt,et al. Genetic basis and molecular mechanism for idiopathic ventricular fibrillation , 1998, Nature.
[25] W. Catterall,et al. Molecular Determinants of Na+ Channel Function in the Extracellular Domain of the β1 Subunit* , 1998, The Journal of Biological Chemistry.
[26] R. Vaillancourt,et al. Growth Factor Receptor Tyrosine Kinases Acutely Regulate Neuronal Sodium Channels through the Src Signaling Pathway , 1998, The Journal of Neuroscience.
[27] W. Catterall,et al. A Critical Role for the S4-S5 Intracellular Loop in Domain IV of the Sodium Channel α-Subunit in Fast Inactivation* , 1998, The Journal of Biological Chemistry.
[28] F. J. White,et al. Whole-Cell Plasticity in Cocaine Withdrawal: Reduced Sodium Currents in Nucleus Accumbens Neurons , 1998, The Journal of Neuroscience.
[29] H. Lerche,et al. Role in fast inactivation of the IV/S4–S5 loop of the human muscle Na+ channel probed by cysteine mutagenesis , 1997, The Journal of physiology.
[30] Edward Moczydlowski,et al. On the Structural Basis for Size-selective Permeation of Organic Cations through the Voltage-gated Sodium Channel , 1997, The Journal of general physiology.
[31] Francisco Bezanilla,et al. Voltage-Dependent Proton Transport by the Voltage Sensor of the Shaker K+ Channel , 1997, Neuron.
[32] A. L. Goldin,et al. Interaction between the sodium channel inactivation linker and domain III S4-S5. , 1997, Biophysical journal.
[33] A. L. Goldin,et al. Dopaminergic Modulation of Sodium Current in Hippocampal Neurons via cAMP-Dependent Phosphorylation of Specific Sites in the Sodium Channel α Subunit , 1997, The Journal of Neuroscience.
[34] A. L. Goldin,et al. Sodium Channel Activation Gating Is Affected by Substitutions of Voltage Sensor Positive Charges in All Four Domains , 1997, The Journal of general physiology.
[35] A. L. Goldin,et al. Phosphorylation at a Single Site in the Rat Brain Sodium Channel Is Necessary and Sufficient for Current Reduction by Protein Kinase A , 1997, The Journal of Neuroscience.
[36] S. Moss,et al. A single serine residue confers tetrodotoxin insensitivity on the rat sensory‐neuron‐specific sodium channel SNS , 1997, FEBS letters.
[37] William A. Catterall,et al. Molecular Analysis of the Putative Inactivation Particle in the Inactivation Gate of Brain Type IIA Na+ Channels , 1997, The Journal of general physiology.
[38] W. Catterall,et al. Molecular Analysis of Potential Hinge Residues in the Inactivation Gate of Brain Type IIA Na+ Channels , 1997, The Journal of general physiology.
[39] D. Papazian,et al. Electrostatic interactions between transmembrane segments mediate folding of Shaker K+ channel subunits. , 1997, Biophysical journal.
[40] R. Horn,et al. A unique role for the S4 segment of domain 4 in the inactivation of sodium channels , 1996, The Journal of general physiology.
[41] W. Catterall,et al. Movement of the Na+ Channel Inactivation Gate during Inactivation* , 1996, The Journal of Biological Chemistry.
[42] N. Makita,et al. Molecular Determinants of β1 Subunit-Induced Gating Modulation in Voltage-Dependent Na+ Channels , 1996, The Journal of Neuroscience.
[43] D. Wray,et al. Measurement of the movement of the S4 segment during the activation of a voltage-gated potassium channel , 1996, Pflügers Archiv.
[44] W. Hendrickson,et al. Crystal Structure of the Extracellular Domain from P0, the Major Structural Protein of Peripheral Nerve Myelin , 1996, Neuron.
[45] W. Catterall,et al. Molecular Determinants of High Affinity Binding of α-Scorpion Toxin and Sea Anemone Toxin in the S3-S4 Extracellular Loop in Domain IV of the Na+ Channel α Subunit* , 1996, The Journal of Biological Chemistry.
[46] William A Catterall,et al. Muscarinic Modulation of Sodium Current by Activation of Protein Kinase C in Rat Hippocampal Neurons , 1996, Neuron.
[47] G. Tomaselli,et al. Depth Asymmetries of the Pore-Lining Segments of the Na+ Channel Revealed by Cysteine Mutagenesis , 1996, Neuron.
[48] A. L. Goldin,et al. Phosphorylation of brain sodium channels in the I--II linker modulates channel function in Xenopus oocytes , 1996, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[49] P. Bjorkman,et al. The (Greek) Key to Structures of Neural Adhesion Molecules , 1996, Neuron.
[50] Ehud Y. Isacoff,et al. Transmembrane Movement of the Shaker K+ Channel S4 , 1996, Neuron.
[51] G. Tomaselli,et al. Structure of the sodium channel pore revealed by serial cysteine mutagenesis. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[52] R. Horn,et al. Molecular Basis of Charge Movement in Voltage-Gated Sodium Channels , 1996, Neuron.
[53] S. Cannon. Sodium channel defects in myotonia and periodic paralysis. , 1996, Annual review of neuroscience.
[54] S. Heinemann,et al. Pore properties of rat brain II sodium channels mutated in the selectivity filter domain , 1996, European Biophysics Journal.
[55] J. Patlak,et al. Transfer of twelve charges is needed to open skeletal muscle Na+ channels , 1995, The Journal of general physiology.
[56] W. Catterall,et al. Structure and function of the β2 subunit of brain sodium channels, a transmembrane glycoprotein with a CAM motif , 1995, Cell.
[57] A. George,et al. Molecular mechanism for an inherited cardiac arrhythmia , 1995, Nature.
[58] R. Horn,et al. Evidence for voltage-dependent S4 movement in sodium channels , 1995, Neuron.
[59] Yu Huang,et al. Electrostatic interactions of S4 voltage sensor in shaker K+ channel , 1995, Neuron.
[60] W. Catterall,et al. A Critical Role for Transmembrane Segment IVS6 of the Sodium Channel α Subunit in Fast Inactivation (*) , 1995, The Journal of Biological Chemistry.
[61] Arthur J Moss,et al. SCN5A mutations associated with an inherited cardiac arrhythmia, long QT syndrome , 1995, Cell.
[62] W. Catterall,et al. Molecular determinants of state-dependent block of Na+ channels by local anesthetics. , 1994, Science.
[63] W. Catterall,et al. Restoration of inactivation and block of open sodium channels by an inactivation gate peptide , 1994, Neuron.
[64] D. Marić,et al. [Paramyotonia congenita]. , 1994, Vojnosanitetski pregled.
[65] S. Rossie,et al. Identification of the sites of selective phosphorylation and dephosphorylation of the rat brain Na+ channel alpha subunit by cAMP-dependent protein kinase and phosphoprotein phosphatases. , 1993, The Journal of biological chemistry.
[66] Ming Li,et al. Convergent regulation of sodium channels by protein kinase C and cAMP-dependent protein kinase. , 1993, Science.
[67] Anonymous,et al. Review: , 2019 .
[68] A molecular basis for gating mode transitions in human skeletal muscle Na+ channels , 1993, FEBS letters.
[69] G. Yellen,et al. The internal quaternary ammonium receptor site of Shaker potassium channels , 1993, Neuron.
[70] D. Surmeier,et al. D1 and D2 dopamine receptor modulation of sodium and potassium currents in rat neostriatal neurons. , 1993, Progress in brain research.
[71] A L Goldin,et al. A cluster of hydrophobic amino acid residues required for fast Na(+)-channel inactivation. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[72] G. Tomaselli,et al. Molecular localization of an ion-binding site within the pore of mammalian sodium channels. , 1992, Science.
[73] Ming Li,et al. Functional modulation of brain sodium channels by cAMP-dependent phosphorylation , 1992, Neuron.
[74] R. Rogart,et al. A Mutant of TTX-Resistant Cardiac Sodium Channels with TTX-Sensitive Properties , 1992, Science.
[75] A L Goldin,et al. Primary structure and functional expression of the beta 1 subunit of the rat brain sodium channel. , 1992, Science.
[76] M. Leppert,et al. Mutations in an S4 segment of the adult skeletal muscle sodium channel cause paramyotonia congenita , 1992, Neuron.
[77] W. Stühmer,et al. Calcium channel characteristics conferred on the sodium channel by single mutations , 1992, Nature.
[78] J. Haines,et al. Temperature-sensitive mutations in the III–IV cytoplasmic loop region of the skeletal muscle sodium channel gene in paramyotonia congenita , 1992, Cell.
[79] B. Welch. The structure , 1992 .
[80] E. Hoffman,et al. A Met-to-Val mutation in the skeletal muscle Na+ channel α-subunit in hyperkalaemic periodic paralysis , 1991, Nature.
[81] Margaret Robertson,et al. Identification of a mutation in the gene causing hyperkalemic periodic paralysis , 1991, Cell.
[82] W. Catterall,et al. A phosphorylation site in the Na+ channel required for modulation by protein kinase C. , 1991, Science.
[83] F. Conti,et al. Mapping the site of block by tetrodotoxin and saxitoxin of sodium channel II , 1991, FEBS letters.
[84] W. Catterall,et al. Functional modulation of brain sodium channels by protein kinase C phosphorylation. , 1991, Science.
[85] C. Miller,et al. 1990: annus mirabilis of potassium channels , 1991, Science.
[86] W. Catterall,et al. Identification of a phenylalkylamine binding region within the alpha 1 subunit of skeletal muscle Ca2+ channels. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[87] N. Dascal,et al. Modulation of vertebrate brain Na+ and K+ channels by subtypes of protein kinase C , 1990, FEBS letters.
[88] W. Stühmer,et al. A single point mutation confers tetrodotoxin and saxitoxin insensitivity on the sodium channel II , 1989, FEBS letters.
[89] W. Catterall,et al. Inhibition of inactivation of single sodium channels by a site-directed antibody. , 1989, Proceedings of the National Academy of Sciences of the United States of America.
[90] F. Conti,et al. Structural parts involved in activation and inactivation of the sodium channel , 1989, Nature.
[91] Ina Ruck,et al. USA , 1969, The Lancet.
[92] P. Vassilev,et al. Identification of an intracellular peptide segment involved in sodium channel inactivation. , 1988, Science.
[93] E. Sigel,et al. Activation of protein kinase C differentially modulates neuronal Na+, Ca2+, and gamma-aminobutyrate type A channels. , 1988, Proceedings of the National Academy of Sciences of the United States of America.
[94] W. Catterall,et al. Cyclic-AMP-dependent phosphorylation of voltage-sensitive sodium channels in primary cultures of rat brain neurons. , 1987, The Journal of biological chemistry.
[95] P. Calabresi,et al. Intracellular studies on the dopamine-induced firing inhibition of neostriatal neurons in vitro: Evidence for D1 receptor involvement , 1987, Neuroscience.
[96] William A. Catterall,et al. Voltage-dependent gating of sodium channels: correlating structure and function , 1986, Trends in Neurosciences.
[97] A. L. Goldin,et al. Messenger RNA coding for only the alpha subunit of the rat brain Na channel is sufficient for expression of functional channels in Xenopus oocytes. , 1986, Proceedings of the National Academy of Sciences of the United States of America.
[98] H. Takeshima,et al. Existence of distinct sodium channel messenger RNAs in rat brain , 1986, Nature.
[99] H. Guy,et al. Molecular model of the action potential sodium channel. , 1986, Proceedings of the National Academy of Sciences of the United States of America.
[100] P. Mueller,et al. Voltage-dependent activation in purified reconstituted sodium channels from rabbit T-tubular membranes. , 1986, Proceedings of the National Academy of Sciences of the United States of America.
[101] H. Takeshima,et al. Expression of functional sodium channels from cloned cDNA , 1986, Nature.
[102] W. Catterall,et al. The sodium channel from rat brain , 1986 .
[103] J. Tanaka,et al. Purification and functional reconstitution of the voltage-sensitive sodium channel from rabbit T-tubular membranes. , 1985, The Journal of biological chemistry.
[104] W. Catterall,et al. Functional reconstitution of the purified brain sodium channel in planar lipid bilayers. , 1985, Proceedings of the National Academy of Sciences of the United States of America.
[105] B. Hille,et al. Ionic channels of excitable membranes , 2001 .
[106] Yuichi Kanaoka,et al. Primary structure of Electrophorus electricus sodium channel deduced from cDNA sequence , 1984, Nature.
[107] W. Catterall,et al. Cyclic AMP-dependent phosphorylation of the alpha subunit of the sodium channel in synaptic nerve ending particles. , 1984, The Journal of biological chemistry.
[108] W. Catterall,et al. Phosphorylation of the alpha subunit of the sodium channel by protein kinase C. , 1984, Cellular and molecular neurobiology.
[109] R. Barchi. Protein Components of the Purified Sodium Channel from Rat Skeletal Muscle Sarcolemma , 1983, Journal of neurochemistry.
[110] J. Miller,et al. Principal glycopeptide of the tetrodotoxin/saxitoxin binding protein from Electrophorus electricus: isolation and partial chemical and physical characterization. , 1983, Biochemistry.
[111] W. Catterall,et al. The saxitoxin receptor of the sodium channel from rat brain. Evidence for two nonidentical beta subunits. , 1982, The Journal of biological chemistry.
[112] W. Catterall,et al. Reconstitution of neurotoxin-stimulated sodium transport by the voltage-sensitive sodium channel purified from rat brain. , 1982, The Journal of biological chemistry.
[113] C. Armstrong,et al. Sodium channels and gating currents. , 1981, Physiological reviews.
[114] W. Catterall,et al. Purification of the saxitoxin receptor of the sodium channel from rat brain. , 1981, Proceedings of the National Academy of Sciences of the United States of America.
[115] S. Levinson,et al. Identification of a large molecular weight peptide associated with a tetrodotoxin binding protein from the electroplax of Electrophorus electricus. , 1980, Biochemical and biophysical research communications.
[116] W. Catterall. Neurotoxins that act on voltage-sensitive sodium channels in excitable membranes. , 1980, Annual review of pharmacology and toxicology.
[117] W. Catterall,et al. Covalent labeling of protein components of the sodium channel with a photoactivable derivative of scorpion toxin. , 1980, Proceedings of the National Academy of Sciences of the United States of America.
[118] J. M. Ritchie,et al. The binding of saxitoxin and tetrodotoxin to excitable tissue. , 1977, Reviews of physiology, biochemistry and pharmacology.
[119] A. Hodgkin,et al. A quantitative description of membrane current and its application to conduction and excitation in nerve , 1952, The Journal of physiology.