Lipopolysaccharide (LPS) Induces the Apoptosis and Inhibits Osteoblast Differentiation Through JNK Pathway in MC3T3-E1 Cells
暂无分享,去创建一个
Chun Guo | Lei Yuan | Guo-Hua Song | Jian-guo Wang | Jin-ling Song | Fei Wang | Xu-Kai Yang | Fu-hua Zhang | Xu-yuan Ma | Qian Cheng
[1] Chun Guo,et al. Lipopolysaccharide (LPS) promotes osteoclast differentiation and activation by enhancing the MAPK pathway and COX-2 expression in RAW264.7 cells. , 2013, International journal of molecular medicine.
[2] G. Park,et al. Endoplasmic Reticulum Stress-Mediated Apoptosis of EBV-Transformed B Cells by Cross-Linking of CD70 Is Dependent upon Generation of Reactive Oxygen Species and Activation of p38 MAPK and JNK Pathway , 2010, The Journal of Immunology.
[3] T. Ohnishi,et al. Molecular mechanisms of the inhibitory effect of lipopolysaccharide (LPS) on osteoblast differentiation. , 2010, Biochemical and biophysical research communications.
[4] Qiang Xu,et al. Porphyromonas gingivalis lipopolysaccharide inhibits the osteoblastic differentiation of preosteoblasts by activating Notch1 signaling , 2010, Journal of cellular physiology.
[5] H. Ochi,et al. The roles of TNFR1 in lipopolysaccharide‐induced bone loss: Dual effects of TNFR1 on bone metabolism via osteoclastogenesis and osteoblast survival , 2009, Journal of orthopaedic research : official publication of the Orthopaedic Research Society.
[6] G. Emons,et al. GnRH-II antagonists induce apoptosis in human endometrial, ovarian, and breast cancer cells via activation of stress-induced MAPKs p38 and JNK and proapoptotic protein Bax. , 2009, Cancer research.
[7] A. Masuda,et al. JNK Activity Is Essential for Atf4 Expression and Late‐Stage Osteoblast Differentiation , 2009, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[8] J. Kim,et al. Mevinolin enhances osteogenic genes (ALP, type I collagen and osteocalcin), CD44, CD47 and CD51 expression during osteogenic differentiation. , 2009, Life sciences.
[9] T. Giese,et al. Lipopolysaccharides (LPS) induce the differentiation of human monocytes to osteoclasts in a tumour necrosis factor (TNF) alpha-dependent manner: a link between infection and pathological bone resorption. , 2008, Molecular immunology.
[10] F. Hassan,et al. Bacterial lipopolysaccharide induces osteoclast formation in RAW 264.7 macrophage cells. , 2007, Biochemical and biophysical research communications.
[11] L. Bonewald,et al. CD40 ligand blocks apoptosis induced by tumor necrosis factor alpha, glucocorticoids, and etoposide in osteoblasts and the osteocyte-like cell line murine long bone osteocyte-Y4. , 2003, Endocrinology.
[12] T. Nishihara,et al. Lipopolysaccharide Promotes the Survival of Osteoclasts Via Toll-Like Receptor 4, but Cytokine Production of Osteoclasts in Response to Lipopolysaccharide Is Different from That of Macrophages 1 , 2003, The Journal of Immunology.
[13] W. Zou,et al. Dual Modulation of Osteoclast Differentiation by Lipopolysaccharide , 2002, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[14] Huitu Liu,et al. MAPK signal pathways in the regulation of cell proliferation in mammalian cells , 2002, Cell Research.
[15] Thomas D. Schmittgen,et al. Analysis of relative gene expression data using real-time quantitative PCR and the 2(-Delta Delta C(T)) Method. , 2001, Methods.
[16] M. Cobb,et al. Mitogen-activated protein (MAP) kinase pathways: regulation and physiological functions. , 2001, Endocrine reviews.
[17] G. Karsenty,et al. Cbfa1 Contributes to the Osteoblast-specific Expression of type I collagen Genes* , 2001, The Journal of Biological Chemistry.
[18] S. Rhodes,et al. Cloning and Functional Analysis of a Family of Nuclear Matrix Transcription Factors (NP/NMP4) that Regulate Type I Collagen Expression in Osteoblasts , 2001, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[19] D. Graves,et al. Interleukin-1 and Tumor Necrosis Factor Activities Partially Account for Calvarial Bone Resorption Induced by Local Injection of Lipopolysaccharide , 1999, Infection and Immunity.
[20] T. Nagata,et al. Inhibition of Osteoblastic Cell Differentiation by Lipopolysaccharide Extract from Porphyromonas gingivalis , 1999, Infection and Immunity.
[21] T. Martin,et al. Modulation of osteoclast differentiation and function by the new members of the tumor necrosis factor receptor and ligand families. , 1999, Endocrine reviews.
[22] E. Levin,et al. Extracellular Signal-regulated Protein Kinase/Jun Kinase Cross-talk Underlies Vascular Endothelial Cell Growth Factor-induced Endothelial Cell Proliferation* , 1998, The Journal of Biological Chemistry.
[23] Roodman Gd. Advances in bone biology: the osteoclast. , 1996, Endocrine reviews.
[24] Michael T. Wilson,et al. Bacterially induced bone destruction: mechanisms and misconceptions , 1996, Infection and immunity.
[25] M. Hirata,et al. Lipopolysaccharides from Porphyromonas gingivalis, Prevotella intermedia and Actinobacillus actinomycetemcomitans promote osteoclastic differentiation in vitro. , 1996, Archives of oral biology.
[26] G. Roodman,et al. Advances in bone biology: the osteoclast. , 1996, Endocrine reviews.
[27] Michael E. Greenberg,et al. Opposing Effects of ERK and JNK-p38 MAP Kinases on Apoptosis , 1995, Science.
[28] M. de Vernejoul,et al. Local bone injections of LPS and M-CSF increase bone resorption by different pathways in vivo in rats. , 1993, The American journal of physiology.