Typical angina is associated with greater coronary endothelial dysfunction but not abnormal vasodilatory reserve
暂无分享,去创建一个
Galen Cook-Wiens | Derek Leong | Carl J Pepine | Daniel S Berman | C Noel Bairey Merz | Leslee J Shaw | D. Berman | L. Shaw | C. Pepine | E. Handberg | L. Thomson | G. Cook-Wiens | M. Nelson | Janet Wei | Puja K Mehta | Eileen M Handberg | P. Mehta | Michael D Nelson | Louise E Thomson | C. Shufelt | Ahmed AlBadri | Chrisandra L Shufelt | D. Leong | A. AlBadri | Janet Wei | C. B. Bairey Merz | Leslee J. Shaw | M. D. Nelson | C. J. Pepine | Louise E. J. Thomson | D. Berman
[1] M. Zubaid,et al. Atypical presentation of acute coronary syndrome: a significant independent predictor of in-hospital mortality. , 2011, Journal of cardiology.
[2] Lloyd D. Fisher,et al. Exercise stress testing. Correlations among history of angina, ST-segment response and prevalence of coronary-artery disease in the Coronary Artery Surgery Study (CASS). , 1979, The New England journal of medicine.
[3] G. Ginsburg,et al. The evaluation of chest pain in women. , 1996, The New England journal of medicine.
[4] K. Smith,et al. Abnormal Coronary Vasomotion as a Prognostic Indicator of Cardiovascular Events in Women: Results From the National Heart, Lung, and Blood Institute–Sponsored Women’s Ischemia Syndrome Evaluation (WISE) , 2004, Circulation.
[5] L. Shaw,et al. Prognosis in Women With Myocardial Ischemia in the Absence of Obstructive Coronary Disease: Results From the National Institutes of Health–National Heart, Lung, and Blood Institute–Sponsored Women’s Ischemia Syndrome Evaluation (WISE) , 2004, Circulation.
[6] Erik Jørgensen,et al. Stable angina pectoris with no obstructive coronary artery disease is associated with increased risks of major adverse cardiovascular events. , 2012, European heart journal.
[7] R A Deyo,et al. Development and evaluation of the Seattle Angina Questionnaire: a new functional status measure for coronary artery disease. , 1995, Journal of the American College of Cardiology.
[8] A. Lammertsma,et al. Myocardial blood flow is altered at rest and after dipyridamole in patients with syndrome X , 1991 .
[9] Gerold Porenta,et al. Typical chest pain and normal coronary angiogram: cardiac risk factor analysis versus PET for detection of microvascular disease. , 2007, Journal of nuclear medicine : official publication, Society of Nuclear Medicine.
[10] L D Fisher,et al. Exercise stress testing. Correlations among history of angina, ST-segment response and prevalence of coronary-artery disease in the Coronary Artery Surgery Study (CASS). , 1980, The New England journal of medicine.
[11] C. Vrints,et al. Impaired endothelium-dependent cholinergic coronary vasodilation in patients with angina and normal coronary arteriograms. , 1992, Journal of the American College of Cardiology.
[12] Debiao Li,et al. Cardiac magnetic resonance myocardial perfusion reserve index is reduced in women with coronary microvascular dysfunction. A National Heart, Lung, and Blood Institute-sponsored study from the Women's Ischemia Syndrome Evaluation. , 2015, Circulation. Cardiovascular imaging.
[13] W. Rogers,et al. Angiographic Prevalence of High‐risk Coronary Artery Disease in Patient Subsets (CASS) , 1981, Circulation.
[14] R. Cannon,et al. Site of increased resistance to coronary flow in patients with angina pectoris and normal epicardial coronary arteries. , 1986, Journal of the American College of Cardiology.
[15] M. Gotsman,et al. Histologic evidence for small-vessel coronary artery disease in patients with angina pectoris and patent large coronary arteries. , 1986, Circulation.
[16] J. Kastrup,et al. Coronary Microvascular Function and Cardiovascular Risk Factors in Women With Angina Pectoris and No Obstructive Coronary Artery Disease: The iPOWER Study , 2016, Journal of the American Heart Association.
[17] M A Hlatky,et al. A brief self-administered questionnaire to determine functional capacity (the Duke Activity Status Index). , 1989, The American journal of cardiology.
[18] J. Kaski,et al. Mechanisms of Angina Pectoris in Syndrome , 2016 .
[19] F. Crea,et al. Mechanisms of angina pectoris in syndrome X. , 1991, Journal of the American College of Cardiology.
[20] Kamlesh Kothawade,et al. Microvascular coronary dysfunction in women: pathophysiology, diagnosis, and management. , 2011, Current problems in cardiology.
[21] P. Libby,et al. Requiem for the 'vulnerable plaque'. , 2015, European heart journal.
[22] F. Crea,et al. Acute Coronary Syndromes Without Obstructive Coronary Atherosclerosis: The Tiles of a Complex Puzzle , 2014, Circulation. Cardiovascular interventions.
[23] Sanjay Kaul,et al. Low diagnostic yield of elective coronary angiography. , 2010, The New England journal of medicine.
[24] J. Kaski. Pathophysiology and Management of Patients With Chest Pain and Normal Coronary Arteriograms (Cardiac Syndrome X) , 2004, Circulation.
[25] O. Rimoldi,et al. Effects of Sex on Coronary Microvascular Dysfunction and Cardiac Outcomes , 2014, Circulation.
[26] Luke K. Hermann,et al. Comparison of frequency of inducible myocardial ischemia in patients presenting to emergency department with typical versus atypical or nonanginal chest pain. , 2010, The American journal of cardiology.
[27] M. C. S. Solano Ruiz,et al. Ischemic heart disease in women. , 2011, Revista latino-americana de enfermagem.
[28] L. Shaw,et al. Safety of coronary reactivity testing in women with no obstructive coronary artery disease: results from the NHLBI-sponsored WISE (Women's Ischemia Syndrome Evaluation) study. , 2012, JACC. Cardiovascular interventions.
[29] J P Ornato,et al. Prevalence, clinical characteristics, and mortality among patients with myocardial infarction presenting without chest pain. , 2000, JAMA.
[30] T. Lüscher,et al. Indirect evidence for release of endothelium-derived relaxing factor in human forearm circulation in vivo. Blunted response in essential hypertension. , 1990, Circulation.
[31] B. Gersh. Low Diagnostic Yield of Elective Coronary Angiography , 2011 .
[32] Galen Cook-Wiens,et al. A randomized, placebo-controlled trial of late Na current inhibition (ranolazine) in coronary microvascular dysfunction (CMD): impact on angina and myocardial perfusion reserve , 2015, European heart journal.
[33] T. Henry,et al. Increasing percutaneous coronary interventions for ST-segment elevation myocardial infarction in the United States: progress and opportunity. , 2015, JACC. Cardiovascular interventions.
[34] Josh Klein,et al. A selection of recent original research papers , 2014, Journal of Nuclear Cardiology.
[35] H. Drexler,et al. Modulation of coronary vasomotor tone in humans. Progressive endothelial dysfunction with different early stages of coronary atherosclerosis. , 1991, Circulation.
[36] David Brieger,et al. Acute coronary syndromes without chest pain, an underdiagnosed and undertreated high-risk group: insights from the Global Registry of Acute Coronary Events. , 2004, Chest.
[37] A. Hall,et al. Poor prognosis of patients presenting with symptomatic myocardial infarction but without chest pain , 2001, Heart.
[38] M. Sugimachi,et al. Impaired coronary blood flow response to acetylcholine in patients with coronary risk factors and proximal atherosclerotic lesions. , 1993, The Journal of clinical investigation.
[39] R. de Caterina,et al. Angina pectoris and myocardial ischemia in the absence of obstructive coronary artery disease: practical considerations for diagnostic tests. , 2014, JACC. Cardiovascular interventions.
[40] G. Diamond. A clinically relevant classification of chest discomfort. , 1983, Journal of the American College of Cardiology.
[41] G. Diamond,et al. Analysis of probability as an aid in the clinical diagnosis of coronary-artery disease. , 1979, The New England journal of medicine.
[42] J. Kaski. Pathophysiology and Management of Patients With Chest Pain and Normal Coronary Arteriograms ( Cardiac Syndrome , 2004 .