RNA-Based next generation sequencing complements but does not replace fluorescence in situ hybridization studies for the classification of aggressive B-Cell lymphomas.
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[1] K. Elenitoba-Johnson,et al. Impact of Genetics on Mature Lymphoid Leukemias and Lymphomas. , 2020, Cold Spring Harbor perspectives in medicine.
[2] Qin Huang,et al. High-Grade B-Cell Lymphoma , 2020, Practical Lymph Node and Bone Marrow Pathology.
[3] S. Bens,et al. Cryptic insertion of MYC exons 2 and 3 into the immunoglobulin heavy chain locus detected by whole genome sequencing in a case of “MYC-negative” Burkitt lymphoma , 2020, Haematologica.
[4] J. Choi,et al. Detection of recurrent, rare, and novel gene fusions in patients with acute leukemia using next‐generation sequencing approaches , 2019, Hematological oncology.
[5] E. Severson,et al. Comparison Between Integrated Genomic DNA/RNA Profiling and Fluorescence In Situ Hybridization in the Detection of MYC, BCL-2, and BCL-6 Gene Rearrangements in Large B-Cell Lymphomas. , 2019, American journal of clinical pathology.
[6] Ryan D. Morin,et al. --The double hit signature identifies double-hit diffuse large B-cell lymphoma with genetic events cryptic to FISH. , 2019, Blood.
[7] C. Mullighan,et al. The Biology of B-Progenitor Acute Lymphoblastic Leukemia. , 2019, Cold Spring Harbor perspectives in medicine.
[8] David M. Thomas,et al. Diagnosis of fusion genes using targeted RNA sequencing , 2019, Nature Communications.
[9] J. Choi,et al. Clinical Evaluation of Massively Parallel RNA Sequencing for Detecting Recurrent Gene Fusions in Hematologic Malignancies. , 2019, The Journal of molecular diagnostics : JMD.
[10] J. Cook,et al. Identification of “Double Hit” Lymphomas Using Updated WHO Criteria: Insights From Routine MYC Immunohistochemistry in 272 Consecutive Cases of Aggressive B-Cell Lymphomas , 2019, Applied immunohistochemistry & molecular morphology : AIMM.
[11] L. Rimsza,et al. Genomics of aggressive B-cell lymphoma. , 2018, Hematology. American Society of Hematology. Education Program.
[12] J. Cortes,et al. Acute myeloid leukaemia , 2018, The Lancet.
[13] Asha A. Nair,et al. Recurrent STAT3-JAK2 fusions in indolent T-cell lymphoproliferative disorder of the gastrointestinal tract. , 2018, Blood.
[14] Saijuan Chen,et al. Identification of fusion genes and characterization of transcriptome features in T-cell acute lymphoblastic leukemia , 2017, Proceedings of the National Academy of Sciences.
[15] J. Passweg,et al. Acute myeloid leukaemia genomics , 2017, British journal of haematology.
[16] G. Ott. Aggressive B‐cell lymphomas in the update of the 4th edition of the World Health Organization classification of haematopoietic and lymphatic tissues: refinements of the classification, new entities and genetic findings , 2017, British journal of haematology.
[17] O. Abdel-Wahab,et al. Diagnosis and classification of hematologic malignancies on the basis of genetics. , 2017, Blood.
[18] A. Feldman,et al. Lymphoma classification update: B-cell non-Hodgkin lymphomas , 2017, Expert review of hematology.
[19] Lars Bullinger,et al. Genomics of Acute Myeloid Leukemia Diagnosis and Pathways. , 2017, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[20] Y. Denizot,et al. The IgH 3’ regulatory region and c-myc-induced B-cell lymphomagenesis , 2016, Oncotarget.
[21] Rosamaria Pinto,et al. Next-generation sequencing: advances and applications in cancer diagnosis , 2016, OncoTargets and therapy.
[22] Angie Duy Vo,et al. Identifying fusion transcripts using next generation sequencing , 2016, Wiley interdisciplinary reviews. RNA.
[23] J. Carpten,et al. Translating RNA sequencing into clinical diagnostics: opportunities and challenges , 2016, Nature Reviews Genetics.
[24] Lauren C. Chong,et al. Genomic Alterations in CIITA Are Frequent in Primary Mediastinal Large B Cell Lymphoma and Are Associated with Diminished MHC Class II Expression. , 2015, Cell reports.
[25] B. Coiffier,et al. Double-hit and double-protein-expression lymphomas: aggressive and refractory lymphomas. , 2015, The Lancet. Oncology.
[26] K. Savage,et al. The biology and management of systemic anaplastic large cell lymphoma. , 2015, Blood.
[27] Laura Pasqualucci,et al. The genetic landscape of diffuse large B-cell lymphoma. , 2015, Seminars in hematology.
[28] A. Feldman,et al. Anaplastic Large Cell Lymphomas: ALK Positive, ALK Negative, and Primary Cutaneous , 2015, Advances in anatomic pathology.
[29] S. Swerdlow. Diagnosis of 'double hit' diffuse large B-cell lymphoma and B-cell lymphoma, unclassifiable, with features intermediate between DLBCL and Burkitt lymphoma: when and how, FISH versus IHC. , 2014, Hematology. American Society of Hematology. Education Program.
[30] Jie Zheng. Oncogenic chromosomal translocations and human cancer (review). , 2013, Oncology reports.
[31] A. Ariza,et al. MYC status determination in aggressive B‐cell lymphoma: the impact of FISH probe selection , 2013, Histopathology.
[32] L. Medeiros,et al. B-cell lymphomas with MYC/8q24 rearrangements and IGH@BCL2/t(14;18)(q32;q21): an aggressive disease with heterogeneous histology, germinal center B-cell immunophenotype and poor outcome , 2012, Modern Pathology.
[33] M. Neat,et al. Fluorescence in situ hybridisation analysis of formalin-fixed paraffin-embedded tissue sections in the diagnostic work-up of non-Burkitt high grade B-cell non-Hodgkin's lymphoma: a single centre's experience , 2011, Journal of Clinical Pathology.
[34] Steven J. M. Jones,et al. MHC class II transactivator CIITA is a recurrent gene fusion partner in lymphoid cancers , 2011, Nature.
[35] Matija Snuderl,et al. B-cell Lymphomas With Concurrent IGH-BCL2 and MYC Rearrangements Are Aggressive Neoplasms With Clinical and Pathologic Features Distinct From Burkitt Lymphoma and Diffuse Large B-cell Lymphoma , 2010, The American journal of surgical pathology.
[36] F. Jardin,et al. Promoter Shuffling by Sequential Genomic Rearrangements in Follicular Lymphoma Reveals an Ongoing Genomic Instability at the BCL6 Locus. , 2006 .
[37] Takashi Akasaka,et al. BCL6 gene translocation in follicular lymphoma: a harbinger of eventual transformation to diffuse aggressive lymphoma. , 2003, Blood.
[38] Riccardo Dalla-Favera,et al. Mechanisms of chromosomal translocations in B cell lymphomas , 2001, Oncogene.
[39] G. Srivastava,et al. Identification and characterization of BCL6 translocation partner genes in primary gastric high‐grade B‐cell lymphoma: Heat shock protein 89 alpha is a novel fusion partner gene of BCL6 , 2000, Genes, chromosomes & cancer.
[40] M. Seto,et al. Identification of heterologous translocation partner genes fused to the BCL6 gene in diffuse large B-cell lymphomas: 5′-RACE and LA – PCR analyses of biopsy samples , 1999, Oncogene.