Oxidase Activation in Neutrophils: Role of Hemorrhagic Shock Induces NAD(P)H
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K. Tracey | Huan Yang | Y. Vodovotz | T. Billiar | Jie Fan | Ryan M. Levy | Yuehua Li | Janet J. Fan | M. Wilson | J. David | Huan Yang
[1] J. Hogg. Neutrophil kinetics and lung injury. , 1987, Physiological reviews.
[2] H. Huttunen,et al. Receptor for Advanced Glycation End Products (RAGE)-mediated Neurite Outgrowth and Activation of NF-κB Require the Cytoplasmic Domain of the Receptor but Different Downstream Signaling Pathways* , 1999, The Journal of Biological Chemistry.
[3] L. Levine,et al. Antibodies and radioimmunoassays for phosphoserine, phosphothreonine and phosphotyrosine. Serologic specificities and levels of the phosphoamino acids in cytoplasmic fractions of rat tissues. , 1989, Journal of immunological methods.
[4] I. Douglas,et al. HMGB1 contributes to the development of acute lung injury after hemorrhage. , 2005, American journal of physiology. Lung cellular and molecular physiology.
[5] Nina Bhardwaj,et al. Danger signals: a time and space continuum. , 2004, Trends in molecular medicine.
[6] K. Tracey,et al. Targeting high mobility group box 1 as a late-acting mediator of inflammation. , 2003, Critical care medicine.
[7] Carsten J. Kirschning,et al. HSP70 as Endogenous Stimulus of the Toll/Interleukin-1 Receptor Signal Pathway* , 2002, The Journal of Biological Chemistry.
[8] C. Sette,et al. Peptide-mediated Interference of TIR Domain Dimerization in MyD88 Inhibits Interleukin-1-dependent Activation of NF-κB* , 2005, Journal of Biological Chemistry.
[9] P. Matzinger. The Danger Model: A Renewed Sense of Self , 2002, Science.
[10] M. Gougerot-Pocidalo,et al. The Mitogen-Activated Protein Kinase Extracellular Signal-Regulated Kinase 1/2 Pathway Is Involved in formyl-Methionyl-Leucyl-Phenylalanine-Induced p47phox Phosphorylation in Human Neutrophils1 , 2000, The Journal of Immunology.
[11] H. Park,et al. Cutting Edge: Direct Interaction of TLR4 with NAD(P)H Oxidase 4 Isozyme Is Essential for Lipopolysaccharide-Induced Production of Reactive Oxygen Species and Activation of NF-κB1 , 2004, The Journal of Immunology.
[12] K. Tracey,et al. High Mobility Group 1 Protein (Hmg-1) Stimulates Proinflammatory Cytokine Synthesis in Human Monocytes , 2000, The Journal of experimental medicine.
[13] F. Morel,et al. Phosphorylation of the NADPH oxidase component p67(PHOX) by ERK2 and P38MAPK: selectivity of phosphorylated sites and existence of an intramolecular regulatory domain in the tetratricopeptide-rich region. , 2003, Biochemistry.
[14] M. Bianchi,et al. Chromatin and cell death. , 2004, Biochimica et biophysica acta.
[15] D. Golenbock,et al. TIRAP: how Toll receptors fraternize , 2001, Nature Immunology.
[16] R. Brandes,et al. Vascular NADPH oxidases: molecular mechanisms of activation. , 2005, Cardiovascular research.
[17] M. Yaffe,et al. Distinct Ligand-dependent Roles for p38 MAPK in Priming and Activation of the Neutrophil NADPH Oxidase* , 2004, Journal of Biological Chemistry.
[18] A. Ridley,et al. Receptor for advanced glycation end products-binding COOH-terminal motif of amphoterin inhibits invasive migration and metastasis. , 2002, Cancer research.
[19] T. Miyata,et al. The receptor for advanced glycation end products (RAGE) is a central mediator of the interaction of AGE-beta2microglobulin with human mononuclear phagocytes via an oxidant-sensitive pathway. Implications for the pathogenesis of dialysis-related amyloidosis. , 1996, The Journal of clinical investigation.
[20] M. Nakano,et al. A sensitive and specific chemiluminescence method for estimating the ability of human granulocytes and monocytes to generate O2-. , 1989, Clinica chimica acta; international journal of clinical chemistry.
[21] L. Larivière,et al. Endotoxin-tolerant Mice Have Mutations in Toll-like Receptor 4 (Tlr4) , 1999, The Journal of experimental medicine.
[22] T. Ahrens,et al. Oligosaccharides of Hyaluronan Activate Dendritic Cells via Toll-like Receptor 4 , 2002, The Journal of experimental medicine.
[23] Krishna Shankara Narayanan,et al. Murine Coronavirus Replication-Induced p38 Mitogen-Activated Protein Kinase Activation Promotes Interleukin-6 Production and Virus Replication in Cultured Cells , 2002, Journal of Virology.
[24] Shizuo Akira,et al. Toll-like receptor signalling , 2004, Nature Reviews Immunology.
[25] G. Arteel,et al. Dependence of Liver Injury After Hemorrhage/Resuscitation in Mice on NADPH Oxidase-Derived Superoxide , 2003, Shock.
[26] M. Monden,et al. Involvement of protein phosphatase 2A in PKC‐independent pathway of neutrophil superoxide generation by fMLP , 1996, Journal of cellular biochemistry.
[27] K. Tracey,et al. Reversing established sepsis with antagonists of endogenous high-mobility group box 1 , 2003, Proceedings of the National Academy of Sciences of the United States of America.
[28] N. Patel,et al. INHIBITORS OF NADPH OXIDASE REDUCE THE ORGAN INJURY IN HEMORRHAGIC SHOCK , 2005, Shock.
[29] Ryan M. O’Connell,et al. Cutting Edge: TLR4 Activation Mediates Liver Ischemia/Reperfusion Inflammatory Response via IFN Regulatory Factor 3-Dependent MyD88-Independent Pathway1 , 2004, The Journal of Immunology.
[30] J. El-Benna. The Mitogen-Activated Protein Kinase Extracellular Signal-Regulated Kinase 1/2 Pathway Is Involved in formyl-Methionyl-Leucyl-Phenylalanine-Induced p47 Phosphorylation in Human Neutrophils , 2000 .
[31] Y. Vodovotz,et al. Toll-like receptor-4 signaling mediates hepatic injury and systemic inflammation in hemorrhagic shock. , 2006, Journal of the American College of Surgeons.
[32] P. Ricciardi-Castagnoli,et al. Defective LPS signaling in C3H/HeJ and C57BL/10ScCr mice: mutations in Tlr4 gene. , 1998, Science.
[33] W. Hancock,et al. Fibrinogen Stimulates Macrophage Chemokine Secretion Through Toll-Like Receptor 41 , 2001, The Journal of Immunology.
[34] K. Tracey,et al. Increased serum concentrations of high-mobility-group protein 1 in haemorrhagic shock , 1999, The Lancet.
[35] M. Lotze,et al. Addicted to Death: Invasive Cancer and the Immune Response to Unscheduled Cell Death , 2005, Journal of immunotherapy.
[36] J. Park,et al. Conformational changes of the leukocyte NADPH oxidase subunit p47(phox) during activation studied through its intrinsic fluorescence. , 1998, Biochimica et biophysica acta.
[37] P. Engel,et al. Cutting Edge: MyD88 Controls Phagocyte NADPH Oxidase Function and Killing of Gram-Negative Bacteria1 , 2005, The Journal of Immunology.
[38] M. Yamakuchi,et al. High mobility group box chromosomal protein 1 plays a role in the pathogenesis of rheumatoid arthritis as a novel cytokine. , 2003, Arthritis and rheumatism.
[39] M. J. Cotter,et al. A novel method for isolation of neutrophils from murine blood using negative immunomagnetic separation. , 2001, The American journal of pathology.
[40] John G. Collard,et al. Lysophosphatidic Acid Induces Threonine Phosphorylation of Tiam1 in Swiss 3T3 Fibroblasts via Activation of Protein Kinase C* , 1997, The Journal of Biological Chemistry.
[41] J. Platt,et al. Receptor-Mediated Monitoring of Tissue Well-Being Via Detection of Soluble Heparan Sulfate by Toll-Like Receptor 41 , 2002, The Journal of Immunology.
[42] E. Moore,et al. Postinjury neutrophil priming and activation: an early vulnerable window. , 1995, Surgery.
[43] C. Epstein,et al. Role of superoxide in hemorrhagic shock-induced P-selectin expression. , 2000, American journal of physiology. Heart and circulatory physiology.
[44] J. Palmblad,et al. High mobility group 1 B‐box mediates activation of human endothelium , 2003, Journal of internal medicine.
[45] E. Abraham,et al. Involvement of Toll-like Receptors 2 and 4 in Cellular Activation by High Mobility Group Box 1 Protein* , 2004, Journal of Biological Chemistry.
[46] J. Lambeth. NOX enzymes and the biology of reactive oxygen , 2004, Nature Reviews Immunology.
[47] Y. Vodovotz,et al. Hemorrhagic shock-activated neutrophils augment TLR4 signaling-induced TLR2 upregulation in alveolar macrophages: role in hemorrhage-primed lung inflammation. , 2006, American journal of physiology. Lung cellular and molecular physiology.
[48] Kevin J. Tracey,et al. High-mobility group box 1 protein (HMGB1): nuclear weapon in the immune arsenal , 2005, Nature Reviews Immunology.
[49] Polly Matzinger,et al. Hydrophobicity: an ancient damage-associated molecular pattern that initiates innate immune responses , 2004, Nature Reviews Immunology.
[50] Kuan-Teh Jeang,et al. Human T Cell Leukemia Virus Type 1 Oncoprotein Tax Targets the Human Mitotic Checkpoint Protein MAD1 , 1998, Cell.
[51] M. Högström,et al. Methods in laboratory investigation. Exclusion of trypan blue from microcarriers by endothelial cells: an in vitro barrier function test. , 1984, Laboratory investigation; a journal of technical methods and pathology.
[52] K. Tracey,et al. HMGB1 as a late mediator of lethal systemic inflammation. , 2001, American journal of respiratory and critical care medicine.
[53] K. Tracey,et al. HMG-1 as a late mediator of endotoxin lethality in mice. , 1999, Science.
[54] E. Abraham,et al. High mobility group box 1 protein interacts with multiple Toll-like receptors. , 2006, American journal of physiology. Cell physiology.
[55] K. Tracey,et al. The nuclear factor HMGB1 mediates hepatic injury after murine liver ischemia-reperfusion , 2005, The Journal of experimental medicine.
[56] Takeshi Imamura,et al. Smad proteins exist as monomers in vivo and undergo homo‐ and hetero‐oligomerization upon activation by serine/threonine kinase receptors , 1998, The EMBO journal.
[57] L. K. Nelson,et al. Analysis of Activation-induced Conformational Changes in p47 phox Using Tryptophan Fluorescence Spectroscopy* , 1997, The Journal of Biological Chemistry.
[58] U. Andersson,et al. Mini‐review: The nuclear protein HMGB1 as a proinflammatory mediator , 2004, European journal of immunology.
[59] G. Camussi,et al. CD40-dependent Activation of Phosphatidylinositol 3-Kinase/Akt Pathway Mediates Endothelial Cell Survival and in Vitro Angiogenesis* , 2003, The Journal of Biological Chemistry.
[60] J. A. Badwey,et al. Reciprocal interactions between protein kinase C and components of the NADPH oxidase complex may regulate superoxide production by neutrophils stimulated with a phorbol ester. , 1994, The Journal of biological chemistry.
[61] K. Rittinger,et al. Activation and assembly of the NADPH oxidase: a structural perspective. , 2005, The Biochemical journal.