Circulating immune complexes of Aβ and IgM in plasma of patients with Alzheimer’s disease
暂无分享,去创建一个
T. Schneider-Axmann | T. Bayer | L. Lannfelt | O. Wirths | M. Degerman-Gunnarsson | Andrea Marcello | Malin Degerman-Gunnarsson | A. Marcello
[1] M. Folstein,et al. Clinical diagnosis of Alzheimer's disease: Report of the NINCDS—ADRDA Work Group under the auspices of Department of Health and Human Services Task Force on Alzheimer's Disease , 2011, Neurology.
[2] T. Schneider-Axmann,et al. Effect of copper intake on CSF parameters in patients with mild Alzheimer’s disease: a pilot phase 2 clinical trial , 2008, Journal of Neural Transmission.
[3] P. Pontisso,et al. Tumour‐specific induction of immune complexes: DCP‐IgM in hepatocellular carcinoma , 2008, European journal of clinical investigation.
[4] E. Matsubara,et al. Plasma antibodies to Aβ40 and Aβ42 in patients with Alzheimer's disease and normal controls , 2008, Brain Research.
[5] M. Shoji,et al. Plasma biomarkers of Alzheimer's disease , 2008, Current opinion in psychiatry.
[6] B. Hyman,et al. Plasma Amyloid and the Risk of Alzheimer Disease and Dementia in Elderly Men , 2008 .
[7] B. Hyman,et al. The normal equilibrium between CSF and plasma amyloid beta levels is disrupted in Alzheimer's disease , 2007, Neuroscience Letters.
[8] U. Nydegger. Immune Complex Pathophysiology , 2007, Annals of the New York Academy of Sciences.
[9] R. Sewell,et al. Differential neuroimmune markers to the onset of Alzheimer's disease neurodegeneration and dementia: Autoantibodies to Aβ(25–35) oligomers, S100b and neurotransmitters , 2007, Journal of Neuroimmunology.
[10] R. Petersen,et al. Association of Low Plasma Aβ42/Aβ40 Ratios With Increased Imminent Risk for Mild Cognitive Impairment and Alzheimer Disease , 2007 .
[11] M. Yoshita,et al. Blood-Brain Barrier Permeability Correlates with Medial Temporal Lobe Atrophy but Not with Amyloid-β Protein Transport across the Blood-Brain Barrier in Alzheimer’s Disease , 2007, Dementia and Geriatric Cognitive Disorders.
[12] A. Hofman,et al. Plasma Aβ1–40 and Aβ1–42 and the risk of dementia: a prospective case-cohort study , 2006, The Lancet Neurology.
[13] C. Mariani,et al. Plasma levels of beta-amyloid (1–42) in Alzheimer's disease and mild cognitive impairment , 2006, Neurobiology of Aging.
[14] P. Mehta,et al. Increased Cerebrospinal Fluid Aβ38/Aβ42 Ratio in Alzheimer Disease , 2006, Neurodegenerative Diseases.
[15] C. Belluco,et al. Detection of Circulating CEA-IgM Complexes in Early Stage Colorectal Cancer , 2005, The International journal of biological markers.
[16] J. Sidtis,et al. Selective reductions in plasma Abeta 1-42 in healthy elderly subjects during longitudinal follow-up: a preliminary report. , 2005, The American journal of geriatric psychiatry : official journal of the American Association for Geriatric Psychiatry.
[17] P. Pontisso,et al. Squamous cell carcinoma antigen‐immunoglobulin M complexes as novel biomarkers for hepatocellular carcinoma , 2005, Cancer.
[18] N. Bogdanovic,et al. Human monoclonal antibodies against amyloid-beta from healthy adults , 2005, Neurobiology of Aging.
[19] C. Jack,et al. Mild cognitive impairment – beyond controversies, towards a consensus: report of the International Working Group on Mild Cognitive Impairment , 2004, Journal of internal medicine.
[20] R. Sewell,et al. Autoimmune Responses to Amyloid Structures of Aβ(25–35) Peptide and Human Lysozyme in the Serum of Patients with Progressive Alzheimer’s Disease , 2004, Dementia and Geriatric Cognitive Disorders.
[21] Kaj Blennow,et al. Cerebrospinal fluid protein biomarkers for Alzheimer’s disease , 2004, NeuroRX.
[22] P. Pontisso,et al. Improvement of Liver Cancer Detection with Simultaneous Assessment of Circulating Levels of Free Alpha-Fetoprotein (AFP) and Afp-Igm Complexes , 2004, The International journal of biological markers.
[23] Juan Manuel Maler,et al. Neurochemical diagnosis of Alzheimer’s dementia by CSF Aβ42, Aβ42/Aβ40 ratio and total tau , 2004, Neurobiology of Aging.
[24] K. Blennow,et al. Intrathecal inflammation precedes development of Alzheimer’s disease , 2003, Journal of neurology, neurosurgery, and psychiatry.
[25] J. Growdon,et al. Age but Not Diagnosis Is the Main Predictor of Plasma Amyloid β-Protein Levels , 2003 .
[26] K. Henke,et al. Antibodies against β-Amyloid Slow Cognitive Decline in Alzheimer's Disease , 2003, Neuron.
[27] H. Möller,et al. Progressive loss of cardiac sympathetic innervation in Parkinson's disease , 2002, Annals of neurology.
[28] N. Relkin,et al. Patients with Alzheimer disease have lower levels of serum anti-amyloid peptide antibodies than healthy elderly individuals , 2002, Experimental Gerontology.
[29] E. Masliah,et al. Absence of elevated anti–&agr;-synuclein and anti-EBV latent membrane protein antibodies in PD , 2002, Neurology.
[30] Patrick L. McGeer,et al. Inflammation, autotoxicity and Alzheimer disease , 2001, Neurobiology of Aging.
[31] W. Oertel,et al. Reduced levels of amyloid β-peptide antibody in Alzheimer disease , 2001, Neurology.
[32] David J. Cummins,et al. Peripheral anti-Aβ antibody alters CNS and plasma Aβ clearance and decreases brain Aβ burden in a mouse model of Alzheimer's disease , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[33] Y. Ihara,et al. Alzheimer β Amyloid Deposition Enhanced by ApoE ϵ4 Gene Precedes Neurofibrillary Pathology in the Frontal Association Cortex of Nondemented Senior Subjects , 2001 .
[34] B. Hyman,et al. Autoantibodies to amyloid‐β and Alzheimer's disease , 2001, Annals of neurology.
[35] P. Mehta,et al. Amyloid β protein 1–40 and 1–42 levels in matched cerebrospinal fluid and plasma from patients with Alzheimer disease , 2001, Neuroscience Letters.
[36] K. Blennow,et al. Evaluation of CSF-tau and CSF-Abeta42 as diagnostic markers for Alzheimer disease in clinical practice. , 2001, Archives of neurology.
[37] Hilkka Soininen,et al. Relationship between apoE genotype and CSF β-amyloid (1–42) and tau in patients with probable and definite Alzheimer’s disease , 2000, Neurobiology of Aging.
[38] D. Galasko,et al. Amyloid-beta peptides interact with plasma proteins and erythrocytes: implications for their quantitation in plasma. , 2000, Biochemical and biophysical research communications.
[39] K. Blennow,et al. Standardization of measurement of β-amyloid(1-42) in cerebrospinal fluid and plasma , 2000, Amyloid : the international journal of experimental and clinical investigation : the official journal of the International Society of Amyloidosis.
[40] B Engvall,et al. Cerebrospinal fluid Aβ42 is increased early in sporadic Alzheimer's disease and declines with disease progression , 1999, Annals of neurology.
[41] E. Matsubara,et al. Lipoprotein‐free amyloidogenic peptides in plasma are elevated in patients with sporadic Alzheimer's disease and Down's syndrome , 1999, Annals of neurology.
[42] J. Morris,et al. Tangles and plaques in nondemented aging and “preclinical” Alzheimer's disease , 1999, Annals of neurology.
[43] J. Kaye,et al. High cerebrospinal fluid tau and low amyloid beta42 levels in the clinical diagnosis of Alzheimer disease and relation to apolipoprotein E genotype. , 1998, Archives of neurology.
[44] S. Hirai,et al. Longitudinal study of cerebrospinal fluid levels of tau, Aβ1–40, and Aβ1–42(43) in Alzheimer's disease: A study in Japan , 1998 .
[45] J. Trojanowski,et al. A new consensus report on biomarkers for the early antemortem diagnosis of Alzheimer disease: current status, relevance to drug discovery, and recommendations for future research. , 1998, Journal of neuropathology and experimental neurology.
[46] F. Gaskin,et al. Increased incidence of anti-β-amyloid autoantibodies secreted by Epstein-Barr virus transformed B cell lines from patients with Alzheimer's disease , 1997, Mechanisms of Ageing and Development.
[47] S. Tsuji,et al. The beta APP717 Alzheimer mutation increases the percentage of plasma amyloid-beta protein ending at A beta 42(43) , 1997, Neurology.
[48] B. Hyman,et al. Amyloid β-Peptide Is Transported on Lipoproteins and Albumin in Human Plasma* , 1996, The Journal of Biological Chemistry.
[49] E. Oguni,et al. Amyloid β protein in plasma from patients with sporadic Alzheimer's disease , 1996, Journal of the Neurological Sciences.
[50] H. Lassmann,et al. APP peptides stimulate lymphocyte proliferation in normals, but not in patients with Alzheimer's Disease , 1996, Neurobiology of Aging.
[51] S. Younkin,et al. An increased percentage of long amyloid beta protein secreted by familial amyloid beta protein precursor (beta APP717) mutants. , 1994, Science.
[52] J. Hardy,et al. The Amyloid Hypothesis of Alzheimer ’ s Disease : Progress and Problems on the Road to Therapeutics , 2009 .
[53] B. Hyman,et al. Plasma beta amyloid and the risk of Alzheimer disease and dementia in elderly men: a prospective, population-based cohort study. , 2008, Archives of neurology.
[54] R. Petersen,et al. Association of low plasma Abeta42/Abeta40 ratios with increased imminent risk for mild cognitive impairment and Alzheimer disease. , 2007, Archives of neurology.
[55] G. Fassina,et al. Detection of prostate-specific antigen coupled to immunoglobulin M in prostate cancer patients. , 2007, Cancer detection and prevention.
[56] Charles D. Smith,et al. Autoantibodies to amyloid β-peptide (Aβ) are increased in Alzheimer’s disease patients and Aβ antibodies can enhance Aβ neurotoxicity , 2007, NeuroMolecular Medicine.
[57] A. Hofman,et al. Plasma Abeta(1-40) and Abeta(1-42) and the risk of dementia: a prospective case-cohort study. , 2006, The Lancet. Neurology.
[58] P. Mehta,et al. Increased cerebrospinal fluid A beta38/A beta42 ratio in Alzheimer disease. , 2005, Neuro-degenerative diseases.
[59] P. Lewczuk,et al. Neurochemical diagnosis of Alzheimer's dementia by CSF Abeta42, Abeta42/Abeta40 ratio and total tau. , 2004, Neurobiology of aging.
[60] Charles D. Smith,et al. Autoantibodies to amyloid beta-peptide (Abeta) are increased in Alzheimer's disease patients and Abeta antibodies can enhance Abeta neurotoxicity: implications for disease pathogenesis and vaccine development. , 2003, Neuromolecular medicine.
[61] J. Growdon,et al. Age but not diagnosis is the main predictor of plasma amyloid beta-protein levels. , 2003, Archives of neurology.
[62] K. Henke,et al. Antibodies against beta-amyloid slow cognitive decline in Alzheimer's disease. , 2003, Neuron.
[63] Y. Ihara,et al. Alzheimer beta amyloid deposition enhanced by apoE epsilon4 gene precedes neurofibrillary pathology in the frontal association cortex of nondemented senior subjects. , 2001, Journal of neuropathology and experimental neurology.
[64] D. Holtzman,et al. Peripheral anti-A beta antibody alters CNS and plasma A beta clearance and decreases brain A beta burden in a mouse model of Alzheimer's disease. , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[65] P. Mehta,et al. Amyloid beta protein 1-40 and 1-42 levels in matched cerebrospinal fluid and plasma from patients with Alzheimer disease. , 2001, Neuroscience letters.
[66] H. Wiśniewski,et al. Plasma and cerebrospinal fluid Levels of amyloid β proteins 1-40 and 1-42 in Alzheimer disease , 2000 .
[67] S. Hirai,et al. Longitudinal study of cerebrospinal fluid levels of tau, A beta1-40, and A beta1-42(43) in Alzheimer's disease: a study in Japan. , 1998, Annals of neurology.
[68] V. Singh,et al. Neuroautoimmunity: pathogenic implications for Alzheimer's disease. , 1997, Gerontology.
[69] E. Oguni,et al. Amyloid beta protein in plasma from patients with sporadic Alzheimer's disease. , 1996, Journal of the neurological sciences.