Phospholipase D1 is up-regulated in the mitochondrial fraction from the brains of Alzheimer's disease patients

[1]  J. Quinn,et al.  Mitochondria are a direct site of A beta accumulation in Alzheimer's disease neurons: implications for free radical generation and oxidative damage in disease progression. , 2006, Human molecular genetics.

[2]  P. Reddy Amyloid precursor protein‐mediated free radicals and oxidative damage: Implications for the development and progression of Alzheimer's disease , 2006, Journal of neurochemistry.

[3]  S. Pasquaré,et al.  Phosphatidic acid and diacylglycerol generation is regulated by insulin in cerebral cortex synaptosomes from adult and aged rats , 2005, Journal of neuroscience research.

[4]  J. Choi,et al.  Increased expression of phospholipase D1 in the brains of scrapie‐infected mice , 2005, Journal of neurochemistry.

[5]  S. Pasquaré,et al.  Phospholipase D and phosphatidate phosphohydrolase activities in rat cerebellum during aging , 2004, Lipids.

[6]  D. Min,et al.  Expression and regulation of phospholipase D during neuronal differentiation of PC12 cells , 2001, Neuropharmacology.

[7]  M. Citron,et al.  Aβ-Generating Enzymes Recent Advances in β- and γ-Secretase Research , 2000, Neuron.

[8]  A. Ramachandran,et al.  Attenuation of intestinal ischemia/reperfusion injury with sodium nitroprusside: studies on mitochondrial function and lipid changes. , 2000, Biochimica et biophysica acta.

[9]  S. Melov,et al.  Mitochondrial disease in mouse results in increased oxidative stress. , 1999, Proceedings of the National Academy of Sciences of the United States of America.

[10]  J. Kanfer,et al.  Phospholipases as mediators of amyloid beta peptide neurotoxicity: an early event contributing to neurodegeneration characteristic of Alzheimer's disease , 1998, Neuroscience Letters.

[11]  J. Kanfer,et al.  Activation of LA-N-2 Cell Phospholipase D by Amyloid Beta Protein (25–35) , 1998, Neurochemical Research.

[12]  C. Kent CTP:phosphocholine cytidylyltransferase. , 1997, Biochimica et biophysica acta.

[13]  S. Ryu,et al.  Activation of phospholipase D and the possible mechanism of activation in wound-induced lipid hydrolysis in castor bean leaves. , 1996, Biochimica et biophysica acta.

[14]  J. Pettegrew,et al.  Phospholipid metabolism in Alzheimer's disease and in a human cholinergic cell. , 1996, Journal of lipid mediators and cell signalling.

[15]  Julia W. Chang,et al.  Interleukin‐1β Induces Prostaglandin G/H Synthase‐2 (Cyclooxygenase‐2) in Primary Murine Astrocyte Cultures , 1996, Journal of neurochemistry.

[16]  J. Kanfer,et al.  Amyloid beta protein (25–35) stimulation of phospholipases A, C and D activities of LA‐N‐2 cells , 1995, FEBS letters.

[17]  G. R. Bartlett Phosphorus assay in column chromatography. , 1959, The Journal of biological chemistry.

[18]  J. Hardy,et al.  The Amyloid Hypothesis of Alzheimer ’ s Disease : Progress and Problems on the Road to Therapeutics , 2009 .

[19]  H. Braak,et al.  Neuropathological stageing of Alzheimer-related changes , 2004, Acta Neuropathologica.

[20]  C. Lee,et al.  Mitochondrial dysfunction and calcium perturbation induced by traumatic brain injury. , 1997, Journal of neurotrauma.

[21]  M. Madesh,et al.  Phospholipase D activity in the intestinal mitochondria: activation by oxygen free radicals. , 1997, Free radical biology & medicine.