Febrile-range temperature modifies cytokine gene expression in LPS-stimulated macrophages by differentially modifying NF-{kappa}B recruitment to cytokine gene promoters.
暂无分享,去创建一个
Aditi Gupta | I. Benjamin | Z. Cooper | I. Singh | J. Hasday | S. Vogel | A. Ghosh | Stefanie N Vogel | T. Maity | Zachary A Cooper | Arundhati Ghosh | Aditi Gupta | Tapan Maity | Ivor J Benjamin | Jeffrey D Hasday | Ishwar S Singh
[1] M. Barton,et al. Hypoxia-induced and stress-specific changes in chromatin structure and function. , 2007, Mutation research.
[2] A. Stephanou,et al. Transcriptional regulation of the heat shock protein genes by STAT family transcription factors. , 1999, Gene expression.
[3] Liwu Li,et al. Endotoxin tolerance disrupts chromatin remodeling and NF-kappaB transactivation at the IL-1beta promoter. , 2005, Journal of Immunology.
[4] I. Singh,et al. Inhibition of tumor necrosis factor-alpha transcription in macrophages exposed to febrile range temperature. A possible role for heat shock factor-1 as a negative transcriptional regulator. , 2000, The Journal of biological chemistry.
[5] A. Nagarsekar,et al. Bacterial Pneumonia Lung Injury in Experimental Gram-Negative Neutrophil Accumulation and Enhances Febrile-Range Hyperthermia Augments , 2005 .
[6] V. Malhotra,et al. Heat shock inhibits activation of NF-kappaB in the absence of heat shock factor-1. , 2002, Biochemical and biophysical research communications.
[7] I. Singh,et al. A High Affinity HSF-1 Binding Site in the 5′-Untranslated Region of the Murine Tumor Necrosis Factor-α Gene Is a Transcriptional Repressor* , 2002, The Journal of Biological Chemistry.
[8] Liwu Li,et al. Endotoxin Tolerance Disrupts Chromatin Remodeling and NF-κB Transactivation at the IL-1β Promoter1 , 2005, The Journal of Immunology.
[9] H. Iwasaka,et al. Changes in cell culture temperature alter release of inflammatory mediators in murine macrophagic RAW264.7 cells , 2007, Inflammation Research.
[10] M. Fenton,et al. TLRs: differential adapter utilization by toll-like receptors mediates TLR-specific patterns of gene expression. , 2003, Molecular interventions.
[11] I. Singh,et al. Hyperthermia in the febrile range induces HSP72 expression proportional to exposure temperature but not to HSF-1 DNA-binding activity in human lung epithelial A549 cells , 2009, Cell Stress and Chaperones.
[12] K. Katayama,et al. Involvement of NF-kappaB p50/p65 heterodimer in activation of the human pro-interleukin-1beta gene at two subregions of the upstream enhancer element. , 1999, Cytokine.
[13] M. Stevenson,et al. Heat Shock Factor 1 Represses Transcription of theIL-1β Gene through Physical Interaction with the Nuclear Factor of Interleukin 6* , 2002, The Journal of Biological Chemistry.
[14] D. Hume,et al. Heat shock enhances transcriptional activation of the murine inducible nitric oxide synthase gene , 2000, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[15] S. Han,et al. Heat shock protein 70 negatively regulates the heat-shock-induced suppression of the IkappaB/NF-kappaB cascade by facilitating IkappaB kinase renaturation and blocking its further denaturation. , 2005, Experimental cell research.
[16] C. McCall,et al. Epigenetic Silencing of Tumor Necrosis Factor α during Endotoxin Tolerance* , 2007, Journal of Biological Chemistry.
[17] Daniel R. Caffrey,et al. LPS-TLR4 Signaling to IRF-3/7 and NF-κB Involves the Toll Adapters TRAM and TRIF , 2003, The Journal of experimental medicine.
[18] Lu Zhang,et al. Hsp70 promotes TNF-mediated apoptosis by binding IKKγ and impairing NF-κB survival signaling , 2004 .
[19] C. Yuan,et al. Potential targets for HSF1 within the preinitiation complex , 2000, Cell stress & chaperones.
[20] D. Isenberg,et al. Signal transducer and activator of transcription-1 and heat shock factor-1 interact and activate the transcription of the Hsp-70 and Hsp-90beta gene promoters. , 1999, The Journal of biological chemistry.
[21] V. Holers,et al. Modulation of Mouse Endotoxic Fever by Complement , 2002, Infection and Immunity.
[22] Thomas Unger,et al. Quantitative real-time RT-PCR data analysis: current concepts and the novel “gene expression’s CT difference” formula , 2006, Journal of Molecular Medicine.
[23] K. Katayama,et al. INVOLVEMENT OF NF-κB p50/p65 HETERODIMER IN ACTIVATION OF THE HUMAN PRO-INTERLEUKIN-1β GENE AT TWO SUBREGIONS OF THE UPSTREAM ENHANCER ELEMENT , 1999 .
[24] C. McCall,et al. Protein-tyrosine Kinase Activation Is Required for Lipopolysaccharide Induction of Interleukin 1β and NFκB Activation, but Not NFκB Nuclear Translocation* , 1996, The Journal of Biological Chemistry.
[25] I. Singh,et al. Inhibition of Tumor Necrosis Factor-α Transcription in Macrophages Exposed to Febrile Range Temperature , 2000, The Journal of Biological Chemistry.
[26] S. Han,et al. The heat-shock-induced suppression of the IkappaB/NF-kappaB cascade is due to inactivation of upstream regulators of IkappaBalpha through insolubilization. , 2004, Experimental cell research.
[27] M. Fenton,et al. Bacterial endotoxin modifies heat shock factor-1 activity in RAW 264.7 cells: implications for TNF-α regulation during exposure to febrile range temperatures , 2004, Journal of endotoxin research.
[28] W. Schaffner,et al. Rapid detection of octamer binding proteins with 'mini-extracts', prepared from a small number of cells. , 1989, Nucleic acids research.
[29] D. Isenberg,et al. Signal Transducer and Activator of Transcription-1 and Heat Shock Factor-1 Interact and Activate the Transcription of the Hsp-70 and Hsp-90β Gene Promoters* , 1999, The Journal of Biological Chemistry.
[30] S. Han,et al. The heat-shock-induced suppression of the IkappaB/NF-kappaB cascade is due to inactivation of upstream regulators of IkappaBalpha through insolubilization. , 2004, Experimental cell research.
[31] L. Mahadevan,et al. Distinct stimulus-specific histone modifications at hsp70 chromatin targeted by the transcription factor heat shock factor-1. , 2004, Molecular cell.
[32] Lu Zhang,et al. Hsp70 promotes TNF-mediated apoptosis by binding IKK gamma and impairing NF-kappa B survival signaling. , 2004, Genes & development.
[33] C. McCall,et al. ENDOTOXIN INDUCIBLE TRANSCRIPTION IS REPRESSED IN ENDOTOXIN TOLERANT CELLS , 2000, Shock.
[34] S. Han,et al. Heat shock protein 70 negatively regulates the heat-shock-induced suppression of the IkappaB/NF-kappaB cascade by facilitating IkappaB kinase renaturation and blocking its further denaturation. , 2005, Experimental cell research.
[35] Aditi Gupta,et al. Heat shock co-activates interleukin-8 transcription. , 2008, American journal of respiratory cell and molecular biology.
[36] A. Nagarsekar,et al. Febrile-Range Hyperthermia Augments Neutrophil Accumulation and Enhances Lung Injury in Experimental Gram-Negative Bacterial Pneumonia1 , 2005, The Journal of Immunology.
[37] Tsonwin Hai,et al. Heat Shock Transcription Factor 1 Opens Chromatin Structure of Interleukin-6 Promoter to Facilitate Binding of an Activator or a Repressor* , 2007, Journal of Biological Chemistry.
[38] C. McCall,et al. Protein-tyrosine kinase activation is required for lipopolysaccharide induction of interleukin 1beta and NFkappaB activation, but not NFkappaB nuclear translocation. , 1996, The Journal of biological chemistry.
[39] L. O’Neill,et al. Toll-like receptors: from the discovery of NFkappaB to new insights into transcriptional regulations in innate immunity. , 2006, Biochemical pharmacology.
[40] G. Dubyak,et al. Differential Requirement of P2X7 Receptor and Intracellular K+ for Caspase-1 Activation Induced by Intracellular and Extracellular Bacteria* , 2007, Journal of Biological Chemistry.
[41] I. Singh,et al. Fever and the heat shock response: distinct, partially overlapping processes , 2000, Cell stress & chaperones.
[42] K. Fairchild,et al. Effects of hypothermia and hyperthermia on cytokine production by cultured human mononuclear phagocytes from adults and newborns. , 2000, Journal of interferon & cytokine research : the official journal of the International Society for Interferon and Cytokine Research.
[43] J. Hasday,et al. Differential effects of hyperthermia on macrophage interleukin-6 and tumor necrosis factor-alpha expression. , 1994, The American journal of physiology.
[44] W. Welch,et al. Stress-Induced Inhibition of the NF-κB Signaling Pathway Results from the Insolubilization of the IκB Kinase Complex following Its Dissociation from Heat Shock Protein 901 , 2005, The Journal of Immunology.
[45] Stefania Hanau,et al. Purinergic Modulation of Interleukin-1  Release from Microglial Cells Stimulated with Bacterial Endotoxin Materials and Methods , 1997 .
[46] Yue Zhang,et al. The Interleukin-1β Gene Is Transcribed from a Poised Promoter Architecture in Monocytes* , 2006, Journal of Biological Chemistry.
[47] A. Kane,et al. Febrile-range temperature modifies early systemic tumor necrosis factor alpha expression in mice challenged with bacterial endotoxin. , 1999, Infection and immunity.
[48] C. Cahill,et al. Transcriptional repression of the prointerleukin 1beta gene by heat shock factor 1. , 1996, The Journal of biological chemistry.
[49] M. J. Cody,et al. TLR4, but not TLR2, mediates IFN-beta-induced STAT1alpha/beta-dependent gene expression in macrophages. , 2002, Nature immunology.
[50] V. Malhotra,et al. Heat Shock Inhibits Activation of NF-κB in the Absence of Heat Shock Factor-1 , 2002 .
[51] S. Akira,et al. TLR signaling. , 2006, Cell death and differentiation.
[52] H. Sarau,et al. Chemokine-Cytokine Cross-talk , 2003, The Journal of Biological Chemistry.
[53] D. McMillan,et al. HSF1 is required for extra‐embryonic development, postnatal growth and protection during inflammatory responses in mice , 1999, The EMBO journal.
[54] D. Hume,et al. NF-IL6 and HSF1 have mutually antagonistic effects on transcription in monocytic cells. , 2002, Biochemical and biophysical research communications.
[55] C. McCall,et al. Epigenetic silencing of tumor necrosis factor alpha during endotoxin tolerance. , 2007, The Journal of biological chemistry.
[56] Chinfei Chen,et al. Heat Shock Factor 1 Represses Ras-induced Transcriptional Activation of the c-fos Gene* , 1997, The Journal of Biological Chemistry.
[57] D. Tang,et al. THE INHIBITION OF LPS-INDUCED PRODUCTION OF INFLAMMATORY CYTOKINES BY HSP70 INVOLVES INACTIVATION OF THE NF-&kgr;B PATHWAY BUT NOT THE MAPK PATHWAYS , 2006, Shock.
[58] C V Jongeneel,et al. Kappa B-type enhancers are involved in lipopolysaccharide-mediated transcriptional activation of the tumor necrosis factor alpha gene in primary macrophages , 1990, The Journal of experimental medicine.