Prediction of Outcome After Endovascular Embolectomy in Anterior Circulation Stroke Using Biomarkers
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K. Blennow | H. Zetterberg | E. Portelius | L. Rosengren | J. Karlsson | K. Höglund | Fani Pujol-Calderón | P. Löwhagen Hendén | A. Rentzos
[1] A. Privat,et al. GFAP null astrocytes are a favorable substrate for neuronal survival and neurite growth , 2000, Glia.
[2] K. Blennow,et al. Normal levels of clusterin in cerebrospinal fluid in Alzheimer's disease, and no change after acute ischemic stroke. , 2001, Journal of Alzheimer's disease : JAD.
[3] M. Kirschner,et al. A protein factor essential for microtubule assembly. , 1975, Proceedings of the National Academy of Sciences of the United States of America.
[4] R. Fukuyama,et al. The Cerebrospinal Fluid Level of Glial Fibrillary Acidic Protein Is Increased in Cerebrospinal Fluid from Alzheimer’s Disease Patients and Correlates with Severity of Dementia , 2001, European Neurology.
[5] K. Blennow,et al. Serum neurofilament light as a biomarker for mild traumatic brain injury in contact sports , 2017, Neurology.
[6] F. Michetti,et al. Immunochemical and immunocytochemical localization of S-100 antigen in normal human skin , 1981, Nature.
[7] O. Sogut,et al. Trauma Scores and Neuron-Specific Enolase, Cytokine and C-Reactive Protein Levels as Predictors of Mortality in Patients with Blunt Head Trauma , 2010, The Journal of international medical research.
[8] Fares Alahdab,et al. Global, regional, and national burden of stroke, 1990–2016: a systematic analysis for the Global Burden of Disease Study 2016 , 2019, The Lancet Neurology.
[9] P. Bottoni,et al. Neuron-Specific Enolase as a Biomarker: Biochemical and Clinical Aspects. , 2015, Advances in experimental medicine and biology.
[10] B. Moore. A soluble protein characteristic of the nervous system. , 1965, Biochemical and biophysical research communications.
[11] A. Demchuk,et al. Endovascular thrombectomy after large-vessel ischaemic stroke: a meta-analysis of individual patient data from five randomised trials , 2016, The Lancet.
[12] H. Nawashiro,et al. Mice lacking GFAP are hypersensitive to traumatic cerebrospinal injury , 1998, Neuroreport.
[13] Henrik Zetterberg,et al. Levels and Age Dependency of Neurofilament Light and Glial Fibrillary Acidic Protein in Healthy Individuals and Their Relation to the Brain Parenchymal Fraction , 2015, PloS one.
[14] J. Sobesky,et al. Prognostic value of somatosensory evoked potentials, neuron-specific enolase, and S100 for short-term outcome in ischemic stroke. , 2016, Journal of neurophysiology.
[15] K. Blennow,et al. Plasma and cerebrospinal fluid tau and neurofilament concentrations in rapidly progressive neurological syndromes: a neuropathology‐based cohort , 2017, European journal of neurology.
[16] A. González-Quevedo,et al. Short-term prognostic value of serum neuron specific enolase and S100B in acute stroke patients. , 2012, Clinical biochemistry.
[17] R. Perrot,et al. Neuronal intermediate filaments and neurodegenerative disorders , 2009, Brain Research Bulletin.
[18] R. Hayes,et al. Assessment of Serum UCH-L1 and GFAP in Acute Stroke Patients , 2016, Scientific Reports.
[19] J. Cronly-Dillon,et al. Preferential histochemical staining of protoplasmic and fibrous astrocytes in rat CNS with GFAP antibodies using different fixatives , 1990, Brain Research.
[20] K. Blennow,et al. Neurofilament changes in serum and cerebrospinal fluid after acute ischemic stroke , 2019, Neuroscience Letters.
[21] G. Davies-Jones,et al. Cerebrospinal fluid enolase in stroke. , 1984, Journal of neurology, neurosurgery, and psychiatry.
[22] K. Muir,et al. How many stroke patients might be eligible for mechanical thrombectomy? , 2016, European stroke journal.
[23] C. Price,et al. Estimating the number of UK stroke patients eligible for endovascular thrombectomy , 2017, European stroke journal.
[24] D. DeMets,et al. Biomarkers and surrogate endpoints: Preferred definitions and conceptual framework , 2001, Clinical pharmacology and therapeutics.
[25] M. Sitzer,et al. Characterisation of the Diagnostic Window of Serum Glial Fibrillary Acidic Protein for the Differentiation of Intracerebral Haemorrhage and Ischaemic Stroke , 2008, Cerebrovascular Diseases.
[26] K. Blennow,et al. Journal of Central Nervous System Disease , 2022 .
[27] J. Silver,et al. Glial fibrillary acidic protein is necessary for mature astrocytes to react to beta-amyloid. , 1999, Glia.
[28] M. Deckert,et al. The Intermediate Filament GFAP Is Important for the Control of Experimental Murine Staphylococcus aureus‐Induced Brain Abscess and Toxoplasma Encephalitis , 2004, Journal of neuropathology and experimental neurology.
[29] A. Demchuk,et al. Use of the Alberta Stroke Program Early CT Score (ASPECTS) for assessing CT scans in patients with acute stroke. , 2001, AJNR. American journal of neuroradiology.
[30] K. Blennow,et al. Neurogranin and tau in cerebrospinal fluid and plasma of patients with acute ischemic stroke , 2017, BMC Neurology.
[31] G. Jullien,et al. Therapeutic application of the molecular adsorbent recirculating system in chronic severe hepatitis patients complicated with multiorgan failure , 2005, Critical Care.
[32] L. Rosengren,et al. Glial fibrillary acidic protein: a potential biomarker for progression in multiple sclerosis , 2010, Journal of Neurology.
[33] K. Blennow,et al. Cerebrospinal fluid markers for Alzheimer's disease evaluated after acute ischemic stroke. , 2000, Journal of Alzheimer's disease : JAD.
[34] J. Noth,et al. Gradual loss of myelin and formation of an astrocytic scar during Wallerian degeneration in the human spinal cord. , 2004, Brain : a journal of neurology.
[35] M. Oblinger,et al. Regional distribution and biochemical characteristics of high molecular weight tau in the nervous system , 1992, Journal of neuroscience research.
[36] R. Thompson,et al. Levels of immunoreactive aldolase C, creatine kinase-BB, neuronal and non-neuronal enolase, and 14-3-3 protein in circulating human blood cells. , 1984, Clinica chimica acta; international journal of clinical chemistry.
[37] C. Heizmann,et al. S100 proteins in mouse and man: from evolution to function and pathology (including an update of the nomenclature). , 2004, Biochemical and biophysical research communications.
[38] L. Schneck. Biochemical and Clinical Aspects , 1975 .
[39] S. Mckinstry,et al. Serum neurone‐specific enolase as an indicator of stroke volume , 1996, European journal of clinical investigation.
[40] K. Blennow,et al. Cerebrospinal fluid neurofilament light concentration in motor neuron disease and frontotemporal dementia predicts survival , 2017, Amyotrophic lateral sclerosis & frontotemporal degeneration.
[41] M. Schluep,et al. Glial and axonal body fluid biomarkers are related to infarct volume, severity, and outcome. , 2008, Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association.
[42] N. Kowall,et al. Immunohistochemical patterns of selective cellular vulnerability in human cerebral ischemia , 1993, Journal of the Neurological Sciences.
[43] Volkmar Glauche,et al. Diffusion tensor imaging detects early Wallerian degeneration of the pyramidal tract after ischemic stroke , 2004, NeuroImage.
[44] M. Brenner. Role of GFAP in CNS injuries , 2014, Neuroscience Letters.
[45] B. Romner,et al. Biochemical serum markers for brain damage: a short review with emphasis on clinical utility in mild head injury. , 2003, Restorative neurology and neuroscience.
[46] L. Hagberg,et al. Difference in pathogenesis between herpes simplex virus type 1 encephalitis and tick-borne encephalitis demonstrated by means of cerebrospinal fluid markers of glial and neuronal destruction , 2000, Journal of Neurology.
[47] G. D. De Marchis,et al. Serum neurofilament light chain in patients with acute cerebrovascular events , 2018, European journal of neurology.
[48] J. Silver,et al. Glial fibrillary acidic protein is necessary for mature astrocytes to react to β‐amyloid , 1999 .
[49] Ji‐Hyung Park,et al. Prediction of early clinical severity and extent of neuronal damage in anterior-circulation infarction using the initial serum neuron-specific enolase level. , 2003, Archives of neurology.
[50] P. Carmeliet,et al. Protective Role of Reactive Astrocytes in Brain Ischemia , 2008, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[51] Lynn Babcock,et al. Are UCH-L1 and GFAP promising biomarkers for children with mild traumatic brain injury? , 2016, Brain injury.
[52] Thomas Brott,et al. The impact of imbalances in baseline stroke severity on outcome in the National Institute of Neurological Disorders and Stroke Recombinant Tissue Plasminogen Activator Stroke Study. , 2005, Annals of emergency medicine.
[53] E. Mandelkow,et al. Tau blocks traffic of organelles, neurofilaments, and APP vesicles in neurons and enhances oxidative stress , 2002, The Journal of cell biology.
[54] Leslie Wilson,et al. Inability of tau to properly regulate neuronal microtubule dynamics: a loss-of-function mechanism by which tau might mediate neuronal cell death. , 2005, Biochimica et biophysica acta.
[55] J. Kacza,et al. Decline of microtubule-associated protein tau after experimental stroke in differently aged wild-type and 3xTg mice with Alzheimer-like alterations , 2016, Neuroscience.
[56] F. Goodwin,et al. Neuronal, non-neuronal and hybrid forms of enolase in brain: Structural, immunological and functional comparisons , 1978, Brain Research.
[57] M. Seipelt,et al. Serum Tau Protein Level as a Marker of Axonal Damage in Acute Ischemic Stroke , 2002, European Neurology.
[58] M. Skalej,et al. Neuron-specific enolase and tau protein as neurobiochemical markers of neuronal damage are related to early clinical course and long-term outcome in acute ischemic stroke , 2006, Clinical Neurology and Neurosurgery.
[59] R. Liem,et al. α-Internexin and Peripherin: Expression, Assembly, Functions, and Roles in Disease. , 2016, Methods in enzymology.
[60] M. Brown,et al. Criteria for a Clinically Informative Serum Biomarker in Acute Ischaemic Stroke: A Review of S100B , 2009, Cerebrovascular Diseases.
[61] L. Rosengren,et al. Neurofilament protein in cerebrospinal fluid: a potential marker of activity in multiple sclerosis , 1998, Journal of neurology, neurosurgery, and psychiatry.
[62] P. Carmeliet,et al. Under stress, the absence of intermediate filaments from Müller cells in the retina has structural and functional consequences , 2004, Journal of Cell Science.
[63] I. Bechmann,et al. Up-regulation of neurofilament light chains is associated with diminished immunoreactivities for MAP2 and tau after ischemic stroke in rodents and in a human case , 2016, Journal of Chemical Neuroanatomy.
[64] M. Hellström,et al. General Anesthesia Versus Conscious Sedation for Endovascular Treatment of Acute Ischemic Stroke: The AnStroke Trial (Anesthesia During Stroke) , 2017, Stroke.
[65] T. Crawford,et al. Subunit composition of neurofilaments specifies axonal diameter , 1996, The Journal of cell biology.
[66] K. Haglid,et al. Determination of S‐100 and Glial Fibrillary Acidic Protein Concentrations in Cerebrospinal Fluid After Brain Infarction , 1991, Stroke.
[67] M. Brown,et al. Hyperacute Detection of Neurofilament Heavy Chain in Serum Following Stroke: A Transient Sign , 2011, Neurochemical Research.
[68] F. Beyersdorf,et al. Neuron-specific enolase correlates to laboratory markers of haemolysis in patients on long-term circulatory support. , 2015, European journal of cardio-thoracic surgery : official journal of the European Association for Cardio-thoracic Surgery.
[69] E. Mandelkow,et al. Tau in physiology and pathology , 2015, Nature Reviews Neuroscience.
[70] K. Blennow,et al. Serum neurofilament light protein predicts clinical outcome in traumatic brain injury , 2016, Scientific Reports.
[71] J. Broderick,et al. Association of Serial Biochemical Markers With Acute Ischemic Stroke: The National Institute of Neurological Disorders and Stroke Recombinant Tissue Plasminogen Activator Stroke Study , 2006, Stroke.
[72] Henrik Zetterberg,et al. Comparison of three analytical platforms for quantification of the neurofilament light chain in blood samples: ELISA, electrochemiluminescence immunoassay and Simoa , 2016, Clinical chemistry and laboratory medicine.
[73] Henrik Zetterberg,et al. Association of Cerebrospinal Fluid Neurofilament Light Concentration With Alzheimer Disease Progression. , 2016, JAMA neurology.
[74] M. L. Schmidt,et al. Distribution of tau proteins in the normal human central and peripheral nervous system. , 1989, The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society.
[75] E. Baudin,et al. Neuron-specific enolase and chromogranin A as markers of neuroendocrine tumours. , 1998, British Journal of Cancer.
[76] C. Wikkelsø,et al. Patients with Amyotrophic Lateral Sclerosis and Other Neurodegenerative Diseases Have Increased Levels of Neurofilament Protein in CSF , 1996, Journal of neurochemistry.
[77] V. Seifert,et al. Serum S-100B protein as a molecular marker in severe traumatic brain injury. , 2003, Restorative neurology and neuroscience.