Poloxamer-188 Attenuates TBI-Induced Blood–Brain Barrier Damage Leading to Decreased Brain Edema and Reduced Cellular Death
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Yu Gao | Z. Qin | Ran Liu | Xi-ping Chen | L. Tao | Mingyang Zhang | Haijun Bao | Long Wang | D. Dai | Tao Wang | Lu Zhang | Yaohui Wang
[1] D. Hovda,et al. The neurophysiology of concussion. , 2014, Progress in neurological surgery.
[2] E. Lo,et al. Kollidon VA64, a Membrane-Resealing Agent, Reduces Histopathology and Improves Functional Outcome after Controlled Cortical Impact in Mice , 2012, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[3] Michelle C LaPlaca,et al. Trauma-induced plasmalemma disruptions in three-dimensional neural cultures are dependent on strain modality and rate. , 2011, Journal of neurotrauma.
[4] Z. Qin,et al. Autophagy is involved in traumatic brain injury-induced cell death and contributes to functional outcome deficits in mice , 2011, Neuroscience.
[5] Y. Kang,et al. Fluid-percussion brain injury induces changes in aquaporin channel expression , 2011, Neuroscience.
[6] Kevin M. Curtis,et al. Aquaporin-4 expression in cultured astrocytes after fluid percussion injury. , 2011, Journal of neurotrauma.
[7] I. Shelef,et al. Blood-Brain Barrier Breakdown Following Traumatic Brain Injury: A Possible Role in Posttraumatic Epilepsy , 2011, Cardiovascular psychiatry and neurology.
[8] Alon Friedman,et al. Blood–brain barrier breakdown as a therapeutic target in traumatic brain injury , 2010, Nature Reviews Neurology.
[9] L. Puybasset,et al. Effect of acute poly(ADP-ribose) polymerase inhibition by 3-AB on blood-brain barrier permeability and edema formation after focal traumatic brain injury in rats. , 2010, Journal of neurotrauma.
[10] R. Vink,et al. Mechanisms of cerebral edema in traumatic brain injury: therapeutic developments , 2010, Current opinion in neurology.
[11] R. Hunter,et al. Poloxamer 188 inhibition of ischemia/reperfusion injury: evidence for a novel anti-adhesive mechanism. , 2010, Annals of clinical and laboratory science.
[12] Patrick O. Eghwrudjakpor,et al. Decompressive craniectomy following brain injury: factors important to patient outcome. , 2010, The Libyan journal of medicine.
[13] Patrick O. Eghwrudjakpor,et al. Decompressive craniectomy following brain injury: factors important to patient outcome , 2010, The Libyan journal of medicine.
[14] Tao,et al. Comparison of labeling methods and time course of traumatic brain injury-induced cell death in mice , 2010 .
[15] Crystal M. Simon,et al. Plasma membrane damage as a marker of neuronal injury , 2009, 2009 Annual International Conference of the IEEE Engineering in Medicine and Biology Society.
[16] A. Verkman,et al. Glial Cell Aquaporin-4 Overexpression in Transgenic Mice Accelerates Cytotoxic Brain Swelling* , 2008, Journal of Biological Chemistry.
[17] Shankar Gopinath,et al. Management of intracranial hypertension. , 2008, Neurologic clinics.
[18] M. Moskowitz,et al. Acute Plasmalemma Permeability and Protracted Clearance of Injured Cells after Controlled Cortical Impact in Mice , 2008, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[19] BenjaminBierbach,et al. Emboli Formation Rather Than Inflammatory Mediators Are Responsible for Increased Cerebral Water Content After Conventional and Assisted Beating-Heart Myocardial Revascularization in a Porcine Model , 2008 .
[20] A. Doerfler,et al. Differentiation of cytotoxic and vasogenic edema in a patient with reversible posterior leukoencephalopathy syndrome using diffusion-weighted MRI. , 2007, Diagnostic and interventional radiology.
[21] N. Plesnila,et al. Delayed neuronal death after brain trauma involves p53-dependent inhibition of NF-κB transcriptional activity , 2007, Cell Death and Differentiation.
[22] Anthony Marmarou,et al. A review of progress in understanding the pathophysiology and treatment of brain edema. , 2007, Neurosurgical focus.
[23] M. Papadopoulos,et al. Aquaporins in the Central Nervous System , 2007 .
[24] R. Haut,et al. Treatment with the Non-ionic Surfactant Poloxamer P188 Reduces DNA Fragmentation in Cells from Bovine Chondral Explants Exposed to Injurious Unconfined Compression , 2006, Biomechanics and modeling in mechanobiology.
[25] K. Barbee,et al. Mechanisms of cell death and neuroprotection by poloxamer 188 after mechanical trauma , 2006, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[26] R. Berman,et al. Diffusion-weighted imaging of edema following traumatic brain injury in rats: effects of secondary hypoxia. , 2005, Journal of neurotrauma.
[27] K. Barbee,et al. The effect of poloxamer-188 on neuronal cell recovery from mechanical injury. , 2005, Journal of neurotrauma.
[28] P. Kochanek,et al. Bench-to-bedside review: Apoptosis/programmed cell death triggered by traumatic brain injury , 2004, Critical care.
[29] 刘伟国,et al. Correlation of cell apoptosis with brain edema and elevated intracranial pressure in traumatic brain injury , 2005 .
[30] B. Nico,et al. The role of aquaporin-4 in the blood–brain barrier development and integrity: Studies in animal and cell culture models , 2004, Neuroscience.
[31] Raphael C. Lee,et al. Poloxamer 188 prevents acute necrosis of adult skeletal muscle cells following high-dose irradiation. , 2004, Burns : journal of the International Society for Burn Injuries.
[32] Turgay Dalkara,et al. Apoptotic and Necrotic Death Mechanisms Are Concomitantly Activated in the Same Cell After Cerebral Ischemia , 2004, Stroke.
[33] Raphael C. Lee,et al. Surfactant poloxamer 188-related decreases in inflammation and tissue damage after experimental brain injury in rats. , 2004, Journal of neurosurgery.
[34] R. Raghupathi,et al. Cell Death Mechanisms Following Traumatic Brain Injury , 2004, Brain pathology.
[35] YujiMatsumoto,et al. Na+/H+ Exchanger Inhibitor, SM-20220, Is Protective Against Excitotoxicity in Cultured Cortical Neurons , 2004 .
[36] N. Plesnila,et al. Effect of decompression craniotomy on increase of contusion volume and functional outcome after controlled cortical impact in mice. , 2003, Journal of neurotrauma.
[37] M. Papadopoulos,et al. Water transport becomes uncoupled from K+ siphoning in brain contusion, bacterial meningitis, and brain tumours: immunohistochemical case review , 2003, Journal of clinical pathology.
[38] M. Robbins,et al. Nuclear factor-kappaB translocation mediates double-stranded ribonucleic acid-induced NIT-1 beta-cell apoptosis and up-regulates caspase-12 and tumor necrosis factor receptor-associated ligand (TRAIL). , 2003, Endocrinology.
[39] J. P. Schwartz,et al. Closed-head minimal traumatic brain injury produces long-term cognitive deficits in mice , 2003, Neuroscience.
[40] C. Honey,et al. Regulation of aquaporin-4 in a traumatic brain injury model in rats. , 2003, Journal of neurosurgery.
[41] A. Marmarou,et al. Pathophysiology of traumatic brain edema: current concepts. , 2003, Acta neurochirurgica. Supplement.
[42] N. Abbott,et al. Astrocyte–endothelial interactions and blood–brain barrier permeability * , 2002 .
[43] N. Abbott. Astrocyte–endothelial interactions and blood–brain barrier permeability , 2002, Journal of anatomy.
[44] W. Poon,et al. Heterogeneous responses of aquaporin-4 in oedema formation in a replicated severe traumatic brain injury model in rats , 2001, Neuroscience Letters.
[45] J. DeSimone. Focus on "rapid entry of bitter and sweet tastants into liposomes and taste cells: implications for signal transduction". , 2000, American journal of physiology. Cell physiology.
[46] A S Verkman,et al. Structure and function of aquaporin water channels. , 2000, American journal of physiology. Renal physiology.
[47] A. Lentsch,et al. Activation and Regulation of NFκB during Acute Inflammation , 1999 .
[48] A. Lentsch,et al. Activation and regulation of NFkappaB during acute inflammation. , 1999, Clinical chemistry and laboratory medicine.
[49] P. Agre,et al. Specialized Membrane Domains for Water Transport in Glial Cells: High-Resolution Immunogold Cytochemistry of Aquaporin-4 in Rat Brain , 1997, The Journal of Neuroscience.
[50] J. Povlishock,et al. Traumatically induced altered membrane permeability: its relationship to traumatically induced reactive axonal change. , 1994, Journal of neurotrauma.
[51] R. Mechoulam,et al. A nonpsychotropic cannabinoid, HU-211, has cerebroprotective effects after closed head injury in the rat. , 1993, Journal of neurotrauma.
[52] Richard T. Linn,et al. Responses to cortical injury: I. Methodology and local effects of contusions in the rat , 1981, Brain Research.