Compartmentalized PKA signaling events are required for synaptic tagging and capture during hippocampal late-phase long-term potentiation.
暂无分享,去创建一个
[1] T. Abel,et al. Metaplasticity of the late‐phase of long‐term potentiation: a critical role for protein kinase A in synaptic tagging , 2006, The European journal of neuroscience.
[2] Jennie Z. Young,et al. Homosynaptic and Heterosynaptic Inhibition of Synaptic Tagging and Capture of Long-Term Potentiation by Previous Synaptic Activity , 2005, The Journal of Neuroscience.
[3] T. Sacktor,et al. Synaptic Tagging and Cross-Tagging: The Role of Protein Kinase Mζ in Maintaining Long-Term Potentiation But Not Long-Term Depression , 2005, The Journal of Neuroscience.
[4] John D. Scott,et al. AKAP signalling complexes: focal points in space and time , 2004, Nature Reviews Molecular Cell Biology.
[5] S. Sajikumar,et al. Late-associativity, synaptic tagging, and the role of dopamine during LTP and LTD , 2004, Neurobiology of Learning and Memory.
[6] John D. Scott,et al. Orchestration of synaptic plasticity through AKAP signaling complexes , 2004, Neuropharmacology.
[7] M. Greenberg,et al. Localization of 3′,5′-cyclic adenosine monophosphate phosphodiesterase (cAMP-PDEase) activity in isolated bovine thyroid plasma membranes , 2004, Histochemistry.
[8] P. Nguyen,et al. Regulation of hippocampal synaptic plasticity by cyclic AMP-dependent protein kinases , 2003, Progress in Neurobiology.
[9] Ted Abel,et al. Temporal spacing of synaptic stimulation critically modulates the dependence of LTP on cyclic AMP‐dependent protein kinase , 2003, Hippocampus.
[10] T. Abel,et al. Genetic and pharmacological demonstration of a role for cyclic AMP‐dependent protein kinase‐mediated suppression of protein phosphatases in gating the expression of late LTP , 2002, The European journal of neuroscience.
[11] Eric R. Kandel,et al. Expression of Constitutively Active CREB Protein Facilitates the Late Phase of Long-Term Potentiation by Enhancing Synaptic Capture , 2002, Cell.
[12] C. Apperson-Hansen,et al. AKAP-Mediated Targeting of Protein Kinase A Regulates Contractility in Cardiac Myocytes , 2001, Circulation research.
[13] T. Abel,et al. Genetic and pharmacological demonstration of differential recruitment of cAMP-dependent protein kinases by synaptic activity. , 2000, Journal of neurophysiology.
[14] R U Muller,et al. Parallel Instabilities of Long-Term Potentiation, Place Cells, and Learning Caused by Decreased Protein Kinase A Activity , 2000, The Journal of Neuroscience.
[15] R. Huganir,et al. Targeting of PKA to Glutamate Receptors through a MAGUK-AKAP Complex , 2000, Neuron.
[16] S. J. Martin,et al. Synaptic plasticity and memory: an evaluation of the hypothesis. , 2000, Annual review of neuroscience.
[17] R. Nicoll,et al. Long-term potentiation--a decade of progress? , 1999, Science.
[18] M. Sheng,et al. Regulation of NMDA receptors by an associated phosphatase-kinase signaling complex. , 1999, Science.
[19] U. Frey,et al. Weak before strong: dissociating synaptic tagging and plasticity-factor accounts of late-LTP , 1998, Neuropharmacology.
[20] E. Kandel,et al. Genetic Demonstration of a Role for PKA in the Late Phase of LTP and in Hippocampus-Based Long-Term Memory , 1997, Cell.
[21] S. Vijayaraghavan,et al. Protein Kinase A-anchoring Inhibitor Peptides Arrest Mammalian Sperm Motility* , 1997, The Journal of Biological Chemistry.
[22] U. Frey,et al. Synaptic tagging and long-term potentiation , 1997, Nature.
[23] J. Sweatt,et al. Transient Activation of Cyclic AMP-dependent Protein Kinase during Hippocampal Long-term Potentiation* , 1996, The Journal of Biological Chemistry.
[24] E. Kandel,et al. Requirement of a critical period of transcription for induction of a late phase of LTP. , 1994, Science.
[25] E. Kandel,et al. Recruitment of long-lasting and protein kinase A-dependent long-term potentiation in the CA1 region of hippocampus requires repeated tetanization. , 1994, Learning & memory.
[26] E. Kandel,et al. Effects of cAMP simulate a late stage of LTP in hippocampal CA1 neurons. , 1993, Science.
[27] K. Reymann,et al. Protein kinase A inhibitors prevent the maintenance of hippocampal long-term potentiation. , 1993, Neuroreport.
[28] T. Bliss,et al. A synaptic model of memory: long-term potentiation in the hippocampus , 1993, Nature.
[29] R. Cone,et al. Localization of the cAMP-dependent protein kinase to the postsynaptic densities by A-kinase anchoring proteins. Characterization of AKAP 79. , 1992, The Journal of biological chemistry.
[30] R. Nicoll,et al. Postsynaptic contribution to long-term potentiation revealed by the analysis of miniature synaptic currents , 1992, Nature.
[31] U. Frey,et al. Anisomycin, an inhibitor of protein synthesis, blocks late phases of LTP phenomena in the hippocampal CA1 region in vitro , 1988, Brain Research.
[32] R. Wojcikiewicz,et al. Muscarinic acetylcholine receptor activation causes inhibition of cyclic AMP accumulation, prolactin and growth hormone secretion in GH3 rat anterior pituitary tumour cells. , 1984, Biochimica et biophysica acta.
[33] N. J. De Souza,et al. Forskolin: A labdane diterpenoid with antihypertensive, positive inotropic, platelet aggregation inhibitory, and adenylate cyclase activating properties , 1983, Medicinal research reviews.
[34] G. Collingridge,et al. Excitatory amino acids in synaptic transmission in the Schaffer collateral‐commissural pathway of the rat hippocampus. , 1983, The Journal of physiology.
[35] T. Bliss,et al. Long‐lasting potentiation of synaptic transmission in the dentate area of the anaesthetized rabbit following stimulation of the perforant path , 1973, The Journal of physiology.