Ursodeoxycholic Acid Inhibits Inflammatory Responses and Promotes Functional Recovery After Spinal Cord Injury in Rats
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I. Kwon | Donghyun Lee | Seil Sohn | I. Han | W. Ko | S. Kim | Min-Jae Jo | Hyemin Choi | Soo‐Hong Lee
[1] Soo-Hong Lee,et al. Matrix Metalloproteinase-8 Inhibition Prevents Disruption of Blood–Spinal Cord Barrier and Attenuates Inflammation in Rat Model of Spinal Cord Injury , 2017, Molecular Neurobiology.
[2] I. Han,et al. Propitious Therapeutic Modulators to Prevent Blood-Spinal Cord Barrier Disruption in Spinal Cord Injury , 2017, Molecular Neurobiology.
[3] Sung Jun Kim,et al. Anti-inflammatory effects of ursodeoxycholic acid by lipopolysaccharide-stimulated inflammatory responses in RAW 264.7 macrophages , 2017, PloS one.
[4] Hariprakash Haragopal,et al. An efficient device to experimentally model compression injury of mammalian spinal cord , 2015, Experimental Neurology.
[5] S. Chaki,et al. Differential effects of NMDA receptor antagonists at lower and higher doses on basal gamma band oscillation power in rat cortical electroencephalograms , 2014, Neuropharmacology.
[6] Y. Ohkawa,et al. Ly6C+Ly6G− Myeloid‐derived suppressor cells play a critical role in the resolution of acute inflammation and the subsequent tissue repair process after spinal cord injury , 2013, Journal of neurochemistry.
[7] M. Zheng,et al. Traditional Chinese medicines benefit to nonalcoholic fatty liver disease: a systematic review and meta-analysis , 2012, Molecular Biology Reports.
[8] Yu-fei Chen,et al. Tanshinone IIA Attenuates the Inflammatory Response and Apoptosis after Traumatic Injury of the Spinal Cord in Adult Rats , 2012, PloS one.
[9] A. Shunmugavel,et al. The alternative and terminal pathways of complement mediate post-traumatic spinal cord inflammation and injury. , 2010, The American journal of pathology.
[10] T. Woodruff,et al. Quantitative analysis of cellular inflammation after traumatic spinal cord injury: evidence for a multiphasic inflammatory response in the acute to chronic environment. , 2010, Brain : a journal of neurology.
[11] Thomas D. Schmittgen,et al. Analyzing real-time PCR data by the comparative CT method , 2008, Nature Protocols.
[12] E. Mazzon,et al. Evidence for the Role of Mitogen-Activated Protein Kinase Signaling Pathways in the Development of Spinal Cord Injury , 2008, Journal of Pharmacology and Experimental Therapeutics.
[13] P. Popovich,et al. Inflammation and its role in neuroprotection, axonal regeneration and functional recovery after spinal cord injury , 2008, Experimental Neurology.
[14] Tony J Collins,et al. ImageJ for microscopy. , 2007, BioTechniques.
[15] David A Ramsay,et al. The cellular inflammatory response in human spinal cords after injury. , 2006, Brain : a journal of neurology.
[16] Fred H. Gage,et al. Therapeutic interventions after spinal cord injury , 2006, Nature Reviews Neuroscience.
[17] M. Sipski,et al. High-dose methylprednisolone may cause myopathy in acute spinal cord injury patients , 2005, Spinal Cord.
[18] M. Beattie. Inflammation and apoptosis: linked therapeutic targets in spinal cord injury. , 2004, Trends in molecular medicine.
[19] M. Schwab,et al. Inflammation, degeneration and regeneration in the injured spinal cord: insights from DNA microarrays , 2003, Trends in Neurosciences.
[20] O N Hausmann,et al. Post-traumatic inflammation following spinal cord injury , 2003, Spinal Cord.
[21] L. Noble,et al. Matrix Metalloproteinases Limit Functional Recovery after Spinal Cord Injury by Modulation of Early Vascular Events , 2002, The Journal of Neuroscience.
[22] G. Gores,et al. Ursodeoxycholic acid 'mechanisms of action and clinical use in hepatobiliary disorders'. , 2001, Journal of hepatology.
[23] Gyeong-Moon Kim,et al. Glucocorticoid Receptor-Mediated Suppression of Activator Protein-1 Activation and Matrix Metalloproteinase Expression after Spinal Cord Injury , 2001, The Journal of Neuroscience.
[24] E. Senba,et al. Sequential mRNA expression for immediate early genes, cytokines, and neurotrophins in spinal cord injury. , 2000, Journal of neurotrauma.
[25] M. C. Acosta,et al. Systemically administered interleukin-10 reduces tumor necrosis factor-alpha production and significantly improves functional recovery following traumatic spinal cord injury in rats. , 1999, Journal of neurotrauma.
[26] J. Deleo,et al. Acute peripheral inflammation induces moderate glial activation and spinal IL-1β expression that correlates with pain behavior in the rat 1 Published on the World Wide Web on 17 March 1999. 1 , 1999, Brain Research.
[27] J. Boyer,et al. Ursodeoxycholic acid in cholestasis: Potential mechanisms of action and therapeutic applications , 1998, Hepatology.
[28] J. L. Rodriguez,et al. Consequences of high-dose steroid therapy for acute spinal cord injury. , 1997, The Journal of trauma.
[29] M. Schwab,et al. Methylprednisolone inhibits early inflammatory processes but not ischemic cell death after experimental spinal cord lesion in the rat , 1995, Brain Research.
[30] D. Basso,et al. A sensitive and reliable locomotor rating scale for open field testing in rats. , 1995, Journal of neurotrauma.
[31] F. Zwas,et al. Ursodeoxycholic acid in the treatment of chronic liver disease. , 1994, The American journal of gastroenterology.
[32] A. Hofmann,et al. Ursodeoxycholic acid in the treatment of cholesterol cholelithiasis part II , 1982, Digestive Diseases and Sciences.
[33] A. Nilsson,et al. Ursodeoxycholic acid increases the activities of alkaline sphingomyelinase and caspase-3 in the rat colon. , 1999, Scandinavian journal of gastroenterology.