Knockdown of SETDB1 inhibits breast cancer progression by miR-381-3p-related regulation
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Yan Li | Yushuang Luo | Rong Chen | Yinzhuo Diao | Qijing Guo | Baohua Fan | Milu Wu | Nannan Lv
[1] Jun Zhao,et al. microRNA-381 suppresses the growth and increases cisplatin sensitivity in non-small cell lung cancer cells through inhibition of nuclear factor-κB signaling. , 2018, Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie.
[2] X. Yang,et al. miR-381-3p suppresses the proliferation of oral squamous cell carcinoma cells by directly targeting FGFR2. , 2017, American journal of cancer research.
[3] Dahong Zhang,et al. miR-381 inhibited breast cancer cells proliferation, epithelial-to-mesenchymal transition and metastasis by targeting CXCR4. , 2017, Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie.
[4] A. Jemal,et al. Cancer treatment and survivorship statistics, 2016 , 2016, CA: a cancer journal for clinicians.
[5] Ting Li,et al. MiRNA-21 induces epithelial to mesenchymal transition and gemcitabine resistance via the PTEN/AKT pathway in breast cancer , 2016, Tumor Biology.
[6] I. Ng,et al. Up‐regulation of histone methyltransferase SETDB1 by multiple mechanisms in hepatocellular carcinoma promotes cancer metastasis , 2016, Hepatology.
[7] Jidong Zhu,et al. Histone methyltransferase SETDB1 regulates liver cancer cell growth through methylation of p53 , 2015, Nature Communications.
[8] L. Pfeffer,et al. MiRNA-621 sensitizes breast cancer to chemotherapy by suppressing FBXO11 and enhancing p53 activity , 2015, Oncogene.
[9] G. Stein,et al. Targeting of Runx2 by miR-135 and miR-203 Impairs Progression of Breast Cancer and Metastatic Bone Disease. , 2015, Cancer research.
[10] Jer-Yen Yang,et al. Molecular and Cellular Pathobiology H 3 K 9 Histone Methyltransferase , KMT 1 E / SETDB 1 , Cooperates with the SMAD 2 / 3 Pathway to Suppress Lung Cancer Metastasis , 2014 .
[11] Jing Wang,et al. MiR‐7, Inhibited Indirectly by LincRNA HOTAIR, Directly Inhibits SETDB1 and Reverses the EMT of Breast Cancer Stem Cells by Downregulating the STAT3 Pathway , 2014, Stem cells.
[12] A. Papavassiliou,et al. High‐frequency p16INK4A promoter methylation is associated with histone methyltransferase SETDB1 expression in sporadic cutaneous melanoma , 2014, Experimental dermatology.
[13] A. Papavassiliou,et al. Role of Histone Lysine Methyltransferases SUV39H1 and SETDB1 in Gliomagenesis: Modulation of Cell Proliferation, Migration, and Colony Formation , 2014, NeuroMolecular Medicine.
[14] J. Hou,et al. Histone methyltransferase SETDB1 is required for prostate cancer cell proliferation, migration and invasion , 2014, Asian journal of andrology.
[15] M. Esteller,et al. Gene amplification of the histone methyltransferase SETDB1 contributes to human lung tumorigenesis , 2013, Oncogene.
[16] T. Sellers,et al. Upregulation of miRNA-155 promotes tumour angiogenesis by targeting VHL and is associated with poor prognosis and triple-negative breast cancer , 2013, Oncogene.
[17] D. Hwang,et al. Notch1 counteracts WNT/β-catenin signaling through chromatin modification in colorectal cancer. , 2012, The Journal of clinical investigation.
[18] A. Yoshimura,et al. MiR-23a regulates TGF-β-induced epithelial-mesenchymal transition by targeting E-cadherin in lung cancer cells , 2012, International journal of oncology.
[19] R. Agami,et al. MicroRNA regulation by RNA-binding proteins and its implications for cancer , 2011, Nature Reviews Cancer.
[20] S. Minucci,et al. SILAC-based proteomic analysis to dissect the “histone modification signature” of human breast cancer cells , 2011, Amino Acids.
[21] Steven J. M. Jones,et al. DNA methylation and SETDB1/H3K9me3 regulate predominantly distinct sets of genes, retroelements, and chimeric transcripts in mESCs. , 2011, Cell stem cell.
[22] David A. Orlando,et al. The histone methyltransferase SETDB1 is recurrently amplified in melanoma and accelerates its onset , 2011, Nature.
[23] Peter T Nelson,et al. The miR-15/107 group of microRNA genes: evolutionary biology, cellular functions, and roles in human diseases. , 2010, Journal of molecular biology.
[24] H. Kimura,et al. Proviral silencing in embryonic stem cells requires the histone methyltransferase ESET , 2010, Nature.
[25] C. Croce. Causes and consequences of microRNA dysregulation in cancer , 2009, Nature Reviews Genetics.
[26] J. Issa,et al. Downregulation of Histone H3 Lysine 9 Methyltransferase G9a Induces Centrosome Disruption and Chromosome Instability in Cancer Cells , 2008, PloS one.
[27] W. Gerald,et al. Endogenous human microRNAs that suppress breast cancer metastasis , 2008, Nature.
[28] J. Lieberman,et al. let-7 Regulates Self Renewal and Tumorigenicity of Breast Cancer Cells , 2007, Cell.
[29] R. Weinberg,et al. Tumour invasion and metastasis initiated by microRNA-10b in breast cancer , 2007, Nature.
[30] Anwar Hossain,et al. Mir-17-5p Regulates Breast Cancer Cell Proliferation by Inhibiting Translation of AIB1 mRNA , 2006, Molecular and Cellular Biology.
[31] A. Riggs,et al. The Histone Methyltransferase SETDB1 and the DNA Methyltransferase DNMT3A Interact Directly and Localize to Promoters Silenced in Cancer Cells* , 2006, Journal of Biological Chemistry.
[32] T. Tuschl,et al. Identification of Novel Argonaute-Associated Proteins , 2005, Current Biology.
[33] Cyrus Martin,et al. The diverse functions of histone lysine methylation , 2005, Nature Reviews Molecular Cell Biology.
[34] C. Croce,et al. MicroRNA gene expression deregulation in human breast cancer. , 2005, Cancer research.
[35] J. Castle,et al. Microarray analysis shows that some microRNAs downregulate large numbers of target mRNAs , 2005, Nature.
[36] Shireen A. Sarraf,et al. Methyl-CpG binding protein MBD1 couples histone H3 methylation at lysine 9 by SETDB1 to DNA replication and chromatin assembly. , 2004, Molecular cell.
[37] M. Rederstorff,et al. Dual luciferase gene reporter assays to study miRNA function. , 2015, Methods in molecular biology.
[38] A. Jemal,et al. Breast cancer statistics, 2013 , 2014, CA: a cancer journal for clinicians.
[39] A. Jemal,et al. Breast Cancer Statistics , 2013 .