Chronic and intermittent hypoxia differentially regulate left ventricular inflammatory and extracellular matrix responses

[1]  F. Carreño,et al.  Chronic intermittent hypoxia increases blood pressure and expression of FosB/DeltaFosB in central autonomic regions. , 2011, American journal of physiology. Regulatory, integrative and comparative physiology.

[2]  D. Taub,et al.  The effects of ghrelin on inflammation and the immune system , 2011, Molecular and Cellular Endocrinology.

[3]  J. Cigarroa,et al.  CC chemokine receptor 5 deletion impairs macrophage activation and induces adverse remodeling following myocardial infarction. , 2011, American journal of physiology. Heart and circulatory physiology.

[4]  Kedar S Vaidya,et al.  Gli1 enhances migration and invasion via up-regulation of MMP-11 and promotes metastasis in ERα negative breast cancer cell lines , 2011, Clinical & Experimental Metastasis.

[5]  Nguyen T. Nguyen,et al.  SPARC mediates early extracellular matrix remodeling following myocardial infarction. , 2011, American journal of physiology. Heart and circulatory physiology.

[6]  Stephen T. Brown,et al.  Divergent roles of reactive oxygen species in the responses of perinatal adrenal chromaffin cells to hypoxic challenges , 2010, Respiratory Physiology & Neurobiology.

[7]  A. Newman,et al.  Prospective Study of Obstructive Sleep Apnea and Incident Coronary Heart Disease and Heart Failure: The Sleep Heart Health Study , 2010, Circulation.

[8]  Kurt R Stenmark,et al.  Animal models of pulmonary arterial hypertension: the hope for etiological discovery and pharmacological cure. , 2009, American journal of physiology. Lung cellular and molecular physiology.

[9]  N. Chesler,et al.  Collagen-related gene and protein expression changes in the lung in response to chronic hypoxia , 2009, Biomechanics and modeling in mechanobiology.

[10]  W. Fu,et al.  Hypoxia‐induced matrix metalloproteinase‐13 expression in astrocytes enhances permeability of brain endothelial cells , 2009, Journal of cellular physiology.

[11]  D. Sajkov,et al.  Obstructive sleep apnea and pulmonary hypertension. , 2009, Progress in cardiovascular diseases.

[12]  F. Carreño,et al.  Chronic sustained and intermittent hypoxia reduce function of ATP-sensitive potassium channels in nucleus of the solitary tract. , 2008, American journal of physiology. Regulatory, integrative and comparative physiology.

[13]  R. Senior,et al.  Fragments of extracellular matrix as mediators of inflammation. , 2008, The international journal of biochemistry & cell biology.

[14]  Hai-Chao Han,et al.  Age-related cardiac muscle sarcopenia: Combining experimental and mathematical modeling to identify mechanisms , 2008, Experimental Gerontology.

[15]  J. Nanduri,et al.  Cellular mechanisms associated with intermittent hypoxia. , 2007, Essays in biochemistry.

[16]  S. Mifflin,et al.  Chronic intermittent hypoxia alters NMDA and AMPA-evoked currents in NTS neurons receiving carotid body chemoreceptor inputs. , 2007, American journal of physiology. Regulatory, integrative and comparative physiology.

[17]  F. Tsai,et al.  Eccentric cardiac hypertrophy was induced by long‐term intermittent hypoxia in rats , 2007, Experimental physiology.

[18]  N. Voelkel,et al.  Gene expression profiling in pulmonary hypertension. , 2007, Proceedings of the American Thoracic Society.

[19]  J. Uitto,et al.  Extracellular matrix protein 1 inhibits the activity of matrix metalloproteinase 9 through high‐affinity protein/protein interactions , 2006, Experimental dermatology.

[20]  M. Frid,et al.  Role of the adventitia in pulmonary vascular remodeling. , 2006, Physiology.

[21]  T. Mak,et al.  Cutting Edge: Tissue Inhibitor of Metalloproteinase 3 Regulates TNF-Dependent Systemic Inflammation , 2006, The Journal of Immunology.

[22]  C. Hinojosa-Laborde,et al.  Sex Differences in Blood Pressure Response to Intermittent Hypoxia in Rats , 2005, Hypertension.

[23]  Eugene A. Kapp,et al.  Overview of the HUPO Plasma Proteome Project: Results from the pilot phase with 35 collaborating laboratories and multiple analytical groups, generating a core dataset of 3020 proteins and a publicly‐available database , 2005, Proteomics.

[24]  M. Nomizu,et al.  Functional Sites in the Laminin Alpha Chains , 2005, Connective tissue research.

[25]  B. Fingleton,et al.  Abnormal TNF activity in Timp3−/− mice leads to chronic hepatic inflammation and failure of liver regeneration , 2004, Nature Genetics.

[26]  S. Mifflin,et al.  Chronic hypoxia abolishes posthypoxia frequency decline in the anesthetized rat. , 2003, American journal of physiology. Regulatory, integrative and comparative physiology.

[27]  Richard T. Lee,et al.  Effect of a Cleavage-Resistant Collagen Mutation on Left Ventricular Remodeling , 2003, Circulation research.

[28]  J. Hedner,et al.  Hypoxic pressor response, cardiac size, and natriuretic peptides are modified by long-term intermittent hypoxia. , 1999, Journal of applied physiology.

[29]  E. Fletcher,et al.  Acute blood pressure elevation during repetitive hypocapnic and eucapnic hypoxia in rats. , 1997, Journal of applied physiology.

[30]  A. Shinagawa,et al.  Cell growth-promoting activity of tissue inhibitor of metalloproteinases-2 (TIMP-2). , 1994, Journal of cell science.

[31]  M. Spach,et al.  Picrosirius red staining of cardiac muscle following phosphomolybdic acid treatment. , 1987, Stain technology.