Alterations in different populations of striatal dopamine receptors produced by 18 months continuous administration of cis- or trans-flupenthixol to rats

[1]  C. Marsden,et al.  Increased striatal acetylcholine after 14 months cis-flupenthixol treatment in rats suggests functional supersensitivity of dopamine receptors. , 1982, Life sciences.

[2]  P. Jenner,et al.  Chronic continuous administration of neuroleptic drugs alters cerebral dopamine receptors and increases spontaneous dopaminergic action in the striatum , 1982, Nature.

[3]  M. Gnegy,et al.  Chronic haloperidol treatment increased calcium-dependent phosphorylation in rat striatum. , 1982, Life sciences.

[4]  J. Leysen,et al.  Optimal Conditions for [3H]Apomorphine Binding and Anomalous Equilibrium Binding of [3H]Apomorphine and [3H]Spiperone to Rat Striatal Membranes: Involvement of Surface Phenomena Versus Multiple Binding Sites , 1981, Journal of neurochemistry.

[5]  J. Leysen.  Review on neuroleptic receptors: specificity and multiplicity of in vitro binding related to pharmacological activity , 1981 .

[6]  P. Seeman.  Brain dopamine receptors. , 1980, Pharmacological reviews.

[7]  C. Marsden,et al.  Changes in rat striatal dopamine turnover and receptor activity during one years neuroleptic administration. , 1980, European journal of pharmacology.

[8]  J. Waddington,et al.  Functional distinction between DA-stimulated adenylate cyclase and 3H-spiperone binding sites in rat striatum. , 1979, European journal of pharmacology.

[9]  C. Marsden,et al.  Changes in dopamine-mediated behaviour during one year's neuroleptic administration. , 1979, European journal of pharmacology.

[10]  J. Kebabian,et al.  Multiple receptors for dopamine , 1979, Nature.

[11]  J. Leysen,et al.  Spiperone: a ligand of choice for neuroleptic receptors. 1. Kinetics and characteristics of in vitro binding. , 1978, Biochemical pharmacology.

[12]  B. Scatton.  Differential regional development of tolerance to increase in dopamine turnover upon repeated neuroleptic administration. , 1977, European journal of pharmacology.

[13]  C. Marsden,et al.  Substituted benzamides as cerebral dopamine antagonists in rodents , 1977, Neuropharmacology.

[14]  S. Snyder,et al.  Antischizophrenic drugs: chronic treatment elevates dopamine receptor binding in brain. , 1977, Science.

[15]  C. Marsden,et al.  Stereotyped behaviour patterns and hyperactivity induced by amphetamine and apomorphine after discrete 6-hydroxydopamine lesions of extrapyramidal and mesolimbic nuclei , 1977, Brain Research.

[16]  A. M. Johnson,et al.  STIMULANT PROPERTIES OF BROMOCRIPTINE ON CENTRAL DOPAMINE RECEPTORS IN COMPARISON TO APOMORPHINE, (+)‐AMPHETAMINE AND l‐DOPA , 1976, British journal of pharmacology.

[17]  R. Baldessarini,et al.  Behavioural supersensitivity to apomorphine following chronic treatment with drugs which interfere with the synaptic function of catecholamines. , 1974, Neuropharmacology.

[18]  L. Iversen,et al.  The Action of Neuroleptic Drugs on Dopamine-Stimulated Adenosine Cyclic 3',5'-Monophosphate Production in Rat Neostriatum and Limbic Forebrain , 1974 .

[19]  M. Baggiolini,et al.  Tolerance phenomena with neuroleptics catalepsy, apomorphine stereotypies and striatal dopamine metabolism in the rat after single and repeated administration of loxapine and haloperidol. , 1973, European journal of pharmacology.