California Wildfires of 2008: Coarse and Fine Particulate Matter Toxicity
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[1] K. Akakura. [Inflammatory cytokines]. , 2011, Nihon rinsho. Japanese journal of clinical medicine.
[2] Lars Barregard,et al. Population exposure to fine particles and estimated excess mortality in Finland from an East European wildfire episode , 2009, Journal of Exposure Science and Environmental Epidemiology.
[3] J. Last,et al. Lung response to coarse PM: bioassay in mice. , 2008, Toxicology and applied pharmacology.
[4] W. Zin,et al. Composition of Diesel Particles Influences Acute Pulmonary Toxicity: An Experimental Study in MICE , 2008, Inhalation toxicology.
[5] W. Zin,et al. Composition of diesel particles influences acute pulmonary toxicity: an experimental study in mice. , 2008, Inhalation toxicology.
[6] Chris Piacitelli,et al. Particle size-dependent radical generation from wildland fire smoke. , 2007, Toxicology.
[7] M. Brauer,et al. Woodsmoke Health Effects: A Review , 2007, Inhalation toxicology.
[8] R. Hillamo,et al. In vitro inflammatory and cytotoxic effects of size-segregated particulate samples collected during long-range transport of wildfire smoke to Helsinki. , 2006, Toxicology and applied pharmacology.
[9] A. Ledbetter,et al. Comparative pulmonary toxicological assessment of oil combustion particles following inhalation or instillation exposure. , 2006, Toxicological sciences : an official journal of the Society of Toxicology.
[10] K. Donaldson,et al. Explorer In vivo and in vitro proinflammatory effects of particulate air pollution ( PM 10 ) , 2006 .
[11] David L. Crow. San Joaquin Valley Air Pollution Control District Air Pollution Control Officer’s Determination of VOC Emission Factors for Dairies , 2005 .
[12] J. Veranth,et al. Inflammatory cytokines and cell death in BEAS-2B lung cells treated with soil dust, lipopolysaccharide, and surface-modified particles. , 2004, Toxicological sciences : an official journal of the Society of Toxicology.
[13] J. Weiss,et al. Quinones and Aromatic Chemical Compounds in Particulate Matter Induce Mitochondrial Dysfunction: Implications for Ultrafine Particle Toxicity , 2004, Environmental health perspectives.
[14] F. Cassee,et al. Ambient fine and coarse particle suppression of alveolar macrophage functions. , 2003, Toxicology letters.
[15] J. Antonini,et al. Residual oil fly ash increases the susceptibility to infection and severely damages the lungs after pulmonary challenge with a bacterial pathogen. , 2002, Toxicological sciences : an official journal of the Society of Toxicology.
[16] B. Jordan,et al. Indices of antioxidant status in rats subjected to wood smoke inhalation and/or thermal injury. , 2002, Toxicology.
[17] J. Zelikoff,et al. THE TOXICOLOGY OF INHALED WOODSMOKE , 2002, Journal of toxicology and environmental health. Part B, Critical reviews.
[18] Irfan Rahman,et al. PM10-exposed macrophages stimulate a proinflammatory response in lung epithelial cells via TNF-α , 2002 .
[19] W. MacNee,et al. PM(10)-exposed macrophages stimulate a proinflammatory response in lung epithelial cells via TNF-alpha. , 2002, American journal of physiology. Lung cellular and molecular physiology.
[20] Jerold A. Last,et al. TOXIC RESPONSES OF THE RESPIRATORY SYSTEM , 2001 .
[21] Xianglin Shi,et al. Wood smoke particles generate free radicals and cause lipid peroxidation, DNA damage, NFκB activation and TNF-α release in macrophages , 2000 .
[22] A. Imrich,et al. Insoluble components of concentrated air particles mediate alveolar macrophage responses in vitro. , 2000, Toxicology and applied pharmacology.
[23] B. Jordan,et al. Wood smoke particles generate free radicals and cause lipid peroxidation, DNA damage, NFkappaB activation and TNF-alpha release in macrophages. , 2000, Toxicology.
[24] N. Urosevic,et al. Variations in LPS responsiveness among different mouse substrains of C3H lineage and their congenic derivative sublines , 1999, Immunogenetics.
[25] I. Adamson,et al. Pulmonary toxicity of an atmospheric particulate sample is due to the soluble fraction. , 1999, Toxicology and applied pharmacology.
[26] C. Monn,et al. Cytotoxicity and induction of proinflammatory cytokines from human monocytes exposed to fine (PM2.5) and coarse particles (PM10-2.5) in outdoor and indoor air. , 1999, Toxicology and applied pharmacology.
[27] J. Carter,et al. Cytokine production by human airway epithelial cells after exposure to an air pollution particle is metal-dependent. , 1997, Toxicology and applied pharmacology.
[28] W. MacNee,et al. In vivo and in vitro proinflammatory effects of particulate air pollution (PM10). , 1997, Environmental health perspectives.
[29] C. Sioutas,et al. Alveolar macrophage interaction with air pollution particulates. , 1997, Environmental health perspectives.
[30] J. Timbrell. Casarett and Doull's Toxicology: The Basic Science of Poisons , 1981 .