Intrarectal administration of mCRAMP-encoding plasmid reverses exacerbated colitis in Cnlp−/− mice
暂无分享,去创建一个
Xj Wang | C. Cho | J. J. Sung | R. Gallo | C. W. Lee | X. Wang | H. Wong | J. Sung | W. K. Wu | C. Cho | E. Tai | L. Yu | Z. J. Li | C. Wong | J. Yu | ZJ Li | L. Yu | Ekk Tai | Wkk Wu | Hps Wong | Cw Lee | Ccm Wong | RL Gallo
[1] S. Targan,et al. Cathelicidin signaling via the Toll-like receptor protects against colitis in mice. , 2011, Gastroenterology.
[2] Fiona Powrie,et al. Intestinal homeostasis and its breakdown in inflammatory bowel disease , 2011, Nature.
[3] R. Xavier,et al. Genetics and pathogenesis of inflammatory bowel disease , 2011, Nature.
[4] S. Itzkowitz,et al. Intestinal inflammation and cancer. , 2011, Gastroenterology.
[5] W. Strober,et al. Proinflammatory cytokines in the pathogenesis of inflammatory bowel diseases. , 2011, Gastroenterology.
[6] L. Peyrin-Biroulet,et al. Recent advances in cytokines: therapeutic implications for inflammatory bowel diseases. , 2011, World journal of gastroenterology.
[7] W. Wu,et al. Cathelicidins in inflammation and tissue repair: Potential therapeutic applications for gastrointestinal disorders , 2010, Acta Pharmacologica Sinica.
[8] Y. Haikel,et al. Antimicrobial Peptides Present in Mammalian Skin and Gut are Multifunctional Defence Molecules , 2010 .
[9] J. Schulzke,et al. Altered permeability in inflammatory bowel disease: pathophysiology and clinical implications , 2007, Current opinion in gastroenterology.
[10] W. Wu,et al. A New Role for Cathelicidin in Ulcerative Colitis in Mice , 2007, Experimental biology and medicine.
[11] H. Kawasaki,et al. Naked gene therapy of hepatocyte growth factor for dextran sulfate sodium-induced colitis in mice. , 2006, Biochemical and biophysical research communications.
[12] J. Meijerink,et al. Muc2-deficient mice spontaneously develop colitis, indicating that MUC2 is critical for colonic protection. , 2006, Gastroenterology.
[13] R. Gallo,et al. Heterogeneous expression of human cathelicidin hCAP18/LL-37 in inflammatory bowel diseases , 2006, European journal of gastroenterology & hepatology.
[14] E. Doménech. Inflammatory Bowel Disease: Current Therapeutic Options , 2006, Digestion.
[15] Y. Shai,et al. Endotoxin (Lipopolysaccharide) Neutralization by Innate Immunity Host-Defense Peptides , 2006, Journal of Biological Chemistry.
[16] J. Mathis,et al. Reversal of experimental colitis disease activity in mice following administration of an adenoviral IL-10 vector , 2005, Journal of Inflammation.
[17] P. Gibson,et al. Reinforcing the mucus: a new therapeutic approach for ulcerative colitis? , 2005, Gut.
[18] T. Strom,et al. Tumor necrosis factor-α, interleukin-1β, and interleukin-6 expression in inflammatory bowel disease , 1992, Digestive Diseases and Sciences.
[19] Katsuhiro Hayashi,et al. Hepatocyte Growth Factor Facilitates Colonic Mucosal Repair in Experimental Ulcerative Colitis in Rats , 2003, Journal of Pharmacology and Experimental Therapeutics.
[20] S. Leung,et al. Cigarette smoke exposure increases ulcerative colitis-associated colonic adenoma formation in mice. , 2003, Carcinogenesis.
[21] M. Kagnoff,et al. Cell Differentiation Is a Key Determinant of Cathelicidin LL-37/Human Cationic Antimicrobial Protein 18 Expression by Human Colon Epithelium , 2002, Infection and Immunity.
[22] Takaaki Ohtake,et al. Innate antimicrobial peptide protects the skin from invasive bacterial infection , 2001, Nature.
[23] Ji Ming Wang,et al. Ll-37, the Neutrophil Granule–And Epithelial Cell–Derived Cathelicidin, Utilizes Formyl Peptide Receptor–Like 1 (Fprl1) as a Receptor to Chemoattract Human Peripheral Blood Neutrophils, Monocytes, and T Cells , 2000, The Journal of experimental medicine.
[24] C. Cho,et al. The role of polyamines in gastric mucus synthesis inhibited by cigarette smoke or its extract , 2000, Gut.
[25] C. Cho,et al. Involvement of neutrophils and free radicals in the potentiating effects of passive cigarette smoking on inflammatory bowel disease in rats. , 1999, Gastroenterology.
[26] D. Hommes,et al. Treatment of Crohn's disease with anti-tumor necrosis factor chimeric monoclonal antibody (cA2). , 1995, Gastroenterology.
[27] R. Newcombe,et al. Thickness of adherent mucus gel on colonic mucosa in humans and its relevance to colitis. , 1994, Gut.
[28] H. Cooper,et al. Clinicopathologic study of dextran sulfate sodium experimental murine colitis. , 1993, Laboratory investigation; a journal of technical methods and pathology.
[29] S. Ben‐Sasson,et al. Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation , 1992, The Journal of cell biology.
[30] A. Corfield,et al. Mucin degradation in the human colon: production of sialidase, sialate O-acetylesterase, N-acetylneuraminate lyase, arylesterase, and glycosulfatase activities by strains of fecal bacteria , 1992, Infection and immunity.
[31] M. Kobayashi,et al. Enhanced mucosal cytokine production in inflammatory bowel disease. , 1992, Gastroenterology.