Maternal blood metal concentrations are associated with C-reactive protein and cell adhesion molecules among pregnant women in Puerto Rico
暂无分享,去创建一个
A. Alshawabkeh | J. Meeker | B. Mukherjee | C. Vélez-Vega | J. Cordero | D. Watkins | A. Cathey | Christine Kim | Deborah J. Watkins | Z. Rosario-Pabón | Amber L. Cathey
[1] A. Baccarelli,et al. Prospective Associations of Early Pregnancy Metal Mixtures with Mitochondria DNA Copy Number and Telomere Length in Maternal and Cord Blood , 2021, Environmental health perspectives.
[2] G. Giskeødegård,et al. Cytokine Patterns in Maternal Serum From First Trimester to Term and Beyond , 2021, Frontiers in Immunology.
[3] P. Villeneuve,et al. Ambient air pollution and inflammatory effects in a Canadian pregnancy cohort , 2021, Environmental Epidemiology.
[4] S. Chemtob,et al. Inflammatory Amplification: A Central Tenet of Uterine Transition for Labor , 2021, Frontiers in Cellular and Infection Microbiology.
[5] J. Wetterö,et al. Plasma C-Reactive Protein and Pentraxin-3 Reference Intervals During Normal Pregnancy , 2021, Frontiers in Immunology.
[6] Min-Sun Kim,et al. Association between heavy metals, high-sensitivity C-reaction protein and 10-year risk of cardiovascular diseases among adult Korean population , 2021, Scientific Reports.
[7] E. Colicino,et al. Early pregnancy exposure to metal mixture and birth outcomes - A prospective study in Project Viva. , 2021, Environment international.
[8] Z. Tahergorabi,et al. Toxic Mechanisms of Five Heavy Metals: Mercury, Lead, Chromium, Cadmium, and Arsenic , 2021, Frontiers in Pharmacology.
[9] S. Biswal,et al. Prenatal Environmental Metal Exposure and Preterm Birth: A Scoping Review , 2021, International journal of environmental research and public health.
[10] G. Pershagen,et al. Longitudinal plasma inflammatory proteome profiling during pregnancy in the Born into Life study , 2020, Scientific Reports.
[11] R. Böger,et al. Male fetal sex is associated with low maternal plasma anti-inflammatory cytokine profile in the first trimester of healthy pregnancies. , 2020, Cytokine.
[12] K. Lo,et al. Environmental heavy metals and cardiovascular diseases: Status and future direction , 2020, Chronic diseases and translational medicine.
[13] A. Alshawabkeh,et al. Maternal blood metal and metalloid concentrations in association with birth outcomes in Northern Puerto Rico. , 2020, Environment international.
[14] R. Nikbakht,et al. Maternal serum levels of C-reactive protein at early pregnancy to predict fetal growth restriction and preterm delivery: A prospective cohort study , 2020, International journal of reproductive biomedicine.
[15] T. Woodruff,et al. Heightened susceptibility: A review of how pregnancy and chemical exposures influence maternal health. , 2020, Reproductive toxicology.
[16] F. Hu,et al. Multiple plasma metals, genetic risk and serum C-reactive protein: A metal-metal and gene-metal interaction study , 2019, Redox biology.
[17] A. D’Alessandro,et al. Male fetus susceptibility to maternal inflammation: C-reactive protein and brain development , 2019, Psychological Medicine.
[18] W. Xia,et al. Prenatal and postnatal cadmium exposure and cellular immune responses among pre-school children. , 2019, Environment international.
[19] C. Burant,et al. Early pregnancy exposure to endocrine disrupting chemical mixtures are associated with inflammatory changes in maternal and neonatal circulation , 2019, Scientific Reports.
[20] F. Montecucco,et al. C-Reactive Protein Levels at the Midpregnancy Can Predict Gestational Complications , 2018, BioMed research international.
[21] Marta Jagodic,et al. Sex-Dependent Impact of Low-Level Lead Exposure during Prenatal Period on Child Psychomotor Functions , 2018, International journal of environmental research and public health.
[22] J. Meeker,et al. Urinary trace metals individually and in mixtures in association with preterm birth. , 2018, Environment international.
[23] I. König,et al. C-Reactive Protein Stimulates Nicotinic Acetylcholine Receptors to Control ATP-Mediated Monocytic Inflammasome Activation , 2018, Front. Immunol..
[24] I. Baranowska-Bosiacka,et al. Lead (Pb) Exposure Enhances Expression of Factors Associated with Inflammation , 2018, International journal of molecular sciences.
[25] F. Albarède,et al. Differential effects of TNF-α and IL-1β on the control of metal metabolism and cadmium-induced cell death in chronic inflammation , 2018, PloS one.
[26] J. Ashworth,et al. Role of C-Reactive Protein at Sites of Inflammation and Infection , 2018, Front. Immunol..
[27] S. Buka,et al. Association of adverse birth outcomes with prenatal exposure to vanadium: a population-based cohort study. , 2017, The Lancet. Planetary health.
[28] K. Kordas,et al. Effects of low-level prenatal lead exposure on child IQ at 4 and 8 years in a UK birth cohort study , 2017, Neurotoxicology.
[29] E. Colicino,et al. Exposure to Low Levels of Lead in Utero and Umbilical Cord Blood DNA Methylation in Project Viva: An Epigenome-Wide Association Study , 2017, Environmental health perspectives.
[30] Y. Fang,et al. Effect of Fetal Sex on Maternal and Obstetric Outcomes , 2017, Front. Pediatr..
[31] P. Tsai,et al. Age-dependent changes in mean and variance of gene expression across tissues in a twin cohort , 2016, bioRxiv.
[32] N. Kaciroti,et al. Low-level prenatal lead exposure and infant sensory function , 2016, Environmental Health.
[33] R. Yolken,et al. Maternal mid-pregnancy C-reactive protein and risk of autism spectrum disorders: the early markers for autism study , 2016, Translational Psychiatry.
[34] J. Schneider,et al. Effects of low level lead exposure on associative learning and memory in the rat: Influences of sex and developmental timing of exposure. , 2016, Toxicology letters.
[35] M. Esteban,et al. Heavy metals produce toxicity, oxidative stress and apoptosis in the marine teleost fish SAF-1 cell line. , 2016, Chemosphere.
[36] J. Meeker,et al. Associations between Maternal Biomarkers of Phthalate Exposure and Inflammation Using Repeated Measurements across Pregnancy , 2015, PloS one.
[37] S. Rifas-Shiman,et al. Very low maternal lead level in pregnancy and birth outcomes in an eastern Massachusetts population. , 2014, Annals of epidemiology.
[38] J. Meeker,et al. Longitudinal Profiling of Inflammatory Cytokines and C‐reactive Protein during Uncomplicated and Preterm Pregnancy , 2014, American journal of reproductive immunology.
[39] S. Markovic,et al. Fetal Sex-Based Differences in Maternal Hormones, Angiogenic Factors, and Immune Mediators During Pregnancy and the Postpartum Period , 2014, American journal of reproductive immunology.
[40] H. Ayatollahi,et al. A Comparative Study of Serum Level of Vascular Cell Adhesion Molecule-1 (sVCAM-1), Intercellular Adhesion Molecule-1(ICAM-1) and High Sensitive C - reactive protein (hs-CRP) in Normal and Pre-eclamptic Pregnancies , 2013, Iranian journal of basic medical sciences.
[41] J. Newnham,et al. Fetal sex and preterm birth. , 2013, Placenta.
[42] A. Hofman,et al. C-reactive protein levels in early pregnancy, fetal growth patterns, and the risk for neonatal complications: the Generation R Study. , 2011, American journal of obstetrics and gynecology.
[43] G. Mor,et al. Inflammation and pregnancy: the role of the immune system at the implantation site , 2011, Annals of the New York Academy of Sciences.
[44] A. Szarka,et al. Circulating cytokines, chemokines and adhesion molecules in normal pregnancy and preeclampsia determined by multiplex suspension array , 2010, BMC Immunology.
[45] U. Sack,et al. Metal ions affecting the immune system. , 2010, Metal ions in life sciences.
[46] M. Vahter,et al. Arsenic-Associated Oxidative Stress, Inflammation, and Immune Disruption in Human Placenta and Cord Blood , 2010, Environmental health perspectives.
[47] V. Clifton. Review: Sex and the human placenta: mediating differential strategies of fetal growth and survival. , 2010, Placenta.
[48] Mina Abbassi-Ghanavati,et al. Pregnancy and Laboratory Studies: A Reference Table for Clinicians , 2009, Obstetrics and gynecology.
[49] W. Ho,et al. Cadmium exposure is associated with elevated blood C-reactive protein and fibrinogen in the U. S. population: the third national health and nutrition examination survey (NHANES III, 1988-1994). , 2009, Annals of epidemiology.
[50] G. Reid,et al. Effect of Lactobacillus rhamnosus GR-1 supernatant and fetal sex on lipopolysaccharide-induced cytokine and prostaglandin-regulating enzymes in human placental trophoblast cells: implications for treatment of bacterial vaginosis and prevention of preterm labor. , 2009, American journal of obstetrics and gynecology.
[51] Young Han Kim,et al. Maternal serum highly sensitive C‐reactive protein in normal pregnancy and pre‐eclampsia , 2007, International journal of gynaecology and obstetrics: the official organ of the International Federation of Gynaecology and Obstetrics.
[52] M. Kibriya,et al. Association between Arsenic Exposure from Drinking Water and Plasma Levels of Soluble Cell Adhesion Molecules , 2007, Environmental health perspectives.
[53] Michelle A. Williams,et al. Maternal serum C-reactive protein concentrations in early pregnancy and subsequent risk of preterm delivery. , 2007, Clinical biochemistry.
[54] C. Salafia,et al. Histologic placental lesions in women with recurrent preterm delivery , 2005, Acta obstetricia et gynecologica Scandinavica.
[55] D. Mergler,et al. Very low level environmental exposure to lead and prolactin levels during pregnancy. , 2005, Neurotoxicology and teratology.
[56] So-Yeon Park,et al. Maternal Serum Levels of VCAM-1, ICAM-1 and E-selectin in Preeclampsia , 2004, Journal of Korean medical science.
[57] Yi-Sook Jung,et al. Cadmium stimulates the expression of ICAM-1 via NF-kappaB activation in cerebrovascular endothelial cells. , 2004, Biochemical and biophysical research communications.
[58] V. Clifton,et al. Maternal asthma as a model for examining fetal sex-specific effects on maternal physiology and placental mechanisms that regulate human fetal growth. , 2004, Placenta.
[59] M. Blankenstein,et al. Elevated C-reactive protein levels during first trimester of pregnancy are indicative of preeclampsia and intrauterine growth restriction. , 2003, Journal of reproductive immunology.
[60] N. Aykin-Burns,et al. Toxic metals and oxidative stress part I: mechanisms involved in metal-induced oxidative damage. , 2001, Current topics in medicinal chemistry.
[61] M. Sans,et al. VCAM-1 and ICAM-1 mediate leukocyte-endothelial cell adhesion in rat experimental colitis. , 1999, Gastroenterology.
[62] S. Parthasarathy,et al. Maternal Serum Vascular Cell Adhesion Molecule Concentration During Pregnancy , 1997, The Journal of the Society for Gynecologic Investigation: JSGI.
[63] D. Wong,et al. Expression of vascular cell adhesion molecule-1 (VCAM-1) by human brain microvessel endothelial cells in primary culture. , 1995, Microvascular research.
[64] E. Baráth,et al. Fundamentals of Biostatistics. , 1992 .
[65] Rose B. Al-Meerany,et al. C-reactive protein as a biochemical marker for idiopathic preterm delivery , 2015 .
[66] E. Marth,et al. The effect of heavy metals on the immune system at low concentrations. , 2001, International journal of occupational medicine and environmental health.
[67] C. Redman,et al. Increased maternal plasma levels of soluble adhesion molecules (ICAM-1, VCAM-1, E-selectin) in preeclampsia. , 1997, European journal of obstetrics, gynecology, and reproductive biology.
[68] P. Oszukowski,et al. [C-reactive protein in normal pregnancy]. , 1996, Ginekologia polska.
[69] Y. Benjamini,et al. Controlling the false discovery rate: a practical and powerful approach to multiple testing , 1995 .