Impact of type 2 diabetes on vascular reactivity to cGMP generators in human internal thoracic arteries.

[1]  T. Asai,et al.  Endothelial dysfunction of internal thoracic artery graft in patients with chronic kidney disease , 2017, The Journal of thoracic and cardiovascular surgery.

[2]  T. Okamura,et al.  Soluble guanylate cyclase redox state under oxidative stress conditions in isolated monkey coronary arteries , 2016, Pharmacology research & perspectives.

[3]  T. Imamura,et al.  Effects of hydrogen peroxide on relaxation through the NO/sGC/cGMP pathway in isolated rat iliac arteries , 2015, Free radical research.

[4]  Yingzi Zhao,et al.  Vascular nitric oxide: Beyond eNOS. , 2015, Journal of pharmacological sciences.

[5]  T. Imamura,et al.  Influence of smoking on vascular reactivity to cGMP generators in human internal thoracic arteries , 2015, BMC Pharmacology and Toxicology.

[6]  A. Ergul,et al.  Reduced vascular responses to soluble guanylyl cyclase but increased sensitivity to sildenafil in female rats with type 2 diabetes. , 2015, American journal of physiology. Heart and circulatory physiology.

[7]  J. D. de Haan,et al.  Are reactive oxygen species still the basis for diabetic complications? , 2015, Clinical science.

[8]  B. Eliasson,et al.  Long-term prognosis in patients with type 1 and 2 diabetes mellitus after coronary artery bypass grafting. , 2015, Journal of the American College of Cardiology.

[9]  T. Imamura,et al.  Different Influences of Extracellular and Intracellular Superoxide on Relaxation Through the NO/sGC/cGMP Pathway in Isolated Rat Iliac Arteries , 2014, Journal of cardiovascular pharmacology.

[10]  T. Imamura,et al.  Effects of Peroxynitrite on Relaxation through the NO/sGC/cGMP Pathway in Isolated Rat Iliac Arteries , 2015, Journal of Vascular Research.

[11]  G. De Nucci,et al.  Soluble Guanylyl Cyclase (sGC) Degradation and Impairment of Nitric Oxide-Mediated Responses in Urethra from Obese Mice: Reversal by the sGC Activator BAY 60-2770 , 2014, The Journal of Pharmacology and Experimental Therapeutics.

[12]  M. Cooper,et al.  Mechanisms of diabetic complications. , 2013, Physiological reviews.

[13]  G. De Nucci,et al.  Immunohistochemical and functional characterization of nitric oxide signaling pathway in isolated aorta from Crotalus durissus terrificus. , 2012, Comparative biochemistry and physiology. Toxicology & pharmacology : CBP.

[14]  K. Matschke,et al.  Preservation of human artery function following prolonged cold storage with a new solution. , 2011, Journal of vascular surgery.

[15]  Kenneth J. Doka,et al.  The Role of Gender , 2011 .

[16]  P. McKeown,et al.  Role of gender, smoking profile, hypertension, and diabetes on saphenous vein and internal mammary artery endothelial relaxation in patients with coronary artery bypass grafting , 2010, Oxidative medicine and cellular longevity.

[17]  T. Carrel,et al.  Human internal thoracic arteries from diabetic patients are resistant to endothelial dysfunction , 2009, Fundamental & clinical pharmacology.

[18]  P. Tourneux,et al.  Cinaciguat, a soluble guanylate cyclase activator, causes potent and sustained pulmonary vasodilation in the ovine fetus. , 2009, American journal of physiology. Lung cellular and molecular physiology.

[19]  J. Stasch,et al.  Nitric Oxide-independent Activation of Soluble Guanylate Cyclase by BAY 60-2770 in Experimental Liver Fibrosis , 2008, Arzneimittel-Forschung (Drug Research).

[20]  D. Hess,et al.  Inhaled agonists of soluble guanylate cyclase induce selective pulmonary vasodilation. , 2007, American journal of respiratory and critical care medicine.

[21]  J. Catravas,et al.  Nitric oxide and the endothelium: history and impact on cardiovascular disease. , 2006, Vascular pharmacology.

[22]  V. Mahadevan,et al.  Internal mammary artery smooth muscle cells resist migration and possess high antioxidant capacity. , 2006, Cardiovascular research.

[23]  O. V. Evgenov,et al.  NO-independent stimulators and activators of soluble guanylate cyclase: discovery and therapeutic potential , 2006, Nature Reviews Drug Discovery.

[24]  J. Stasch,et al.  Targeting the heme-oxidized nitric oxide receptor for selective vasodilatation of diseased blood vessels. , 2006, The Journal of clinical investigation.

[25]  W. Linz,et al.  Biochemistry and Pharmacology of Novel Anthranilic Acid Derivatives Activating Heme-Oxidized Soluble Guanylyl Cyclase , 2006, Molecular Pharmacology.

[26]  J. Dunning,et al.  What is the optimal vasodilator for preventing spasm in the left internal mammary artery during coronary arterial bypass grafting? , 2005, Interactive cardiovascular and thoracic surgery.

[27]  B. McManus,et al.  Compromised arterial function in human type 2 diabetic patients. , 2005, Diabetes.

[28]  O. Yildiz,et al.  The Relationship Between Risk Factors and Testosterone-Induced Relaxations in Human Internal Mammary Artery , 2005, Journal of cardiovascular pharmacology.

[29]  C. Vahl,et al.  Nitric oxide-sensitive soluble guanylyl cyclase activity is preserved in internal mammary artery of type 2 diabetic patients. , 2004, Diabetes.

[30]  R. Raddino,et al.  Influence of type 2 diabetes on functional and structural properties of coronary artery bypass conduits. , 2003, Diabetes.

[31]  H. Schäfers,et al.  Vasoreactivity of arterial grafts in the patient with diabetes mellitus: investigations on internal thoracic artery and radial artery conduits. , 2001, European journal of cardio-thoracic surgery : official journal of the European Association for Cardio-thoracic Surgery.

[32]  D. Harrison,et al.  Superoxide production, risk factors, and endothelium-dependent relaxations in human internal mammary arteries. , 1999, Circulation.

[33]  G. Fraedrich,et al.  Increased tissue endothelin-1-like immunoreactivity in the internal mammary artery of patients with diabetes or hypercholesterolemia modulates the graft flow in the peri-operative period. , 1998, European journal of cardio-thoracic surgery : official journal of the European Association for Cardio-thoracic Surgery.

[34]  A. Voors,et al.  Dyslipidemia and endothelium-dependent relaxation in internal mammary arteries used for coronary bypass surgery. , 1997, Cardiovascular research.

[35]  B. Mayer,et al.  Characterization of 1H-[1,2,4]oxadiazolo[4,3-a]quinoxalin-1-one as a heme-site inhibitor of nitric oxide-sensitive guanylyl cyclase. , 1996, Molecular pharmacology.

[36]  H. Soncul,et al.  Effects of non-insulin dependent diabetes mellitus on the reactivity of human internal mammary artery and human saphenous vein. , 1995, Life sciences.

[37]  G. Angelini,et al.  Overcoming perioperative spasm of the internal mammary artery: which is the best vasodilator? , 1992, The Journal of thoracic and cardiovascular surgery.

[38]  B. Toth Internal mammary artery graft. , 1987, The Journal of thoracic and cardiovascular surgery.

[39]  P. O'brien Superoxide production. , 1984, Methods in enzymology.

[40]  G. Johnson,et al.  The internal mammary artery graft. Its longevity after coronary bypass. , 1981, JAMA.