Protective effect of exogenous 70-kDa heat shock protein during endotoxic shock (sepsis)

[1]  D. Butterfield,et al.  Redox regulation in neurodegeneration and longevity: role of the heme oxygenase and HSP70 systems in brain stress tolerance. , 2004, Antioxidants & redox signaling.

[2]  K. Triantafilou,et al.  Heat-shock protein 70 and heat-shock protein 90 associate with Toll-like receptor 4 in response to bacterial lipopolysaccharide. , 2004, Biochemical Society transactions.

[3]  P. Ward,et al.  The enigma of sepsis. , 2003, The Journal of clinical investigation.

[4]  A. De Maio,et al.  Geldanamycin treatment ameliorates the response to LPS in murine macrophages by decreasing CD14 surface expression. , 2003, Molecular biology of the cell.

[5]  A. Harken,et al.  The Interaction Between HSP70 and TNF-&agr; Expression: A Novel Mechanism for Protection of the Myocardium Against Post-Injury Depression , 2002, Shock.

[6]  X. Ding,et al.  Over-expression of hsp-70 inhibits bacterial lipopolysaccharide-induced production of cytokines in human monocyte-derived macrophages. , 2001, Cytokine.

[7]  R. Salomão,et al.  Pathogenetic aspects of sepsis and possible targets for adjunctive therapy. , 2001, Current Drug Targets - Immune, Endocrine & Metabolic Disorders.

[8]  K. Matsushima,et al.  The molecular pathogenesis of endotoxic shock and organ failure. , 1999, Molecular medicine today.

[9]  Z. Darieva,et al.  Effects of exogenous stress protein 70 on the functional properties of human promonocytes through binding to cell surface and internalization. , 1998, Cell stress & chaperones.

[10]  T. van der Poll,et al.  The cytokine‐mediated imbalance between coagulant and anticoagulant mechanisms in sepsis and endotoxaemia , 1997, European journal of clinical investigation.

[11]  R. Hotchkiss,et al.  EFFECT OF ETHANOL AND SODIUM ARSENITE ON HSP‐72 FORMATION AND ON SURVIVAL IN A MURINE ENDOTOXIN MODEL , 1994, Shock.