Plaque burden, arterial remodeling and plaque vulnerability: determined by systemic factors?

[1]  B Hillen,et al.  The impact of atherosclerotic arterial remodeling on percentage of luminal stenosis varies widely within the arterial system. A postmortem study. , 1997, Arteriosclerosis, thrombosis, and vascular biology.

[2]  G Pasterkamp,et al.  Inflammation of the atherosclerotic cap and shoulder of the plaque is a common and locally observed feature in unruptured plaques of femoral and coronary arteries. , 1999, Arteriosclerosis, thrombosis, and vascular biology.

[3]  A. Becker,et al.  Site of intimal rupture or erosion of thrombosed coronary atherosclerotic plaques is characterized by an inflammatory process irrespective of the dominant plaque morphology. , 1994, Circulation.

[4]  G. Gabbiani,et al.  Expression of class II transplantation antigen on vascular smooth muscle cells in human atherosclerosis. , 1985, The Journal of clinical investigation.

[5]  G. Heiss,et al.  Relations of Intimal‐Medial Thickness Among Sites Within the Carotid Artery as Evaluated by B‐Mode Ultrasound , 1994, Stroke.

[6]  P. Saikku,et al.  Detection of Chlamydia pneumoniae-reactive T lymphocytes in human atherosclerotic plaques of carotid artery. , 2000, Arteriosclerosis, thrombosis, and vascular biology.

[7]  P. Ridker,et al.  Inflammation, aspirin, and the risk of cardiovascular disease in apparently healthy men. , 1997, The New England journal of medicine.

[8]  R. Virmani,et al.  Coronary risk factors and plaque morphology in men with coronary disease who died suddenly. , 1997, The New England journal of medicine.

[9]  S E Nissen,et al.  Extent and direction of arterial remodeling in stable versus unstable coronary syndromes : an intravascular ultrasound study. , 2000, Circulation.

[10]  M. Davies,et al.  Risk of thrombosis in human atherosclerotic plaques: role of extracellular lipid, macrophage, and smooth muscle cell content. , 1993, British heart journal.

[11]  J. Witztum,et al.  T lymphocytes from human atherosclerotic plaques recognize oxidized low density lipoprotein. , 1995, Proceedings of the National Academy of Sciences of the United States of America.

[12]  M. Leon,et al.  Paradoxic decreases in atherosclerotic plaque mass in insulin-treated diabetic patients. , 1998, The American journal of cardiology.

[13]  J. Stengård,et al.  Antibodies to glutamic acid decarboxylase as predictors of insulin-dependent diabetes mellitus before clinical onset of disease , 1994, The Lancet.

[14]  B Hillen,et al.  Paradoxical arterial wall shrinkage may contribute to luminal narrowing of human atherosclerotic femoral arteries. , 1995, Circulation.

[15]  B Hillen,et al.  Relation of arterial geometry to luminal narrowing and histologic markers for plaque vulnerability: the remodeling paradox. , 1998, Journal of the American College of Cardiology.

[16]  N. Weissman,et al.  Intravascular ultrasonic analysis of plaque characteristics associated with coronary artery remodeling. , 1999, The American journal of cardiology.

[17]  Y. Tsujita,et al.  Reduction of serum cholesterol levels alters lesional composition of atherosclerotic plaques. Effect of pravastatin sodium on atherosclerosis in mature WHHL rabbits. , 1995, Arteriosclerosis, thrombosis, and vascular biology.

[18]  C. Wong Atherosclerotic arterial remodeling in the superficial femoral artery: individual variation in local compensatory enlargement response. , 1997, Circulation.

[19]  V. Fuster,et al.  Coronary plaque disruption. , 1995, Circulation.

[20]  J. Dean,et al.  Audit of cardiac catheterisation in a district general hospital: implications for training , 1999, Heart.

[21]  C. Zarins,et al.  Compensatory enlargement of human atherosclerotic coronary arteries. , 1987, The New England journal of medicine.

[22]  PaulHolvoet,et al.  LDL Hypercholesterolemia Is Associated With Accumulation of Oxidized LDL, Atherosclerotic Plaque Growth, and Compensatory Vessel Enlargement in Coronary Arteries of Miniature Pigs , 1998 .

[23]  J. Lindsay,et al.  Predictors of coronary arterial remodeling patterns in patients with myocardial ischemia. , 1997, The American journal of cardiology.

[24]  G. Stone,et al.  Preintervention arterial remodeling as an independent predictor of target-lesion revascularization after nonstent coronary intervention: an analysis of 777 lesions with intravascular ultrasound imaging. , 1999, Circulation.

[25]  C. Kim,et al.  Contribution of inadequate compensatory enlargement to development of human coronary artery stenosis: an in vivo intravascular ultrasound study. , 1996, Journal of the American College of Cardiology.