Fueling Normal and Diseased Hearts: Myocardial Bioenergetics

[1]  A. Terzic,et al.  Cardiac system bioenergetics: metabolic basis of the Frank‐Starling law , 2006, The Journal of physiology.

[2]  R. Bache,et al.  Regulation of coronary vasomotor tone under normal conditions and during acute myocardial hypoperfusion. , 2000, Pharmacology & therapeutics.

[3]  F. Mochel,et al.  Pyruvate carboxylase deficiency: clinical and biochemical response to anaplerotic diet therapy. , 2005, Molecular genetics and metabolism.

[4]  Raimond L Winslow,et al.  A computational model integrating electrophysiology, contraction, and mitochondrial bioenergetics in the ventricular myocyte. , 2006, Biophysical journal.

[5]  Brian O'Rourke,et al.  Elevated Cytosolic Na+ Decreases Mitochondrial Ca2+ Uptake During Excitation–Contraction Coupling and Impairs Energetic Adaptation in Cardiac Myocytes , 2006, Circulation research.

[6]  M. Atalay,et al.  Oxygen consumption and usage during physical exercise: the balance between oxidative stress and ROS-dependent adaptive signaling. , 2013, Antioxidants & redox signaling.

[7]  G. Shulman,et al.  Physiological role of AMP-activated protein kinase in the heart: graded activation during exercise. , 2003, American journal of physiology. Endocrinology and metabolism.

[8]  A. From,et al.  Regulation of the oxidative phosphorylation rate in the intact cell. , 1990, Biochemistry.

[9]  R. Bache,et al.  Regulation of coronary blood flow during exercise. , 2008, Physiological reviews.

[10]  J. Mazat,et al.  Virtual Mitochondria : Metabolic Modelling and Control , 2004, Molecular Biology Reports.

[11]  M. L. Genova,et al.  Functional role of mitochondrial respiratory supercomplexes. , 2014, Biochimica et biophysica acta.

[12]  Yi Zhang,et al.  Oxygen delivery does not limit cardiac performance during high work states. , 1999, American journal of physiology. Heart and circulatory physiology.

[13]  L. Opie,et al.  Metabolic mechanisms in heart failure. , 2007, Circulation.

[14]  R. Winslow,et al.  An integrated mitochondrial ROS production and scavenging model: implications for heart failure. , 2013, Biophysical journal.

[15]  A. Das Regulation of the mitochondrial ATP-synthase in health and disease. , 2003, Molecular genetics and metabolism.

[16]  M. Noble,et al.  Preferential uptake of lactate by the normal myocardium in dogs. , 1980, Cardiovascular research.

[17]  R. Bache,et al.  ATP-sensitive K+ channels, adenosine, and nitric oxide-mediated mechanisms account for coronary vasodilation during exercise. , 1998, Circulation research.

[18]  William C Stanley,et al.  Myocardial substrate metabolism in the normal and failing heart. , 2005, Physiological reviews.

[19]  M. Vendelin,et al.  Functional coupling as a basic mechanism of feedback regulation of cardiac energy metabolism , 2004, Molecular and Cellular Biochemistry.

[20]  Mohit M. Jain,et al.  Cardiac-Specific Overexpression of GLUT1 Prevents the Development of Heart Failure Attributable to Pressure Overload in Mice , 2002, Circulation.

[21]  C. Sen ARS turns fifteen: la quinceañera bonita. , 2013, Antioxidants & redox signaling.

[22]  G. Lopaschuk,et al.  AMP-activated protein kinase (AMPK) control of fatty acid and glucose metabolism in the ischemic heart. , 2003, Progress in lipid research.

[23]  R. Balaban,et al.  Role of mitochondrial Ca2+ in the regulation of cellular energetics. , 2012, Biochemistry.

[24]  B. Korzeniewski Parallel activation in the ATP supply-demand system lessens the impact of inborn enzyme deficiencies, inhibitors, poisons or substrate shortage on oxidative phosphorylation in vivo. , 2002, Biophysical chemistry.

[25]  S. Powers,et al.  Reactive oxygen species: impact on skeletal muscle. , 2011, Comprehensive Physiology.

[26]  A. From,et al.  Signaling and expression for mitochondrial membrane proteins during left ventricular remodeling and contractile failure after myocardial infarction. , 2000, Journal of the American College of Cardiology.

[27]  W WardChristopher,et al.  Mechanical Stretch-Induced Activation of ROS/RNS Signaling in Striated Muscle , 2014 .

[28]  M. Noble,et al.  Chronic Catecholamine Depletion Switches Myocardium from Carbohydrate to Lipid Utilisation , 2001, Cardiovascular Drugs and Therapy.

[29]  V. Mootha,et al.  Ca2+ activation of heart mitochondrial oxidative phosphorylation: role of the F0/F1-ATPase , 2000 .

[30]  H. Taegtmeyer,et al.  Reactivation of Peroxisome Proliferator-activated Receptor α Is Associated with Contractile Dysfunction in Hypertrophied Rat Heart* , 2001, The Journal of Biological Chemistry.

[31]  B. O’Rourke,et al.  Elevated Cytosolic Na+ Increases Mitochondrial Formation of Reactive Oxygen Species in Failing Cardiac Myocytes , 2010, Circulation.

[32]  D. Kelly,et al.  Transcriptional Activation Of Energy Metabolic Switches In The Developing And Hypertrophied Heart , 2002, Clinical and experimental pharmacology & physiology.

[33]  M. Kushmerick,et al.  Energetics of muscle contraction: the whole is less than the sum of its parts. , 2001, Biochemical Society transactions.

[34]  M. Roden How free fatty acids inhibit glucose utilization in human skeletal muscle. , 2004, News in physiological sciences : an international journal of physiology produced jointly by the International Union of Physiological Sciences and the American Physiological Society.

[35]  A. Bonen,et al.  Protein-mediated fatty acid uptake: regulation by contraction, AMP-activated protein kinase, and endocrine signals. , 2007, Applied physiology, nutrition, and metabolism = Physiologie appliquee, nutrition et metabolisme.

[36]  M. Hüttemann,et al.  Cytochrome c Oxidase and the Regulation of Oxidative Phosphorylation , 2001, Chembiochem : a European journal of chemical biology.

[37]  M. Murphy,et al.  Mitochondrially targeted compounds and their impact on cellular bioenergetics☆ , 2013, Redox biology.

[38]  Y. Usson,et al.  Modular organization of cardiac energy metabolism: energy conversion, transfer and feedback regulation , 2015, Acta physiologica.

[39]  H. E. Keurs,et al.  The Sarcomeric Control of Energy Conversion , 2005, Annals of the New York Academy of Sciences.

[40]  M. Brand,et al.  Simplifying metabolic complexity. , 2001, Biochemical Society transactions.

[41]  B. O’Rourke,et al.  Excitation-contraction coupling and mitochondrial energetics , 2007, Basic Research in Cardiology.

[42]  E. Feigl,et al.  Feedforward sympathetic coronary vasodilation in exercising dogs. , 2000, Journal of applied physiology.

[43]  F. Mochel,et al.  Anaplerotic diet therapy in inherited metabolic disease: Therapeutic potential , 2006, Journal of Inherited Metabolic Disease.

[44]  A. Terzic,et al.  Phosphotransfer networks and cellular energetics , 2003, Journal of Experimental Biology.

[45]  P W Hochachka,et al.  Intracellular convection, homeostasis and metabolic regulation , 2003, Journal of Experimental Biology.

[46]  B. Korzeniewski Theoretical studies on the regulation of oxidative phosphorylation in intact tissues. , 2001, Biochimica et biophysica acta.