c-Jun N-terminal kinase activation contributes to reduced connexin43 and development of atrial arrhythmias.
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V. Fast | G. Walcott | Xun Ai | W. Kong | Jiajie Yan | E. Beyer | Qiang Zhang
[1] P. Platonov,et al. Structural abnormalities in atrial walls are associated with presence and persistency of atrial fibrillation but not with age. , 2011, Journal of the American College of Cardiology.
[2] M. Koch,et al. Connexin 43 gene therapy prevents persistent atrial fibrillation in a porcine model. , 2011, Cardiovascular research.
[3] Jian Huang,et al. Simultaneous optical mapping of transmembrane potential and wall motion in isolated, perfused whole hearts. , 2011, Journal of biomedical optics.
[4] Douglas L. Jones,et al. Atrial tachycardia/fibrillation in the connexin 43 G60S mutant (Oculodentodigital dysplasia) mouse. , 2011, American journal of physiology. Heart and circulatory physiology.
[5] Yibin Wang,et al. Mitogen-activated protein kinase signaling in the heart: angels versus demons in a heart-breaking tale. , 2010, Physiological reviews.
[6] S. Pogwizd,et al. Connexin43 knockdown or overexpression modulates cell coupling in control and failing rabbit left ventricular myocytes. , 2010, Cardiovascular research.
[7] M. Rich,et al. Epidemiology of atrial fibrillation , 2009, Journal of Interventional Cardiac Electrophysiology.
[8] S. Vatner,et al. Sirt1 Regulates Aging and Resistance to Oxidative Stress in the Heart , 2007, Circulation research.
[9] A. Kleber,et al. Relative Contributions of Connexins 40 and 43 to Atrial Impulse Propagation in Synthetic Strands of Neonatal and Fetal Murine Cardiomyocytes , 2006, Circulation research.
[10] P. Boyden,et al. Sodium current function in adult and aged canine atrial cells. , 2006, American journal of physiology. Heart and circulatory physiology.
[11] Congxin Huang,et al. Differences in the aging-associated trends of the monophasic action potential duration and effective refractory period of the right and left atria of the rat. , 2006, Circulation journal : official journal of the Japanese Circulation Society.
[12] S. Pogwizd,et al. Connexin 43 Downregulation and Dephosphorylation in Nonischemic Heart Failure Is Associated With Enhanced Colocalized Protein Phosphatase Type 2A , 2004, Circulation research.
[13] A. Orlandi,et al. Role of ageing and coronary atherosclerosis in the development of cardiac fibrosis in the rabbit. , 2004, Cardiovascular research.
[14] D. Rosenbaum,et al. Targeted Activation of c-Jun N-terminal Kinase in Vivo Induces Restrictive Cardiomyopathy and Conduction Defects*[boxs] , 2004, Journal of Biological Chemistry.
[15] H. Jongsma,et al. Analysis of the rat connexin 43 proximal promoter in neonatal cardiomyocytes. , 2003, Gene.
[16] S. Cook,et al. ΔMEKK3:ER* activation induces a p38α/β2-dependent cell cycle arrest at the G2 checkpoint , 2002, Oncogene.
[17] J. Saffitz,et al. c-Jun N-Terminal Kinase Activation Mediates Downregulation of Connexin43 in Cardiomyocytes , 2002, Circulation research.
[18] Jonathan C. Newton,et al. Intramural Virtual Electrodes During Defibrillation Shocks in Left Ventricular Wall Assessed by Optical Mapping of Membrane Potential , 2002, Circulation.
[19] M. Rosen,et al. Cellular electrophysiologic properties of old canine atria provide a substrate for arrhythmogenesis. , 2002, Cardiovascular research.
[20] Nicholas S Peters,et al. Relative expression of immunolocalized connexins 40 and 43 correlates with human atrial conduction properties. , 2002, Journal of the American College of Cardiology.
[21] David W. Anderson,et al. SP600125, an anthrapyrazolone inhibitor of Jun N-terminal kinase , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[22] J E Saffitz,et al. High resolution optical mapping reveals conduction slowing in connexin43 deficient mice. , 2001, Cardiovascular research.
[23] L. Mahadevan,et al. Anisomycin Selectively Desensitizes Signalling Components Involved in Stress Kinase Activation and fos andjun Induction , 1998, Molecular and Cellular Biology.
[24] H. Jongsma,et al. Characterization of gap junction channels in adult rabbit atrial and ventricular myocardium. , 1997, Circulation research.
[25] J E Saffitz,et al. The Molecular Basis of Anisotropy: Role of Gap Junctions , 1995, Journal of cardiovascular electrophysiology.
[26] D. Levy,et al. Independent risk factors for atrial fibrillation in a population-based cohort. The Framingham Heart Study. , 1994, JAMA.
[27] M. Allessie,et al. Quantification of spatial inhomogeneity in conduction and initiation of reentrant atrial arrhythmias. , 1990, The American journal of physiology.
[28] M. Spach,et al. Relating Extracellular Potentials and Their Derivatives to Anisotropic Propagation at a Microscopic Level in Human Cardiac Muscle: Evidence for Electrical Uncoupling of Side‐to‐Side Fiber Connections with Increasing Age , 1986, Circulation research.
[29] A. L. Wit,et al. Is there a role for remodeled connexins in AF? No simple answers. , 2008, Journal of molecular and cellular cardiology.
[30] M. Karin. Inflammation-activated protein kinases as targets for drug development. , 2005, Proceedings of the American Thoracic Society.