Consumption of fructose- but not glucose-sweetened beverages for 10 weeks increases circulating concentrations of uric acid, retinol binding protein-4, and gamma-glutamyl transferase activity in overweight/obese humans
暂无分享,去创建一个
J. McGahan | L. Berglund | J. Schwarz | N. Keim | S. Griffen | P. Havel | K. Stanhope | A. Bremer | J. Graham | C. L. Cox | B. Hatcher
[1] J. Buzby,et al. Dietary Assessment of Major Trends in U.S. Food Consumption, 1970-2005 , 2012 .
[2] D. Kiortsis,et al. The Metabolic Role of Retinol Binding Protein 4: An Update , 2012, Hormone and Metabolic Research.
[3] E. Schaefer,et al. Metabolic responses to prolonged consumption of glucose- and fructose-sweetened beverages are not associated with postprandial or 24-h glucose and insulin excursions. , 2011, The American journal of clinical nutrition.
[4] Slobodan Ilic,et al. Nutrition/Metabolism , 2011, Intensive Care Medicine.
[5] K. Adeli,et al. Fructose: a highly lipogenic nutrient implicated in insulin resistance, hepatic steatosis, and the metabolic syndrome. , 2010, American journal of physiology. Endocrinology and metabolism.
[6] E. Boyko,et al. Association between serum uric acid level and chronic liver disease in the United States , 2010, Hepatology.
[7] J. Schwarz,et al. The role of fructose in the pathogenesis of NAFLD and the metabolic syndrome , 2010, Nature Reviews Gastroenterology &Hepatology.
[8] R. Kreis,et al. Effects of a short-term overfeeding with fructose or glucose in healthy young males. , 2010, The British journal of nutrition.
[9] Duk-Hee Kang,et al. Oxidative stress with an activation of the renin–angiotensin system in human vascular endothelial cells as a novel mechanism of uric acid-induced endothelial dysfunction , 2010, Journal of hypertension.
[10] J. Schold,et al. Excessive fructose intake induces the features of metabolic syndrome in healthy adult men: role of uric acid in the hypertensive response , 2010, International Journal of Obesity.
[11] L. Gaal,et al. Visceral adipose tissue and inflammation correlate with elevated liver tests in a cohort of overweight and obese patients , 2010, International Journal of Obesity.
[12] G. Lippi,et al. Epidemiological Association between Uric Acid Concentration in Plasma, Lipoprotein(a), and the Traditional Lipid Profile , 2010, Clinical cardiology.
[13] F. Tsai,et al. Levels of retinol-binding protein 4 and uric acid in patients with type 2 diabetes mellitus. , 2009, Metabolism: clinical and experimental.
[14] M. Boban,et al. Acute, food-induced moderate elevation of plasma uric acid protects against hyperoxia-induced oxidative stress and increase in arterial stiffness in healthy humans. , 2009, Atherosclerosis.
[15] D. Jacobs,et al. Serum γ-glutamyltransferase: new insights about an old enzyme , 2009, Journal of Epidemiology & Community Health.
[16] Wei Zhang,et al. Consuming fructose-sweetened, not glucose-sweetened, beverages increases visceral adiposity and lipids and decreases insulin sensitivity in overweight/obese humans. , 2009, The Journal of clinical investigation.
[17] L. Gesualdo,et al. Systemic inflammation, metabolic syndrome and progressive renal disease. , 2009, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.
[18] W. Goessling,et al. Aminotransferase levels and 20-year risk of metabolic syndrome, diabetes, and cardiovascular disease. , 2008, Gastroenterology.
[19] S. Kaser,et al. Retinol‐binding Protein 4, Visceral Fat, and the Metabolic Syndrome: Effects of Weight Loss , 2008, Obesity.
[20] G. Marchesini,et al. Hyperinsulinemia and insulin resistance are independently associated with plasma lipids, uric acid and blood pressure in non-diabetic subjects. The GISIR database. , 2008, Nutrition, metabolism, and cardiovascular diseases : NMCD.
[21] Yu-Hung Chang,et al. Elevated serum retinol‐binding protein 4 concentrations are associated with renal dysfunction and uric acid in type 2 diabetic patients , 2008, Diabetes/metabolism research and reviews.
[22] R. Cecchini,et al. Influence of uric acid and gamma-glutamyltransferase on total antioxidant capacity and oxidative stress in patients with metabolic syndrome. , 2008, Nutrition.
[23] J. Shaw,et al. Should liver function tests be included in definitions of metabolic syndrome? Evidence from the association between liver function tests, components of metabolic syndrome and prevalent cardiovascular disease , 2008, Diabetic medicine : a journal of the British Diabetic Association.
[24] P. Havel,et al. Fructose consumption: potential mechanisms for its effects to increase visceral adiposity and induce dyslipidemia and insulin resistance , 2008, Current opinion in lipidology.
[25] N. Al-Daghri,et al. Adipocytokine profile of type 2 diabetics in metabolic syndrome as defined by various criteria , 2008, Diabetes/metabolism research and reviews.
[26] Y. Terauchi,et al. Hepatic fat content-independent association of the serum level of gamma-glutamyltransferase with visceral adiposity, but not subcutaneous adiposity. , 2008, Diabetes research and clinical practice.
[27] Takahiko Nakagawa,et al. Potential role of sugar (fructose) in the epidemic of hypertension, obesity and the metabolic syndrome, diabetes, kidney disease, and cardiovascular disease. , 2007, The American journal of clinical nutrition.
[28] G. Lippi,et al. Relationship between γ-glutamyltransferase, lipids and lipoprotein(a) in the general population , 2007 .
[29] M. Fasshauer,et al. Serum retinol-binding protein is more highly expressed in visceral than in subcutaneous adipose tissue and is a marker of intra-abdominal fat mass. , 2007, Cell metabolism.
[30] K. Adeli,et al. Fructose and the metabolic syndrome: pathophysiology and molecular mechanisms. , 2007, Nutrition reviews.
[31] G. Wolf. Serum retinol-binding protein: a link between obesity, insulin resistance, and type 2 diabetes. , 2007, Nutrition reviews.
[32] G. Lippi,et al. Relationship between gamma-glutamyltransferase, lipids and lipoprotein(a) in the general population. , 2007, Clinica chimica acta; international journal of clinical chemistry.
[33] A. Yamashina,et al. Elevated serum levels of alanine aminotransferase and gamma glutamyltransferase are markers of inflammation and oxidative stress independent of the metabolic syndrome. , 2006, Atherosclerosis.
[34] U. Smith,et al. Retinol-binding protein 4 and insulin resistance in lean, obese, and diabetic subjects. , 2006, The New England journal of medicine.
[35] D. Galaris,et al. Uric acid and oxidative stress. , 2005, Current pharmaceutical design.
[36] Nimesh Mody,et al. Serum retinol binding protein 4 contributes to insulin resistance in obesity and type 2 diabetes , 2005, Nature.
[37] P. Havel. Dietary fructose: implications for dysregulation of energy homeostasis and lipid/carbohydrate metabolism. , 2005, Nutrition reviews.
[38] F. Schick,et al. Elevated serum GGT concentrations predict reduced insulin sensitivity and increased intrahepatic lipids. , 2005, Hormone and metabolic research = Hormon- und Stoffwechselforschung = Hormones et metabolisme.
[39] R. Townsend,et al. Dietary fructose reduces circulating insulin and leptin, attenuates postprandial suppression of ghrelin, and increases triglycerides in women. , 2004, The Journal of clinical endocrinology and metabolism.
[40] G. Bray,et al. Consumption of high-fructose corn syrup in beverages may play a role in the epidemic of obesity. , 2004, The American journal of clinical nutrition.
[41] S. Genuth. Lowering the criterion for impaired fasting glucose is in order. , 2003, Diabetes care.
[42] J. Shaw,et al. Follow-up report on the diagnosis of diabetes mellitus. , 2003, Diabetes care.
[43] R. Townsend,et al. High-fat meals reduce 24-h circulating leptin concentrations in women. , 1999, Diabetes.
[44] P. Mayes,et al. Intermediary metabolism of fructose. , 1993, The American journal of clinical nutrition.
[45] M. Mifflin,et al. A new predictive equation for resting energy expenditure in healthy individuals. , 1990, The American journal of clinical nutrition.
[46] K. Ellwood,et al. Blood lipids, lipoproteins, apoproteins, and uric acid in men fed diets containing fructose or high-amylose cornstarch. , 1989, The American journal of clinical nutrition.
[47] R. Teschke,et al. Hepatic gamma-glutamyltransferase activity in alcoholic fatty liver: comparison with other liver enzymes in man and rats. , 1983, Gut.
[48] D. Goldberg,et al. Gamma-glutamyl transpeptidase, triglycerides, and enzyme induction. , 1975, British medical journal.
[49] B. Emmerson. Effect of oral fructose on urate production. , 1974, Annals of the rheumatic diseases.