Dependent Mice Is T Cell Autoantigen-Specific and Rat Insulin Promoter-mCD 80-Transgenic Low Dose Streptozotocin-Induced Diabetes in
暂无分享,去创建一个
[1] D. Harlan,et al. Mice expressing both B7-1 and viral glycoprotein on pancreatic beta cells along with glycoprotein-specific transgenic T cells develop diabetes due to a breakdown of T-lymphocyte unresponsiveness. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[2] W. Deppert,et al. References Subscriptions Permissions Email Alerts Relationship Among Immunodominance of Single , 2013 .
[3] A. Müllbacher,et al. The generation of memory antigen‐specific cytotoxic T cell responses by CD28/CD80 interactions in the absence of antigen , 1997, European journal of immunology.
[4] 望月 圭. B7/BB-1 Expression and Hepatitis Activity in Liver Tissues of Patients With Chronic Hepatitis C , 1997 .
[5] D. Harlan,et al. Protection against lethal toxic shock by targeted disruption of the CD28 gene , 1996, The Journal of experimental medicine.
[6] S. Swain,et al. Single cell analyses of cytokine production. , 1997, Current opinion in immunology.
[7] P. Linsley,et al. Comparative analysis of B7-1 and B7-2 costimulatory ligands: expression and function , 1994, The Journal of experimental medicine.
[8] C. June,et al. CD28‐deficient mice generate an impaired Th2 response to Schistosoma mansoni infection , 1996, European journal of immunology.
[9] J. Gribben,et al. Structure, expression, and T cell costimulatory activity of the murine homologue of the human B lymphocyte activation antigen B7 , 1991, The Journal of experimental medicine.
[10] C. Janeway,et al. Expression of the Co-stimulator Molecule B7–1 in Pancreatic β-Cells Accelerates Diabetes in the NOD Mouse , 1995, Diabetes.
[11] J. Bluestone,et al. CD28/B7 costimulation regulates autoimmune diabetes induced with multiple low doses of streptozotocin. , 1997, Journal of immunology.
[12] C. Janeway,et al. CD8 T cell clones from young nonobese diabetic (NOD) islets can transfer rapid onset of diabetes in NOD mice in the absence of CD4 cells , 1996, The Journal of experimental medicine.
[13] Y. T. Kim,et al. Immunologic Studies on the Induction of Diabetes in Experimental Animals: Cellular Basis for the Induction of Diabetes by Streptozotocin , 1984, Diabetes.
[14] Y. Paterson,et al. Identification of an autologous insulin B chain peptide as a target antigen for H-2Kb-restricted cytotoxic T lymphocytes , 1992, The Journal of experimental medicine.
[15] H. Pircher,et al. T cell priming versus T cell tolerance induced by synthetic peptides , 1995, The Journal of experimental medicine.
[16] N. Sarvetnick,et al. Transgenic mice expressing IFN-gamma in pancreatic beta-cells are resistant to streptozotocin-induced diabetes. , 1995, The American journal of physiology.
[17] M. Oldstone,et al. Optimal lymphocytic choriomeningitis virus sequences restricted by H-2Db major histocompatibility complex class I molecules and presented to cytotoxic T lymphocytes , 1995, Journal of virology.
[18] D. Harlan,et al. Very-Low-Dose Streptozotocin Induces Diabetes in Insulin Promoter-mB7-1 Transgenic Mice , 1995, Diabetes.
[19] R. Flavell,et al. Coexpression of B7-1 and viral ("self") transgenes in pancreatic beta cells can break peripheral ignorance and lead to spontaneous autoimmune diabetes. , 1995, Immunity.
[20] K. Okumura,et al. CD86 (B70/B7-2) on endothelial cells co-stimulates allogeneic CD4+ T cells. , 1995, International immunology.
[21] T. Mak,et al. CD28-deficient mice are highly resistant to collagen-induced arthritis. , 1999, Journal of immunology.
[22] C. Thompson,et al. CD28 is required for germinal center formation. , 1996, Journal of immunology.
[23] S. Bonner-Weir,et al. Beta cell mass and growth after syngeneic islet cell transplantation in normal and streptozocin diabetic C57BL/6 mice. , 1993, The Journal of clinical investigation.
[24] H. von Boehmer,et al. Autoimmune insulitis and diabetes in the absence of antigen‐specific contact between T cells and β‐islet cells , 1999, European journal of immunology.
[25] D. Hanahan,et al. Self Antigens Expressed by Solid Tumors Do Not Efficiently Stimulate Naive or Activated T Cells: Implications for Immunotherapy , 1997, The Journal of experimental medicine.
[26] A. Rossini,et al. Genetic Influence of the Streptozotocin-induced Insulitis and Hyperglycemia , 1977, Diabetes.
[27] Y. Matsuzawa,et al. The NOD mouse. , 1994, Diabetes research and clinical practice.
[28] J. Sidney,et al. Uncovering subdominant cytotoxic T-lymphocyte responses in lymphocytic choriomeningitis virus-infected BALB/c mice , 1997, Journal of virology.
[29] P. Linsley,et al. Functional expression of the costimulatory molecule, B7/BB1, on murine dendritic cell populations , 1992, The Journal of experimental medicine.
[30] D. Wegmann. The immune response to islets in experimental diabetes and insulin-dependent diabetes mellitus. , 1996, Current opinion in immunology.
[31] H. Pircher,et al. Ablation of “tolerance” and induction of diabetes by virus infection in viral antigen transgenic mice , 1991, Cell.
[32] C. June,et al. Primary porcine endothelial cells express membrane-bound B7-2 (CD86) and a soluble factor that co-stimulate cyclosporin A-resistant and CD28-dependent human T cell proliferation. , 1996, International immunology.
[33] A. Rossini,et al. Streptozotocin-induced pancreatic insulitis: new model of diabetes mellitus. , 1976, Science.
[34] K. Haskins,et al. Diabetogenic T-Cell Clones , 1996, Diabetes.
[35] K. Haskins,et al. Acceleration of diabetes in young NOD mice with a CD4+ islet-specific T cell clone. , 1990, Science.
[36] C. Janeway,et al. Signals and signs for lymphocyte responses , 1994, Cell.
[37] J. Rotter,et al. Genetic and physical mapping of a type 1 diabetes susceptibility gene (IDDM12) to a 100-kb phagemid artificial chromosome clone containing D2S72-CTLA4-D2S105 on chromosome 2q33. , 2000, Diabetes.
[38] T. Mak,et al. CD28 is not required for rejection of unmanipulated syngeneic and autologous tumors , 1997, European journal of immunology.
[39] S. Tonegawa,et al. CD28 costimulation is crucial for the development of spontaneous autoimmune encephalomyelitis. , 1999, Journal of immunology.
[40] W. Chick,et al. Studies of streptozotocin-induced insulitis and diabetes. , 1977, Proceedings of the National Academy of Sciences of the United States of America.
[41] T. Mak,et al. Skin allograft rejection in CD28-deficient mice. , 1996, Transplantation.
[42] W. Heath,et al. Cytotoxic T lymphocyte activation by cross-priming. , 1999, Current opinion in immunology.
[43] P. Linsley,et al. Costimulator B7-1 confers antigen-presenting-cell function to parenchymal tissue and in conjunction with tumor necrosis factor alpha leads to autoimmunity in transgenic mice. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[44] R. Flavell,et al. The role of the T cell costimulator B7-1 in autoimmunity and the induction and maintenance of tolerance to peripheral antigen. , 1994, Immunity.
[45] E. Fuchs,et al. CD28/B7 regulation of Th1 and Th2 subsets in the development of autoimmune diabetes. , 1996, Immunity.
[46] J. Miller,et al. Constitutive class I-restricted exogenous presentation of self antigens in vivo , 1996, The Journal of experimental medicine.
[47] A. Rossini,et al. Sex differences in the multiple-dose streptozotocin model of diabetes. , 1978, Endocrinology.
[48] J. Bluestone,et al. Absence of B7-dependent responses in CD28-deficient mice. , 1994, Immunity.
[49] L. Orci,et al. Expression of a tumor necrosis factor alpha transgene in murine pancreatic beta cells results in severe and permanent insulitis without evolution towards diabetes , 1992, The Journal of experimental medicine.
[50] H. Kolb,et al. Genetic control of low-dose streptozotocin-induced autoimmune diabetes in mice. , 1983, Journal of immunology.
[51] A. Monaco,et al. An improved method for isolation of mouse pancreatic islets. , 1985, Transplantation.
[52] P. Linsley,et al. The role of the CD28 receptor during T cell responses to antigen. , 1993, Annual review of immunology.
[53] D. Harlan,et al. Inflammatory cytokines IFN-gamma plus TNF-alpha induce regulated expression of CD80 (B7-1) but not CD86 (B7-2) on murine fibroblasts. , 1997, Journal of immunology.
[54] J. Kekow,et al. Measurement of Rat Insulin: Enzyme-Linked Immunosorbent Assay With Increased Sensitivity, High Accuracy, and Greater Practicability Than Established Radioimmunoassay , 1988, Diabetes.
[55] B Zheng,et al. Cellular interaction in germinal centers. Roles of CD40 ligand and B7-2 in established germinal centers. , 1995, Journal of immunology.
[56] K P Lee,et al. Differential T cell costimulatory requirements in CD28-deficient mice. , 1993, Science.