ETV6/RUNX1-positive relapses evolve from an ancestral clone and frequently acquire deletions of genes implicated in glucocorticoid signaling.
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R. Arceci | A. Hall | J. Harbott | O. Haas | R. Kofler | M. Kauer | R. Marschalek | U. Kaindl | C. Meyer | G. Mann | S. Strehl | M. Metzler | G. Krapf | R. Grausenburger | L. Meyer | G. Fuka | A. Inthal | L. Kuster | R. Panzer-Grümayer | Johannes Rainer