Association between OPG, RANK and RANKL gene polymorphisms and susceptibility to acute coronary syndrome in Korean population

[1]  B. Coll,et al.  RANKL Increases Vascular Smooth Muscle Cell Calcification Through a RANK-BMP4–Dependent Pathway , 2009, Circulation research.

[2]  J. Marc,et al.  Tumour necrosis factor superfamily member 11 gene promoter polymorphisms modulate promoter activity and influence bone mineral density in postmenopausal women with osteoporosis. , 2008, Journal of molecular endocrinology.

[3]  Kari Stefansson,et al.  Multiple genetic loci for bone mineral density and fractures. , 2008, The New England journal of medicine.

[4]  L. Xing,et al.  Functions of RANKL/RANK/OPG in bone modeling and remodeling. , 2008, Archives of biochemistry and biophysics.

[5]  S. Ku,et al.  Association between osteoprotegerin (OPG), receptor activator of nuclear factor-&kgr;B (RANK), and RANK ligand (RANKL) gene polymorphisms and circulating OPG, soluble RANKL levels, and bone mineral density in Korean postmenopausal women , 2007, Menopause.

[6]  Qingbo Xu,et al.  Soluble Receptor Activator of Nuclear Factor-&kgr;B Ligand and Risk for Cardiovascular Disease , 2007, Circulation.

[7]  D. Gosselin,et al.  Translational regulation of PGHS-1 mRNA: 5' untranslated region and first two exons conferring negative regulation. , 2007, Biochimica et biophysica acta.

[8]  Hui Shen,et al.  Robust and Comprehensive Analysis of 20 Osteoporosis Candidate Genes by Very High‐Density Single‐Nucleotide Polymorphism Screen Among 405 White Nuclear Families Identified Significant Association and Gene–Gene Interaction , 2006, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.

[9]  Sun Woo Kim,et al.  The relationship between four single nucleotide polymorphisms in the promoter region of the osteoprotegerin gene and aortic calcification or coronary artery disease in Koreans , 2006, Clinical endocrinology.

[10]  G. Hansson,et al.  Enhanced T-Cell Expression of RANK Ligand in Acute Coronary Syndrome: Possible Role in Plaque Destabilization , 2006, Arteriosclerosis, thrombosis, and vascular biology.

[11]  K. Gunderson,et al.  A genome-wide scalable SNP genotyping assay using microarray technology , 2005, Nature Genetics.

[12]  L. Hofbauer,et al.  Osteoprotegerin gene polymorphisms in men with coronary artery disease. , 2004, The Journal of clinical endocrinology and metabolism.

[13]  G. Gambaro,et al.  Identification of a novel splice site mutation of CLCN5 gene and characterization of a new alternative 5′ UTR end of ClC-5 mRNA in human renal tissue and leukocytes , 2004, Journal of Human Genetics.

[14]  S. Khosla,et al.  Minireview: the OPG/RANKL/RANK system. , 2001, Endocrinology.

[15]  P. Geusens,et al.  Differential Expression of Bone Matrix Regulatory Proteins in Human Atherosclerotic Plaques , 2001, Arteriosclerosis, thrombosis, and vascular biology.

[16]  E. Braunwald,et al.  A classification of unstable angina revisited. , 2000, Circulation.

[17]  P. Libby Molecular bases of the acute coronary syndromes. , 1995, Circulation.

[18]  R. Punnett,et al.  The journal of genetics , 1910, Zeitschrift für Induktive Abstammungs- und Vererbungslehre.

[19]  T. Niu,et al.  Variation in genes involved in the RANKL/RANK/OPG bone remodeling pathway are associated with bone mineral density at different skeletal sites in men , 2005, Human Genetics.

[20]  F Milazzotto,et al.  [Classification of unstable angina]. , 1991, Giornale italiano di cardiologia.