Escherichia coli can survive stress by noisy growth modulation
暂无分享,去创建一个
A. Phillips | J. Locke | Matt Jones | Om Patange | Christian Schwall | Casandra E. Villava | Douglas A. Griffith
[1] J. Wade,et al. Spurious transcription and its impact on cell function , 2018, Transcription.
[2] Sahand Hormoz,et al. Molecular Time Sharing through Dynamic Pulsing in Single Cells , 2018, Cell systems.
[3] M. Sørensen,et al. Prophages and Growth Dynamics Confound Experimental Results with Antibiotic-Tolerant Persister Cells , 2017, mBio.
[4] T. Bollenbach,et al. Noisy Response to Antibiotic Stress Predicts Subsequent Single-Cell Survival in an Acidic Environment. , 2017, Cell systems.
[5] Matthias Heinemann,et al. Molecular Systems Biology Peer Review Process File Bacterial Persistence Is an Active Σ S Stress Response to Metabolic Flux Limitation Transaction Report , 2022 .
[6] Burak Okumus,et al. Mechanical slowing-down of cytoplasmic diffusion allows in vivo counting of proteins in individual cells , 2016, Nature Communications.
[7] Martin Ackermann,et al. Phenotypic heterogeneity driven by nutrient limitation promotes growth in fluctuating environments , 2016, Nature Microbiology.
[8] Jatin Narula,et al. Slowdown of growth controls cellular differentiation , 2016, Molecular systems biology.
[9] T. Wood,et al. Persistence Increases in the Absence of the Alarmone Guanosine Tetraphosphate by Reducing Cell Growth , 2016, Scientific Reports.
[10] Yu Tanouchi,et al. A noisy linear map underlies oscillations in cell size and gene expression in bacteria , 2015, Nature.
[11] John T. Sauls,et al. Cell-Size Control and Homeostasis in Bacteria , 2015, Current Biology.
[12] D. J. Kiviet,et al. Stochasticity of metabolism and growth at the single-cell level , 2014, Nature.
[13] Stefan Klumpp,et al. A Model for Sigma Factor Competition in Bacterial Cells , 2014, PLoS Comput. Biol..
[14] Viola Vogel,et al. Bistable Expression of Virulence Genes in Salmonella Leads to the Formation of an Antibiotic-Tolerant Subpopulation , 2014, PLoS biology.
[15] A. Simons,et al. Experimental evolution of bet hedging under manipulated environmental uncertainty in Neurospora crassa , 2014, Proceedings of the Royal Society B: Biological Sciences.
[16] Jan Kok,et al. Bet-hedging during bacterial diauxic shift , 2014, Proceedings of the National Academy of Sciences.
[17] B. Palsson,et al. Genome-scale reconstruction of the sigma factor network in Escherichia coli: topology and functional states , 2014, BMC Biology.
[18] Eshel Ben-Jacob,et al. Growth feedback as a basis for persister bistability , 2013, Proceedings of the National Academy of Sciences.
[19] K. Gerdes,et al. RETRACTED: (p)ppGpp Controls Bacterial Persistence by Stochastic Induction of Toxin-Antitoxin Activity , 2013, Cell.
[20] Stanislas Leibler,et al. Dynamic Persistence of Antibiotic-Stressed Mycobacteria , 2013, Science.
[21] Sasha F. Levy,et al. Bet Hedging in Yeast by Heterogeneous, Age-Correlated Expression of a Stress Protectant , 2012, PLoS biology.
[22] Eric Mjolsness,et al. Measuring single-cell gene expression dynamics in bacteria using fluorescence time-lapse microscopy , 2011, Nature Protocols.
[23] James C. W. Locke,et al. Stochastic Pulse Regulation in Bacterial Stress Response , 2011, Science.
[24] N. Majdalani,et al. The RpoS-mediated general stress response in Escherichia coli. , 2011, Annual review of microbiology.
[25] N. Philippe,et al. ppGpp is the major source of growth rate control in E. coli. , 2011, Environmental microbiology.
[26] T. Conway,et al. Discretely calibrated regulatory loops controlled by ppGpp partition gene induction across the ‘feast to famine’ gradient in Escherichia coli , 2011, Molecular microbiology.
[27] T. Hwa,et al. Interdependence of Cell Growth and Gene Expression: Origins and Consequences , 2010, Science.
[28] Paul J. Choi,et al. Quantifying E. coli Proteome and Transcriptome with Single-Molecule Sensitivity in Single Cells , 2010, Science.
[29] Andrew Wright,et al. Robust Growth of Escherichia coli , 2010, Current Biology.
[30] Elizabeth A. Calle,et al. Quantifying E. coli Proteome and Transcriptome with Single-Molecule Sensitivity in Single Cells , 2010, Science.
[31] T. Hwa,et al. Growth Rate-Dependent Global Effects on Gene Expression in Bacteria , 2009, Cell.
[32] L. You,et al. Emergent bistability by a growth-modulating positive feedback circuit. , 2009, Nature chemical biology.
[33] T. Hwa,et al. Growth-rate-dependent partitioning of RNA polymerases in bacteria , 2008, Proceedings of the National Academy of Sciences.
[34] Jerome T. Mettetal,et al. Stochastic switching as a survival strategy in fluctuating environments , 2008, Nature Genetics.
[35] H. Schellhorn,et al. Control of RpoS in global gene expression of Escherichia coli in minimal media , 2008, Molecular Genetics and Genomics.
[36] S. Gottesman,et al. ppGpp regulation of RpoS degradation via anti-adaptor protein IraP , 2007, Proceedings of the National Academy of Sciences.
[37] D. L. Venable. Bet hedging in a guild of desert annuals. , 2007, Ecology.
[38] A. Arkin,et al. From Fluctuations to Phenotypes: The Physiology of Noise , 2006, Science's STKE.
[39] Nir Friedman,et al. Linking stochastic dynamics to population distribution: an analytical framework of gene expression. , 2006, Physical review letters.
[40] U. Alon,et al. A comprehensive library of fluorescent transcriptional reporters for Escherichia coli , 2006, Nature Methods.
[41] H. Mori,et al. Construction of Escherichia coli K-12 in-frame, single-gene knockout mutants: the Keio collection , 2006, Molecular systems biology.
[42] J Hasty,et al. Cellular growth and division in the Gillespie algorithm. , 2004, Systems biology.
[43] S. Leibler,et al. Bacterial Persistence as a Phenotypic Switch , 2004, Science.
[44] M. Thattai,et al. Stochastic Gene Expression in Fluctuating Environments , 2004, Genetics.
[45] K. Niklas,et al. Springer-Verlag 2004 , 2004 .
[46] H. Schellhorn,et al. Controlled induction of the RpoS regulon in Escherichia coli, using an RpoS-expressing plasmid. , 2003, Canadian journal of microbiology.
[47] C. Rao,et al. Stochastic chemical kinetics and the quasi-steady-state assumption: Application to the Gillespie algorithm , 2003 .
[48] R. Hengge-aronis,et al. Stationary phase gene regulation: what makes an Escherichia coli promoter sigmaS-selective? , 2002, Current opinion in microbiology.
[49] P. Swain,et al. Stochastic Gene Expression in a Single Cell , 2002, Science.
[50] J. Imlay,et al. Alkyl Hydroperoxide Reductase Is the Primary Scavenger of Endogenous Hydrogen Peroxide in Escherichia coli , 2001, Journal of bacteriology.
[51] N. Fujita,et al. Competition among seven Escherichia coli sigma subunits: relative binding affinities to the core RNA polymerase. , 2000, Nucleic acids research.
[52] Akira Ishihama,et al. Transcriptional Organization and In Vivo Role of theEscherichia coli rsd Gene, Encoding the Regulator of RNA Polymerase Sigma D , 1999, Journal of bacteriology.
[53] U. Alon,et al. Robustness in bacterial chemotaxis , 2022 .
[54] T. Nyström,et al. Negative regulation by RpoS: a case of sigma factor competition , 1998, Molecular microbiology.
[55] A. Ishihama,et al. A stationary phase protein in Escherichia coli with binding activity to the major sigma subunit of RNA polymerase. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[56] R. Hengge-aronis,et al. The cellular concentration of the sigma S subunit of RNA polymerase in Escherichia coli is controlled at the levels of transcription, translation, and protein stability. , 1994, Genes & development.
[57] A. Ishihama,et al. Heterogeneity of the principal sigma factor in Escherichia coli: the rpoS gene product, sigma 38, is a second principal sigma factor of RNA polymerase in stationary-phase Escherichia coli. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[58] Akira Ishihama,et al. Heterogeneity of the principal sigma factor in Escherichia coli: the rpoS gene product, sigma 38, is a second principal sigma factor of RNA polymerase in stationary-phase Escherichia coli. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[59] N. Henneberg,et al. Osmotic regulation of rpoS-dependent genes in Escherichia coli , 1993, Journal of bacteriology.
[60] H. Xiao,et al. Residual guanosine 3',5'-bispyrophosphate synthetic activity of relA null mutants can be eliminated by spoT null mutations. , 1991, The Journal of biological chemistry.
[61] R. Hengge-aronis,et al. Identification of a central regulator of stationary‐phase gene expression in Escherichia coli , 1991, Molecular microbiology.
[62] G. Schreiber,et al. Characterization of the relA1 mutation and a comparison of relA1 with new relA null alleles in Escherichia coli. , 1989, The Journal of biological chemistry.
[63] H. Schellhorn,et al. Transcriptional regulation of katE in Escherichia coli K-12 , 1988, Journal of bacteriology.
[64] P. Loewen,et al. Catalases HPI and HPII in Escherichia coli are induced independently. , 1985, Archives of biochemistry and biophysics.
[65] William W. Ward,et al. SPECTROPHOTOMETRIC IDENTITY OF THE ENERGY TRANSFER CHROMOPHORES IN RENILLA AND AEQUOREA GREEN‐FLUORESCENT PROTEINS , 1980 .
[66] D. Gillespie. Exact Stochastic Simulation of Coupled Chemical Reactions , 1977 .
[67] O. Maaløe,et al. Dependency on medium and temperature of cell size and chemical composition during balanced grown of Salmonella typhimurium. , 1958, Journal of general microbiology.
[68] J. Bigger. TREATMENT OF STAPHYLOCOCCAL INFECTIONS WITH PENICILLIN BY INTERMITTENT STERILISATION , 1944 .