Pathophysiology of the brain extracellular matrix: a new target for remyelination
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[1] V. Belegu,et al. Chondroitin sulfate proteoglycans inhibit oligodendrocyte myelination through PTPσ , 2013, Experimental Neurology.
[2] T. Montine,et al. Digestion products of the PH20 hyaluronidase inhibit remyelination , 2013, Annals of neurology.
[3] Chao Zhao,et al. Fibronectin aggregation in multiple sclerosis lesions impairs remyelination. , 2013, Brain : a journal of neurology.
[4] B. Porter,et al. The perineuronal net component of the extracellular matrix in plasticity and epilepsy , 2012, Neurochemistry International.
[5] B. Trapp,et al. Cortical remyelination: A new target for repair therapies in multiple sclerosis , 2012, Annals of neurology.
[6] S. Takeda,et al. Laminin regulates postnatal oligodendrocyte production by promoting oligodendrocyte progenitor survival in the subventricular zone , 2012, Glia.
[7] M. Selzer,et al. Scar-mediated inhibition and CSPG receptors in the CNS , 2012, Experimental Neurology.
[8] Scott Sloka,et al. Chondroitin sulfate proteoglycans in demyelinated lesions impair remyelination , 2012, Annals of neurology.
[9] D. Vocadlo,et al. Developing inhibitors of glycan processing enzymes as tools for enabling glycobiology. , 2012, Nature chemical biology.
[10] M. Fehlings,et al. Chondroitinase and Growth Factors Enhance Activation and Oligodendrocyte Differentiation of Endogenous Neural Precursor Cells after Spinal Cord Injury , 2012, PloS one.
[11] L. Hsieh‐Wilson,et al. A sulfated carbohydrate epitope inhibits axon regeneration after injury , 2012, Proceedings of the National Academy of Sciences.
[12] H. M. Geller,et al. NgR1 and NgR3 are Receptors for Chondroitin Sulfate Proteoglycans , 2012, Nature Neuroscience.
[13] 田内 亮吏. The endogenous proteoglycan-degrading enzyme ADAMTS-4 promotes functional recovery after spinal cord injury , 2012 .
[14] R. Shinjo,et al. The endogenous proteoglycan-degrading enzyme ADAMTS-4 promotes functional recovery after spinal cord injury , 2012, Journal of Neuroinflammation.
[15] V. Yong,et al. Reduced inflammation accompanies diminished myelin damage and repair in the NG2 null mouse spinal cord , 2011, Journal of Neuroinflammation.
[16] J. Fawcett,et al. Extracellular matrix and perineuronal nets in CNS repair , 2011, Developmental neurobiology.
[17] M. Sheng,et al. Leukocyte Common Antigen-Related Phosphatase Is a Functional Receptor for Chondroitin Sulfate Proteoglycan Axon Growth Inhibitors , 2011, The Journal of Neuroscience.
[18] D. Osterhout,et al. The inhibitory effects of chondroitin sulfate proteoglycans on oligodendrocytes , 2011, Journal of neurochemistry.
[19] D. Stelzner,et al. Chondroitinase treatment following spinal contusion injury increases migration of oligodendrocyte progenitor cells , 2011, Experimental Neurology.
[20] J. Fawcett,et al. 6-Sulphated Chondroitins Have a Positive Influence on Axonal Regeneration , 2011, PloS one.
[21] R. Bellamkonda,et al. Targeted downregulation of N‐acetylgalactosamine 4‐sulfate 6‐O‐sulfotransferase significantly mitigates chondroitin sulfate proteoglycan‐mediated inhibition , 2011, Glia.
[22] J. Fawcett,et al. Integrin Activation Promotes Axon Growth on Inhibitory Chondroitin Sulfate Proteoglycans by Enhancing Integrin Signaling , 2011, The Journal of Neuroscience.
[23] M. Sofroniew,et al. Reactive astrocytes as therapeutic targets for CNS disorders , 2010, Neurotherapeutics.
[24] A. Minagar. Extracellular Matrix in Multiple Sclerosis Lesions: Fibrillar Collagens, Biglycan and Decorin are Upregulated and Associated with Infiltrating Immune Cells , 2011 .
[25] L. Sorokin. The impact of the extracellular matrix on inflammation , 2010, Nature Reviews Immunology.
[26] J. Newcombe,et al. Extracellular Matrix in Multiple Sclerosis Lesions: Fibrillar Collagens, Biglycan and Decorin are Upregulated and Associated with Infiltrating Immune Cells , 2010, Brain pathology.
[27] L. Vargova,et al. Bral1: Its Role in Diffusion Barrier Formation and Conduction Velocity in the CNS , 2010, The Journal of Neuroscience.
[28] C. Overall,et al. Matrix metalloproteinases: what do they not do? New substrates and biological roles identified by murine models and proteomics. , 2010, Biochimica et biophysica acta.
[29] K. Horn,et al. PTPσ Is a Receptor for Chondroitin Sulfate Proteoglycan, an Inhibitor of Neural Regeneration , 2009, Science.
[30] V. Yong,et al. Fibronectin attenuates process outgrowth in oligodendrocytes by mislocalizing MMP-9 activity , 2009, Molecular and Cellular Neuroscience.
[31] M. Simons,et al. Actomyosin contractility controls cell surface area of oligodendrocytes , 2009, BMC Cell Biology.
[32] S. Apte. A Disintegrin-like and Metalloprotease (Reprolysin-type) with Thrombospondin Type 1 Motif (ADAMTS) Superfamily: Functions and Mechanisms* , 2009, The Journal of Biological Chemistry.
[33] Wan-Wan Lin,et al. Carcinoma-produced factors activate myeloid cells through TLR2 to stimulate metastasis , 2009, Nature.
[34] S. Love,et al. Elevated Matrix Metalloproteinase-9 and Degradation of Perineuronal Nets in Cerebrocortical Multiple Sclerosis Plaques , 2008, Journal of neuropathology and experimental neurology.
[35] Michal Schwartz,et al. America , 2012 .
[36] C. Wegner,et al. Differentiation block of oligodendroglial progenitor cells as a cause for remyelination failure in chronic multiple sclerosis. , 2008, Brain : a journal of neurology.
[37] J. Silver,et al. CNS injury, glial scars, and inflammation: Inhibitory extracellular matrices and regeneration failure , 2008, Experimental Neurology.
[38] S. Back,et al. A ‘GAG’ reflex prevents repair of the damaged CNS , 2008, Trends in Neurosciences.
[39] H. M. Geller,et al. Inhibiting Glycosaminoglycan Chain Polymerization Decreases the Inhibitory Activity of Astrocyte-Derived Chondroitin Sulfate Proteoglycans , 2007, The Journal of Neuroscience.
[40] Helga E de Vries,et al. The extracellular matrix in multiple sclerosis pathology , 2007, Journal of neurochemistry.
[41] A. Shuaib,et al. Critical role of microvasculature basal lamina in ischemic brain injury , 2007, Progress in Neurobiology.
[42] T. Mikami,et al. Chondroitin/dermatan sulfate in the central nervous system. , 2007, Current opinion in structural biology.
[43] U. Rauch. Brain matrix: structure, turnover and necessity. , 2007, Biochemical Society transactions.
[44] James W. Fawcett,et al. The role of chondroitin sulfate proteoglycans in regeneration and plasticity in the central nervous system , 2007, Brain Research Reviews.
[45] M. Pizzi,et al. Transplantation of fibroblasts that overexpress matrix metalloproteinase-3 into the site of spinal cord injury in rats. , 2006, Journal of neurotrauma.
[46] L. Bö,et al. Extensive extracellular matrix depositions in active multiple sclerosis lesions , 2006, Neurobiology of Disease.
[47] D. Hoekstra,et al. Fibronectin impedes “myelin” sheet-directed flow in oligodendrocytes: A role for a beta 1 integrin-mediated PKC signaling pathway in vesicular trafficking , 2006, Molecular and Cellular Neuroscience.
[48] Z. Werb,et al. Matrix Metalloproteinase-2 Facilitates Wound Healing Events That Promote Functional Recovery after Spinal Cord Injury , 2006, The Journal of Neuroscience.
[49] Zhigang He,et al. Glial inhibition of CNS axon regeneration , 2006, Nature Reviews Neuroscience.
[50] V. Yong,et al. Metalloproteinases: Mediators of Pathology and Regeneration in the CNS , 2005, Nature Reviews Neuroscience.
[51] Zhigang He,et al. EGFR Activation Mediates Inhibition of Axon Regeneration by Myelin and Chondroitin Sulfate Proteoglycans , 2005, Science.
[52] C. ffrench-Constant,et al. Human diseases reveal novel roles for neural laminins , 2005, Trends in Neurosciences.
[53] Mahendra S Rao,et al. Hyaluronan accumulates in demyelinated lesions and inhibits oligodendrocyte progenitor maturation , 2005, Nature Medicine.
[54] Ravi V. Bellamkonda,et al. CS-4,6 is differentially upregulated in glial scar and is a potent inhibitor of neurite extension , 2005, Molecular and Cellular Neuroscience.
[55] Daniel J Brat,et al. Microregional extracellular matrix heterogeneity in brain modulates glioma cell invasion. , 2004, The international journal of biochemistry & cell biology.
[56] Barbara Grimpe,et al. A Novel DNA Enzyme Reduces Glycosaminoglycan Chains in the Glial Scar and Allows Microtransplanted Dorsal Root Ganglia Axons to Regenerate beyond Lesions in the Spinal Cord , 2004, The Journal of Neuroscience.
[57] Jerry Silver,et al. Regeneration beyond the glial scar , 2004, Nature Reviews Neuroscience.
[58] A. Bignami,et al. Hyaluronic acid and hyaluronic acid-binding proteins in brain extracellular matrix , 1993, Anatomy and Embryology.
[59] G. Opdenakker,et al. Matrix Metalloproteinase-9 Facilitates Remyelination in Part by Processing the Inhibitory NG2 Proteoglycan , 2003, The Journal of Neuroscience.
[60] R. Sidman,et al. Integrin-linked kinase is required for laminin-2–induced oligodendrocyte cell spreading and CNS myelination , 2003, The Journal of cell biology.
[61] B. Engelhardt. Development of the blood-brain barrier , 2003, Cell and Tissue Research.
[62] M. Tuszynski,et al. The chondroitin sulfate proteoglycans neurocan, brevican, phosphacan, and versican are differentially regulated following spinal cord injury , 2003, Experimental Neurology.
[63] M. Schachner,et al. Extracellular matrix molecules and synaptic plasticity , 2003, Nature Reviews Neuroscience.
[64] Masahiko Watanabe,et al. Differentiation of proliferated NG2‐positive glial progenitor cells in a remyelinating lesion , 2002, Journal of neuroscience research.
[65] E. Heber-Katz,et al. Recovery from spinal cord injury: A new transection model in the C57Bl/6 mouse , 2002, Journal of neuroscience research.
[66] R. An. Chondroitinase ABC promotes functional recovery after spinal cord injury , 2002 .
[67] R. Sobel,et al. White Matter Extracellular Matrix Chondroitin Sulfate/Dermatan Sulfate Proteoglycans in Multiple Sclerosis , 2001, Journal of neuropathology and experimental neurology.
[68] H. Ichijo,et al. Roles of the Telencephalic Cells and their Chondroitin Sulfate Proteoglycans in Delimiting an Anterior Border of the Retinal Pathway , 2001, The Journal of Neuroscience.
[69] Z. Werb,et al. How matrix metalloproteinases regulate cell behavior. , 2001, Annual review of cell and developmental biology.
[70] P. Yurchenco,et al. Form and function: The laminin family of heterotrimers , 2000, Developmental dynamics : an official publication of the American Association of Anatomists.
[71] C. Bandtlow,et al. Proteoglycans in the developing brain: new conceptual insights for old proteins. , 2000, Physiological reviews.
[72] R. Emeson,et al. Functions and mechanisms of RNA editing. , 2000, Annual review of genetics.
[73] D. Snow,et al. Embryonic Neurons Adapt to the Inhibitory Proteoglycan Aggrecan by Increasing Integrin Expression , 1999, The Journal of Neuroscience.
[74] C. ffrench-Constant,et al. Laminin-2/Integrin Interactions Enhance Myelin Membrane Formation by Oligodendrocytes , 1999, Molecular and Cellular Neuroscience.
[75] T. Ferguson,et al. Neuronal Matrix Metalloproteinase-2 Degrades and Inactivates a Neurite-Inhibiting Chondroitin Sulfate Proteoglycan , 1998, The Journal of Neuroscience.
[76] K. Fukuchi,et al. Alzheimer's disease and heparan sulfate proteoglycan. , 1998, Frontiers in bioscience : a journal and virtual library.
[77] Voon Wee Yong,et al. Matrix metalloproteinases and diseases of the CNS , 1998, Trends in Neurosciences.
[78] C. ffrench-Constant,et al. Expression of αvβ3 and αvβ8 integrins during oligodendrocyte precursor differentiation in the presence and absence of axons , 1997, Glia.
[79] D. Snow,et al. GROWTH CONE BEHAVIOR IN THE PRESENCE OF SOLUBLE CHONDROITIN SULFATE PROTEOGLYCAN (CSPG), COMPARED TO BEHAVIOR ON CSPG BOUND TO LAMININ OR FIBRONECTIN , 1996, International Journal of Developmental Neuroscience.
[80] P. Maurel,et al. Chondroitin sulfate proteoglycans in the developing central nervous system. I. Cellular sites of synthesis of neurocan and phosphacan , 1996, The Journal of comparative neurology.
[81] C. Chauzy,et al. Expression and Effects of Hyaluronan and of the Hyaluronan‐Binding Protein Hyaluronectin in Newborn Rat Brain Glial Cell Cultures , 1994, Journal of neurochemistry.
[82] J. Silver,et al. Reduction of neurite outgrowth in a model of glial scarring following CNS injury is correlated with the expression of inhibitory molecules on reactive astrocytes , 1991, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[83] B. Cragg,et al. Brain extracellular space fixed for electron microscopy , 1979, Neuroscience Letters.