Interaction of Toll-like receptors with bacterial components induces expression of CDX2 and MUC2 in rat biliary epithelium in vivo and in culture
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Yasunori Sato | Y. Nakanuma | K. Harada | M. Sasaki | H. Ikeda | Y. Zen | A. Ishikawa | H. Kazumori
[1] D. Ahn,et al. TNF-alpha Activates MUC2 Transcription via NF-kappaB but Inhibits via JNK Activation , 2005, Cellular Physiology and Biochemistry.
[2] Y. Jan,et al. Aberrant expression of CDX2 is closely related to the intestinal metaplasia and MUC2 expression in intraductal papillary neoplasm of the liver in hepatolithiasis , 2004, Laboratory Investigation.
[3] K. Tsuneyama,et al. Molecular identification of bacterial 16S ribosomal RNA gene in liver tissue of primary biliary cirrhosis: Is Propionibacterium acnes involved in granuloma formation? , 2001, Hepatology.
[4] T. Terada,et al. Are hepatolithiasis and cholangiocarcinoma aetiologically related? , 1985, Virchows Archiv A.
[5] W. Ewig. [Diseases of the bile ducts]. , 1961, Hippokrates.
[6] K. Sugano,et al. Aberrant Expression of CDX2 in the Gastric Mucosa With and Without Intestinal Metaplasia: Effect of Eradication of Helicobacter pylori , 2002, Helicobacter.
[7] T. Terada,et al. Pathologic features of hepatolithiasis in Japan. , 1988, Human pathology.
[8] Y. Jan,et al. Different expression patterns of mucin core proteins and cytokeratins during intrahepatic cholangiocarcinogenesis from biliary intraepithelial neoplasia and intraductal papillary neoplasm of the bile duct--an immunohistochemical study of 110 cases of hepatolithiasis. , 2006, Journal of hepatology.
[9] K. Sugano,et al. Expression of homeobox gene CDX2 precedes that of CDX1 during the progression of intestinal metaplasia , 2002, Journal of Gastroenterology.
[10] S. Fan,et al. Malignancy associated with hepatolithiasis. , 1997, Hepato-gastroenterology.
[11] M. Arditi,et al. Bacterial Lipopolysaccharide Activates NF-κB through Toll-like Receptor 4 (TLR-4) in Cultured Human Dermal Endothelial Cells , 2000, The Journal of Biological Chemistry.
[12] J. Goldberg,et al. Toll‐like Receptor 2‐Mediated Gene Expression in Epithelial Cells During Helicobacter pylori Infection , 2005, Helicobacter.
[13] T. Terada,et al. Morphological examination of intrahepatic bile ducts in hepatolithiasis , 2004, Virchows Archiv A.
[14] Hiroshi Yamamoto,et al. Homeodomain protein CDX2 regulates goblet-specific MUC2 gene expression. , 2003, Biochemical and biophysical research communications.
[15] S. Sheen-Chen,et al. Bacteriology and antimicrobial choice in hepatolithiasis. , 2000, American journal of infection control.
[16] N. Ishimura,et al. Essential Role of MD-2 in TLR4-Dependent Signaling during Helicobacter pylori-Associated Gastritis , 2004, The Journal of Immunology.
[17] K. Kaestner,et al. Cdx2 ectopic expression induces gastric intestinal metaplasia in transgenic mice. , 2002, Gastroenterology.
[18] C. Potten,et al. Helicobacter pylori regulates the expression of inhibitors of DNA binding (Id) proteins by gastric epithelial cells. , 2006, Microbes and infection.
[19] T. Terada,et al. ‘Metaplastic lesions’ in intrahepatic bile ducts in hepatolithiasis: A histochemical and immunohistochemical study , 1990, Journal of gastroenterology and hepatology.
[20] C. Moskaluk,et al. CDX2 and MUC2 protein expression in extrahepatic bile duct carcinoma. , 2005, American journal of clinical pathology.
[21] K. Tsuneyama,et al. Lipopolysaccharide induces overexpression of MUC2 and MUC5AC in cultured biliary epithelial cells: possible key phenomenon of hepatolithiasis. , 2002, The American journal of pathology.
[22] Kazuhiko Takeuchi,et al. Pranlukast Inhibits NF-κB Activation and MUC2 Gene Expression in Cultured Human Epithelial Cells , 2005, Pharmacology.
[23] Y. Kadowaki,et al. Bile acids directly augment caudal related homeobox gene Cdx2 expression in oesophageal keratinocytes in Barrett’s epithelium , 2005, Gut.
[24] L. Ziegler‐Heitbrock,et al. Tolerance Induced by the Lipopeptide Pam3Cys Is Due to Ablation of IL-1R-Associated Kinase-11 , 2004, The Journal of Immunology.
[25] Y. Jan,et al. Increasing expression of gastrointestinal phenotypes and p53 along with histologic progression of intraductal papillary neoplasia of the liver. , 2002, Human pathology.
[26] Y. Jan,et al. Characterization of intrahepatic cholangiocarcinoma of the intraductal growth‐type and its precursor lesions , 2005, Hepatology.
[27] Y. Nakanuma,et al. Polycystic kidney rat is a novel animal model of Caroli's disease associated with congenital hepatic fibrosis. , 2001, The American journal of pathology.
[28] T. Shuto,et al. Transforming Growth Factor-β-Smad Signaling Pathway Cooperates with NF-κB to Mediate NontypeableHaemophilus influenzae-induced MUC2 Mucin Transcription* , 2002, The Journal of Biological Chemistry.
[29] M. Marchetti,et al. Chronic acid exposure leads to activation of the cdx2 intestinal homeobox gene in a long-term culture of mouse esophageal keratinocytes , 2003, Journal of Cell Science.
[30] F. Nakayama,et al. Bacteriology of hepatolithiasis. , 1984, Progress in clinical and biological research.
[31] Y. Jan,et al. Intraductal papillary neoplasia of the liver associated with hepatolithiasis , 2001, Hepatology.
[32] Y. Kim,et al. Expression of apomucins in the intrahepatic biliary tree in hepatolithiasis differs from that in normal liver and extrahepatic biliary obstruction , 1998, Hepatology.
[33] K. Isse,et al. Lipopolysaccharide Activates Nuclear Factor-KappaB through Toll-Like Receptors and Related Molecules in Cultured Biliary Epithelial Cells , 2003, Laboratory Investigation.
[34] 龍田 健. Expression of Cdx2 in early GRCL of Barrett's esophagus induced in rats by duodenal reflux , 2005 .
[35] Y. Nakanuma,et al. Expression Profiles of MUC Mucin Core Protein in the Intrahepatic Biliary System : Physiological Distribution and Pathological Significance , 2005 .
[36] Jian-Dong Li,et al. Activation of NF-kappaB via a Src-dependent Ras-MAPK-pp90rsk pathway is required for Pseudomonas aeruginosa-induced mucin overproduction in epithelial cells. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[37] K. Sugano,et al. Aberrant expression of CDX2 in Barrett's epithelium and inflammatory esophageal mucosa , 2003, Journal of Gastroenterology.
[38] K. Yamaguchi,et al. Hepatolithiasis associated with cholangiocarcinoma. Possible etiologic significance , 1985, Cancer.
[39] H. Ishinaga,et al. Roxithromycin Suppresses Mucin Gene Expression in Epithelial Cells , 2004, Pharmacology.
[40] Y. Yen,et al. Immunohistochemical Staining in the Diagnosis of Pancreatobiliary and Ampulla of Vater Adenocarcinoma: Application of CDX2, CK17, MUC1, and MUC2 , 2005, The American journal of surgical pathology.
[41] M. Rothe,et al. Peptidoglycan- and Lipoteichoic Acid-induced Cell Activation Is Mediated by Toll-like Receptor 2* , 1999, The Journal of Biological Chemistry.