Gene Expression Deficits in a Subclass of GABA Neurons in the Prefrontal Cortex of Subjects with Schizophrenia
暂无分享,去创建一个
A. Sampson | J. Pierri | T. Hashimoto | D. Volk | K. Mirnics | Zhuoxin Sun | S. Eggan | D. Lewis
[1] K Maeda,et al. Abnormal expression of brain-derived neurotrophic factor and its receptor in the corticolimbic system of schizophrenic patients , 2000, Molecular Psychiatry.
[2] A. Sampson,et al. Decreased glutamic acid decarboxylase67 messenger RNA expression in a subset of prefrontal cortical gamma-aminobutyric acid neurons in subjects with schizophrenia. , 2000, Archives of general psychiatry.
[3] Paul J. Harrison,et al. The relative importance of premortem acidosis and postmortem interval for human brain gene expression studies: selective mRNA vulnerability and comparison with their encoded proteins , 1995, Neuroscience Letters.
[4] Robert Zöchling,et al. Brain-derived neurotrophic factor and neurotrophin 3 in schizophrenic psychoses , 2001, Schizophrenia Research.
[5] P. Goldman-Rakic,et al. The synaptology of parvalbumin‐immunoreactive neurons in the primate prefrontal cortex , 1992, The Journal of comparative neurology.
[6] P. Goldman-Rakic,et al. Destruction and Creation of Spatial Tuning by Disinhibition: GABAA Blockade of Prefrontal Cortical Neurons Engaged by Working Memory , 2000, The Journal of Neuroscience.
[7] A. Sampson,et al. Lamina-specific alterations in the dopamine innervation of the prefrontal cortex in schizophrenic subjects. , 1999, The American journal of psychiatry.
[8] J John Mann,et al. The GABAergic system in schizophrenia. , 2002, The international journal of neuropsychopharmacology.
[9] D. L. Martin,et al. Two isoforms of glutamate decarboxylase: why? , 1998, Trends in pharmacological sciences.
[10] J J Bartko,et al. Molecular abnormalities in the major psychiatric illnesses: Classification and Regression Tree (CRT) analysis of post-mortem prefrontal markers , 2002, Molecular Psychiatry.
[11] T. Yagi,et al. Cleft palate and decreased brain gamma-aminobutyric acid in mice lacking the 67-kDa isoform of glutamic acid decarboxylase. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[12] R. Mohs,et al. Consortium to establish a registry for Alzheimer's disease (CERAD) clinical and neuropsychological assessment of Alzheimer's disease. , 2002, Psychopharmacology bulletin.
[13] S. Hirsch,et al. Reduced dendritic spine density on cerebral cortical pyramidal neurons in schizophrenia , 1998, Journal of neurology, neurosurgery, and psychiatry.
[14] R. Miller,et al. Calcium buffering properties of calbindin D28k and parvalbumin in rat sensory neurones. , 1993, The Journal of physiology.
[15] T. Woo,et al. A subclass of prefrontal gamma-aminobutyric acid axon terminals are selectively altered in schizophrenia. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[16] J. Morris,et al. The Consortium to Establish a Registry for Alzheimer's Disease (CERAD). Part XIII. , 1996, Neurology.
[17] K. Grzeschik,et al. The genes for the highly homologous Ca(2+)-binding proteins oncomodulin and parvalbumin are not linked in the human genome. , 1992, Genomics.
[18] Y. Kubota,et al. GABAergic cell subtypes and their synaptic connections in rat frontal cortex. , 1997, Cerebral cortex.
[19] David A Lewis,et al. Pyramidal neuron local axon terminals in monkey prefrontal cortex: differential targeting of subclasses of GABA neurons. , 2003, Cerebral cortex.
[20] R. Littell. SAS System for Mixed Models , 1996 .
[21] S. Hendry,et al. Activity-dependent regulation of GABA expression in the visual cortex of adult monkeys , 1988, Neuron.
[22] J. Hallmayer,et al. Chromosome 22 workshop report. , 1999, American journal of medical genetics.
[23] J. Pierri,et al. Alterations in chandelier neuron axon terminals in the prefrontal cortex of schizophrenic subjects. , 1999, The American journal of psychiatry.
[24] P. Goldman-Rakic,et al. Prefrontal Microcircuits: Membrane Properties and Excitatory Input of Local, Medium, and Wide Arbor Interneurons , 2001, The Journal of Neuroscience.
[25] S. Daviss,et al. Local circuit neurons of the prefrontal cortex in schizophrenia: selective increase in the density of calbindin-immunoreactive neurons , 1995, Psychiatry Research.
[26] A. Sampson,et al. GABA transporter-1 mRNA in the prefrontal cortex in schizophrenia: decreased expression in a subset of neurons. , 2001, The American journal of psychiatry.
[27] J. Pierri,et al. Gene Expression Profiling Reveals Alterations of Specific Metabolic Pathways in Schizophrenia , 2002, The Journal of Neuroscience.
[28] J. Lund,et al. Local circuit neurons of developing and mature macaque prefrontal cortex: Golgi and immunocytochemical characteristics , 1993, The Journal of comparative neurology.
[29] M. Huntsman,et al. Activity-dependent changes in GAD and preprotachykinin mRNAs in visual cortex of adult monkeys. , 1994, Cerebral cortex.
[30] V. Barnett,et al. Applied Linear Statistical Models , 1975 .
[31] J. Pierri,et al. Lamina-specific deficits in parvalbumin-immunoreactive varicosities in the prefrontal cortex of subjects with schizophrenia: evidence for fewer projections from the thalamus. , 2001, The American journal of psychiatry.
[32] German Barrionuevo,et al. Synaptic targets of the intrinsic axon collaterals of supragranular pyramidal neurons in monkey prefrontal cortex , 2001, The Journal of comparative neurology.
[33] L. Glantz,et al. Normal cellular levels of synaptophysin mRNA expression in the prefrontal cortex of subjects with schizophrenia , 2000, Biological Psychiatry.
[34] D. Lewis,et al. Parvalbumin‐immunoreactive axon terminals in macaque monkey and human prefrontal cortex: Laminar, regional, and target specificity of type I and type II synapses , 1999, The Journal of comparative neurology.
[35] D. Lewis,et al. Decreased dendritic spine density on prefrontal cortical pyramidal neurons in schizophrenia. , 2000, Archives of general psychiatry.
[36] Hans R. Gelderblom,et al. Enforcement of Temporal Fidelity in Pyramidal Cells by Somatic Feed-Forward Inhibition , 2001 .
[37] Pat Levitt,et al. Molecular Characterization of Schizophrenia Viewed by Microarray Analysis of Gene Expression in Prefrontal Cortex , 2000, Neuron.
[38] P. Pedarzani,et al. Medium afterhyperpolarization and firing pattern modulation in interneurons of stratum radiatum in the CA3 hippocampal region. , 2001, Journal of neurophysiology.
[39] F. Benes,et al. Differential hippocampal expression of glutamic acid decarboxylase 65 and 67 messenger RNA in bipolar disorder and schizophrenia. , 2002, Archives of general psychiatry.
[40] Françoise Condé,et al. Local circuit neurons immunoreactive for calretinin, calbindin D‐28k or parvalbumin in monkey prefronatal cortex: Distribution and morphology , 1994, The Journal of comparative neurology.
[41] P. Goldman-Rakic,et al. A role for inhibition in shaping the temporal flow of information in prefrontal cortex , 2002, Nature Neuroscience.
[42] O. Caillard,et al. Role of the calcium-binding protein parvalbumin in short-term synaptic plasticity. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[43] S. Hendry,et al. Regulation of calcium-binding protein immunoreactivity in GABA neurons of macaque primary visual cortex. , 1996, Cerebral cortex.
[44] M. Wong-Riley,et al. Metabolic and neurochemical plasticity of γ‐aminobutyric acid‐immunoreactive neurons in the adult macaque striate cortex following monocular impulse blockade: Quantitative electron microscopic analysis , 1996, The Journal of comparative neurology.
[45] L. Squire,et al. The medial temporal lobe memory system , 1991, Science.
[46] C. Beasley,et al. Selective deficits in prefrontal cortical GABAergic neurons in schizophrenia defined by the presence of calcium-binding proteins , 2002, Biological Psychiatry.
[47] David A Lewis,et al. Intrinsic excitatory connections in the prefrontal cortex and the pathophysiology of schizophrenia , 2000, Brain Research Bulletin.
[48] T. Woo,et al. Schizophrenia and the parvalbumin-containing class of cortical local circuit neurons. , 1997, The American journal of psychiatry.
[49] J. Cox,et al. The Ca2+(-)binding proteins parvalbumin and oncomodulin and their genes: new structural and functional findings. , 1996, Biochimica et biophysica acta.
[50] David A Lewis,et al. Catching Up on Schizophrenia Natural History and Neurobiology , 2000, Neuron.
[51] P. Goldman-Rakic. Working memory dysfunction in schizophrenia. , 1994, The Journal of neuropsychiatry and clinical neurosciences.
[52] Yogesh K. Dwivedi,et al. Decrease in reelin and glutamic acid decarboxylase67 (GAD67) expression in schizophrenia and bipolar disorder: a postmortem brain study. , 2000, Archives of general psychiatry.
[53] T. Yagi,et al. Mice lacking the 65 kDa isoform of glutamic acid decarboxylase (GAD65) maintain normal levels of GAD67 and GABA in their brains but are susceptible to seizures. , 1996, Biochemical and biophysical research communications.
[54] P. McGuffin,et al. Linkage and associated studies of schizophrenia. , 2000, American journal of medical genetics.
[55] E. G. Jones,et al. Gene expression for glutamic acid decarboxylase is reduced without loss of neurons in prefrontal cortex of schizophrenics. , 1995, Archives of general psychiatry.
[56] D. Weinberger,et al. Physiologic dysfunction of dorsolateral prefrontal cortex in schizophrenia. I. Regional cerebral blood flow evidence. , 1986, Archives of general psychiatry.
[57] P. Somogyi,et al. Synchronization of neuronal activity in hippocampus by individual GABAergic interneurons , 1995, Nature.
[58] B. Berninger,et al. Neurotrophins and activity-dependent plasticity of cortical interneurons , 1997, Trends in Neurosciences.
[59] Paul Leonard Gabbott,et al. Local circuit neurons in the medial prefrontal cortex (areas 24a,b,c, 25 and 32) in the monkey: I. Cell morphology and morphometrics , 1996, The Journal of comparative neurology.
[60] C. Gerfen,et al. Dopamine differentially regulates dynorphin, substance P, and enkephalin expression in striatal neurons: in situ hybridization histochemical analysis , 1991, The Journal of neuroscience : the official journal of the Society for Neuroscience.