Phospholipase D1 is up-regulated in the mitochondrial fraction from the brains of Alzheimer's disease patients
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Yong-Sun Kim | D. Min | M. Park | Jae-kwang Jin | E. Choi | P. Kozlowski | Yun-Jung Lee | Nam‐Ho Kim | Heui-Soo Kim
[1] J. Quinn,et al. Mitochondria are a direct site of A beta accumulation in Alzheimer's disease neurons: implications for free radical generation and oxidative damage in disease progression. , 2006, Human molecular genetics.
[2] P. Reddy. Amyloid precursor protein‐mediated free radicals and oxidative damage: Implications for the development and progression of Alzheimer's disease , 2006, Journal of neurochemistry.
[3] S. Pasquaré,et al. Phosphatidic acid and diacylglycerol generation is regulated by insulin in cerebral cortex synaptosomes from adult and aged rats , 2005, Journal of neuroscience research.
[4] J. Choi,et al. Increased expression of phospholipase D1 in the brains of scrapie‐infected mice , 2005, Journal of neurochemistry.
[5] S. Pasquaré,et al. Phospholipase D and phosphatidate phosphohydrolase activities in rat cerebellum during aging , 2004, Lipids.
[6] D. Min,et al. Expression and regulation of phospholipase D during neuronal differentiation of PC12 cells , 2001, Neuropharmacology.
[7] M. Citron,et al. Aβ-Generating Enzymes Recent Advances in β- and γ-Secretase Research , 2000, Neuron.
[8] A. Ramachandran,et al. Attenuation of intestinal ischemia/reperfusion injury with sodium nitroprusside: studies on mitochondrial function and lipid changes. , 2000, Biochimica et biophysica acta.
[9] S. Melov,et al. Mitochondrial disease in mouse results in increased oxidative stress. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[10] J. Kanfer,et al. Phospholipases as mediators of amyloid beta peptide neurotoxicity: an early event contributing to neurodegeneration characteristic of Alzheimer's disease , 1998, Neuroscience Letters.
[11] J. Kanfer,et al. Activation of LA-N-2 Cell Phospholipase D by Amyloid Beta Protein (25–35) , 1998, Neurochemical Research.
[12] C. Kent. CTP:phosphocholine cytidylyltransferase. , 1997, Biochimica et biophysica acta.
[13] S. Ryu,et al. Activation of phospholipase D and the possible mechanism of activation in wound-induced lipid hydrolysis in castor bean leaves. , 1996, Biochimica et biophysica acta.
[14] J. Pettegrew,et al. Phospholipid metabolism in Alzheimer's disease and in a human cholinergic cell. , 1996, Journal of lipid mediators and cell signalling.
[15] Julia W. Chang,et al. Interleukin‐1β Induces Prostaglandin G/H Synthase‐2 (Cyclooxygenase‐2) in Primary Murine Astrocyte Cultures , 1996, Journal of neurochemistry.
[16] J. Kanfer,et al. Amyloid beta protein (25–35) stimulation of phospholipases A, C and D activities of LA‐N‐2 cells , 1995, FEBS letters.
[17] G. R. Bartlett. Phosphorus assay in column chromatography. , 1959, The Journal of biological chemistry.
[18] J. Hardy,et al. The Amyloid Hypothesis of Alzheimer ’ s Disease : Progress and Problems on the Road to Therapeutics , 2009 .
[19] H. Braak,et al. Neuropathological stageing of Alzheimer-related changes , 2004, Acta Neuropathologica.
[20] C. Lee,et al. Mitochondrial dysfunction and calcium perturbation induced by traumatic brain injury. , 1997, Journal of neurotrauma.
[21] M. Madesh,et al. Phospholipase D activity in the intestinal mitochondria: activation by oxygen free radicals. , 1997, Free radical biology & medicine.