Sodium thiosulphate attenuates brain inflammation induced by systemic lipopolysaccharide administration in C57BL/6J mice

[1]  P. Pożarowski,et al.  The era of anti-vascular endothelial growth factor (VEGF) drugs in ophthalmology, VEGF and anti-VEGF therapy , 2016, Central-European journal of immunology.

[2]  C. Patrono Cardiovascular effects of cyclooxygenase‐2 inhibitors: a mechanistic and clinical perspective , 2016, British journal of clinical pharmacology.

[3]  P. C. Lopes LPS and neuroinflammation: a matter of timing , 2016, Inflammopharmacology.

[4]  R. Ransohoff How neuroinflammation contributes to neurodegeneration , 2016, Science.

[5]  T. Govezensky,et al.  Alpha-mangostin attenuates brain inflammation induced by peripheral lipopolysaccharide administration in C57BL/6J mice , 2016, Journal of Neuroimmunology.

[6]  S. Ferreira,et al.  Microglial dysfunction connects depression and Alzheimer’s disease , 2016, Brain, Behavior, and Immunity.

[7]  E. Porcellini,et al.  Persistent infections, immune-senescence and Alzheimer's disease , 2016, Oncoscience.

[8]  Paul Edison,et al.  Neuroinflammation in Alzheimer's disease: Current evidence and future directions , 2016, Alzheimer's & Dementia.

[9]  P. Mcgeer,et al.  Sodium thiosulfate attenuates glial-mediated neuroinflammation in degenerative neurological diseases , 2016, Journal of Neuroinflammation.

[10]  A. Salminen,et al.  Inflammation and its role in age-related macular degeneration , 2016, Cellular and Molecular Life Sciences.

[11]  B. Brown,et al.  Infectious disease burden and cognitive function in young to middle-aged adults , 2016, Brain, Behavior, and Immunity.

[12]  J. Godbout,et al.  Sequential activation of microglia and astrocyte cytokine expression precedes increased iba‐1 or GFAP immunoreactivity following systemic immune challenge , 2016, Glia.

[13]  G. Gevorkian,et al.  LPS-induced Murine Neuroinflammation Model: Main Features and Suitability for Pre-clinical Assessment of Nutraceuticals , 2016, Current neuropharmacology.

[14]  B. Liu,et al.  In Vivo Detection of Age- and Disease-Related Increases in Neuroinflammation by 18F-GE180 TSPO MicroPET Imaging in Wild-Type and Alzheimer's Transgenic Mice , 2015, The Journal of Neuroscience.

[15]  Mohajeet B. Bhuckory,et al.  Targeting translocator protein (18 kDa) (TSPO) dampens pro-inflammatory microglia reactivity in the retina and protects from degeneration , 2015, Journal of Neuroinflammation.

[16]  R. Sriram,et al.  Sodium thiosulfate protects brain in rat model of adenine induced vascular calcification , 2015, Neurochemistry International.

[17]  K. Hanaoka,et al.  Thiosulfate Mediates Cytoprotective Effects of Hydrogen Sulfide Against Neuronal Ischemia , 2015, Journal of the American Heart Association.

[18]  T. Wong,et al.  Epidemiology of diabetic retinopathy, diabetic macular edema and related vision loss , 2015, Eye and Vision.

[19]  A. Vezzani,et al.  Neuromodulatory properties of inflammatory cytokines and their impact on neuronal excitability , 2015, Neuropharmacology.

[20]  S. Kondo,et al.  Protective effects of sodium thiosulfate for cisplatin-mediated ototoxicity in patients with head and neck cancer , 2015, Acta oto-laryngologica.

[21]  W. V. van Gool,et al.  Systemic inflammation and microglial activation: systematic review of animal experiments , 2015, Journal of Neuroinflammation.

[22]  V. Perez,et al.  Immune mechanisms in inflammatory and degenerative eye disease. , 2015, Trends in immunology.

[23]  D. Brooks,et al.  The Role of Neuroinflammation in Dementias , 2015, Current Neurology and Neuroscience Reports.

[24]  R. Sankowski,et al.  Systemic Inflammation and the Brain: Novel Roles of Genetic, Molecular, and Environmental Cues as Drivers of Neurodegeneration , 2015, Front. Cell. Neurosci..

[25]  S. Commins,et al.  Lipopolysaccharide-induced sepsis induces long-lasting affective changes in the mouse , 2015, Brain, Behavior, and Immunity.

[26]  K. Hanaoka,et al.  Sodium Thiosulfate Attenuates Acute Lung Injury in Mice , 2014, Anesthesiology.

[27]  N. Terrando,et al.  Prolonged Neuroinflammation after Lipopolysaccharide Exposure in Aged Rats , 2014, PloS one.

[28]  Michael T. Heneka,et al.  Innate immune activation in neurodegenerative disease , 2014, Nature Reviews Immunology.

[29]  Xiaomin Su,et al.  Immune Responses in Parkinson's Disease: Interplay between Central and Peripheral Immune Systems , 2014, BioMed research international.

[30]  S. Ferreira,et al.  Inflammation, defective insulin signaling, and neuronal dysfunction in Alzheimer's disease , 2014, Alzheimer's & Dementia.

[31]  H. Neumann,et al.  Translocator protein (18 kDa) (TSPO) is expressed in reactive retinal microglia and modulates microglial inflammation and phagocytosis , 2014, Journal of Neuroinflammation.

[32]  P. Acton,et al.  Systemic Immune Activation Leads to Neuroinflammation and Sickness Behavior in Mice , 2013, Mediators of inflammation.

[33]  L. Ferraro,et al.  Multi-target action of the novel anti-Alzheimer compound CHF5074: in vivo study of long term treatment in Tg2576 mice , 2013, BMC Neuroscience.

[34]  S. Soroka,et al.  Oral Sodium Thiosulfate as Maintenance Therapy for Calcific Uremic Arteriolopathy: A Case Series , 2013, American Journal of Nephrology.

[35]  G. Biessels,et al.  An update on type 2 diabetes, vascular dementia and Alzheimer's disease , 2012, Experimental Gerontology.

[36]  K. Tokuda,et al.  Inhaled hydrogen sulfide prevents endotoxin-induced systemic inflammation and improves survival by altering sulfide metabolism in mice. , 2012, Antioxidants & redox signaling.

[37]  D. Cribbs,et al.  Amyloid-β Peptide Binds to Cytochrome C Oxidase Subunit 1 , 2011, Alzheimer's & Dementia.

[38]  C. Cunningham,et al.  Exacerbation of CNS inflammation and neurodegeneration by systemic LPS treatment is independent of circulating IL-1β and IL-6 , 2011, Journal of Neuroinflammation.

[39]  G. Wenk,et al.  Caffeine attenuates lipopolysaccharide-induced neuroinflammation , 2010, Neuroscience Letters.

[40]  M. Hayden,et al.  Sodium Thiosulfate: New Hope for the Treatment of Calciphylaxis , 2010, Seminars in dialysis.

[41]  R. Veerhuis,et al.  Neuroinflammation – An Early Event in Both the History and Pathogenesis of Alzheimer’s Disease , 2010, Neurodegenerative Diseases.

[42]  C. Holmes,et al.  Role of Infection in the Pathogenesis of Alzheimer’s Disease , 2009, CNS drugs.

[43]  Ming-Kai Chen,et al.  Translocator protein 18 kDa (TSPO): molecular sensor of brain injury and repair. , 2008, Pharmacology & therapeutics.

[44]  J. Sowers,et al.  Calciphylaxis: calcific uremic arteriolopathy and the emerging role of sodium thiosulfate , 2008, International Urology and Nephrology.

[45]  K. Strauss Antiinflammatory and neuroprotective actions of COX2 inhibitors in the injured brain , 2008, Brain, Behavior, and Immunity.

[46]  M. Gavish,et al.  The peripheral-type benzodiazepine receptor and the cardiovascular system. Implications for drug development. , 2006, Pharmacology & therapeutics.

[47]  S. Rivest,et al.  Cyclooxygenase 2 (COX‐2) inhibition increases the inflammatory response in the brain during systemic immune stimuli , 2005, Journal of neurochemistry.

[48]  K. Karageuzyan Oxidative stress in the molecular mechanism of pathogenesis at different diseased states of organism in clinics and experiment. , 2005, Current drug targets. Inflammation and allergy.

[49]  Ulrich Schraermeyer,et al.  A central role for inflammation in the pathogenesis of diabetic retinopathy , 2004, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[50]  B. Kirchhof,et al.  Contribution of TNF-alpha to leukocyte adhesion, vascular leakage, and apoptotic cell death in endotoxin-induced uveitis in vivo. , 2003, Investigative ophthalmology & visual science.

[51]  P. Sawchenko,et al.  Distinct Brain Vascular Cell Types Manifest Inducible Cyclooxygenase Expression as a Function of the Strength and Nature of Immune Insults , 2002, The Journal of Neuroscience.

[52]  Jeffrey E. Thomas,et al.  Safety of Intraventricular Sodium Nitroprusside and Thiosulfate for the Treatment of Cerebral Vasospasm in the Intensive Care Unit Setting , 2002, Stroke.

[53]  C. Hoffmann COX-2 in brain and spinal cord implications for therapeutic use. , 2000, Current medicinal chemistry.

[54]  K. Nakadate,et al.  Brain Endothelial Cells Express Cyclooxygenase-2 during Lipopolysaccharide-Induced Fever: Light and Electron Microscopic Immunocytochemical Studies , 1998, The Journal of Neuroscience.

[55]  C Verhagen,et al.  Endotoxin-induced uveitis in the rat. The significance of intraocular interleukin-6. , 1992, Investigative ophthalmology & visual science.

[56]  K. Ramana,et al.  Endotoxin-Induced Uveitis in Rodents. , 2019, Methods in molecular biology.

[57]  P. Edison,et al.  Review Article Neuroinflammation in Alzheimer's disease: Current evidence and future directions , 2016 .

[58]  C. de la Torre,et al.  Calciphylaxis treated with sodium thiosulfate: report of two cases. , 2013, Dermatology online journal.

[59]  C. Dotti,et al.  Peripheral cholesterol, metabolic disorders and Alzheimer's disease. , 2012, Frontiers in bioscience.

[60]  Patrizia Mecocci,et al.  A review of the major vascular risk factors related to Alzheimer's disease. , 2012, Journal of Alzheimer's disease : JAD.

[61]  Peng Li,et al.  Teratogenic effects of sodium thiosulfate on developing zebrafish embryos. , 2009, Frontiers in bioscience.

[62]  S. Aid,et al.  Neuroinflammatory response to lipopolysaccharide is exacerbated in mice genetically deficient in cyclooxygenase-2 , 2008, Journal of Neuroinflammation.

[63]  Lisa Kolb,et al.  Vascular ossification-calcification in metabolic syndrome, type 2 diabetes mellitus, chronic kidney disease, and calciphylaxis-calcific uremic arteriolopathy: the emerging role of sodium thiosulfate. , 2005, Cardiovascular diabetology.

[64]  内門 大丈 Activation of vascular endothelial cells and perivascular cells by systemic inflammation : An immunohistochemical study of postmortem human brain tissues , 2004 .

[65]  Sun,et al.  Sodium thiosulfate protects against acrylonitrile-induced elevation of glial fibrillary acidic protein levels by replenishing glutathione. , 2000, Environmental toxicology and pharmacology.