Intrathecal catheterization alone reduces autotomy after sciatic cryoneurolysis in the rat.

[1]  J. Deleo,et al.  Autotomy and decreased spinal substance P following peripheral cryogenic nerve lesion. , 1991, Cryobiology.

[2]  M. Iadarola,et al.  Dynorphin increases in the dorsal spinal cord in rats with a painful peripheral neuropathy , 1990, Peptides.

[3]  A. Lipkowski,et al.  Kappa-opioid receptor-mediated antinociception in the rat. II. Supraspinal in addition to spinal sites of action. , 1989, The Journal of pharmacology and experimental therapeutics.

[4]  B. Morris,et al.  Inflammation of the hind limb as a model of unilateral, localized pain: influence on multiple opioid systems in the spinal cord of the rat , 1988, Pain.

[5]  R. Dubner,et al.  Enhancement of dynorphin gene expression in spinal cord following experimental inflammation: stimulus specificity, behavioral parameters and opioid receptor binding , 1988, Pain.

[6]  J. Holaday,et al.  Neurological dysfunction after intrathecal injection of dynorphin A (1-13) in the rat. I. Injection procedures modify pharmacological responses. , 1988, The Journal of pharmacology and experimental therapeutics.

[7]  R. Przewłocki,et al.  The influence of chronic stress on multiple opioid peptide systems in the rat: pronounced effects upon dynorphin in spinal cord , 1987, Brain Research.

[8]  T. Yaksh,et al.  Dynorphin A and related peptides administered intrathecally in the rat: a search for putative kappa opiate receptor activity. , 1986, The Journal of pharmacology and experimental therapeutics.

[9]  R. Melzack,et al.  Deafferentation and chronic pain in animais: An evaluation of evidence suggesting autotomy is related to pain , 1986, Pain.

[10]  A. Faden,et al.  Changes in substance P and somatostatin in the spinal cord after traumatic spinal injury in the rat , 1985, Neuropeptides.

[11]  A. Faden,et al.  Endogenous opioid immunoreactivity in rat spinal cord following traumatic injury , 1985, Annals of neurology.

[12]  L. Kruger,et al.  Deafferentation in animals as a model for the study of pain: an alternative hypothesis , 1984, Brain Research Reviews.

[13]  R. Hallin,et al.  Continuous naloxone administration via osmotic minipump decreases autotomy but has no effect on nociceptive threshold in the rat , 1983, Pain.

[14]  R. Hallin,et al.  Influence of nerve lesions, strain differences and continuous cold stress on chronic pain behavior in rats , 1981, Physiology & Behavior.

[15]  R. Przewłocki,et al.  The influence of foot-shock stress upon brain, pituitary and spinal cord pools of immunoreactive dynorphin in rats , 1981, Neuroscience Letters.

[16]  W. Sweet.  Animal models of chronic pain: Their possible validation from human experience with posterior rhizotomy and congenital analgesia (Part I of the Second John J. Bonica Lecture) , 1981, PAIN.

[17]  P. D. Wall,et al.  Autotomy following peripheral nerve lesions: experimental anesthesia dolorosa , 1979, Pain.

[18]  T. Yaksh,et al.  Chronic catheterization of the spinal subarachnoid space , 1976, Physiology & Behavior.

[19]  T. Yaksh,et al.  Analgesia mediated by a direct spinal action of narcotics. , 1976, Science.

[20]  R. Dirksen,et al.  Intrathecal somatostatin produces effects dependent on the interval between catheter implantation and drug injection. , 1990, Life sciences.