ACE-inhibition induces NO-formation in cultured bovine endothelial cells and protects isolated ischemic rat hearts.
暂无分享,去创建一个
[1] R. Busse,et al. Ramiprilat Enhances Endothelial Autacoid Formation by Inhibiting Breakdown of Endothelium‐Derived Bradykinin , 1991, Hypertension.
[2] P. Munch,et al. Bradykinin increases myocardial contractility: relation to the Gregg phenomenon. , 1991, The American journal of physiology.
[3] R. Tallarida,et al. Pharmacological evidence that captopril possesses an endothelium-mediated component of vasodilation: effect of sulfhydryl groups on endothelium-derived relaxing factor. , 1991, The Journal of pharmacology and experimental therapeutics.
[4] W. König,et al. Hoe 140 a new potent and long acting bradykinin‐antagonist: in vivo studies , 1991, British journal of pharmacology.
[5] Jon M. Fukuto,et al. Shear Stress‐Induced Release of Nitric Oxide From Endothelial Cells Grown on Beads , 1991, Hypertension.
[6] I. Morita,et al. Synthesis and release of acetylcholine by cultured bovine arterial endothelial cells , 1990, Neuroscience Letters.
[7] V. Miller. Does antihypertensive therapy improve the function of the vascular endothelium? , 1990, Hypertension.
[8] G. Burnstock,et al. Endothelial cells cultured from human umbilical vein release ATP, substance P and acetylcholine in response to increased flow , 1990, Proceedings of the Royal Society of London. Series B: Biological Sciences.
[9] W. Linz,et al. Reduction of infarct size by local angiotensin-converting enzyme inhibition is abolished by a bradykinin antagonist. , 1990, European journal of pharmacology.
[10] B. Lipworth,et al. Assessment of airways, tremor and chronotropic responses to inhaled salbutamol in the quantification of beta 2-adrenoceptor blockade. , 1989, British journal of clinical pharmacology.
[11] O. Carretero,et al. Kinin Antagonist Reverses Converting Enzyme Inhibitor‐Stimulated Vascular Prostaglandin I2 Synthesis , 1989, Hypertension.
[12] J. McMurray,et al. Captopril: a free radical scavenger. , 1989, British journal of clinical pharmacology.
[13] J. Vane,et al. Effect of Captopril on the bradykinin‐induced release of prostacyclin from guinea‐pig lungs and bovine aortic endothelial cells , 1988, British journal of pharmacology.
[14] R. Busse,et al. Differential role of extra‐ and intracellular calcium in the release of EDRF and prostacyclin from cultured endothelial cells , 1988, British journal of pharmacology.
[15] P. D. de Groot,et al. High-molecular weight kininogen is present in cultured human endothelial cells: localization, isolation, and characterization. , 1988, Blood.
[16] M. Bernier,et al. Reperfusion-induced arrhythmias: mechanisms of protection by glucose and mannitol. , 1988, The American journal of physiology.
[17] Sadao Kimura,et al. A novel potent vasoconstrictor peptide produced by vascular endothelial cells , 1988, Nature.
[18] V. Dzau,et al. Endothelial renin-angiotensin pathway: evidence for intracellular synthesis and secretion of angiotensins. , 1987, Circulation research.
[19] T. Metsä-Ketelä,et al. The effects of cyclic AMP and cyclic GMP on redox state and energy state in hypoxic rat atria. , 1984, Life sciences.
[20] J. Eckel,et al. Influence of bradykinin on glucose uptake and metabolism studied in isolated cardiac myocytes and isolated perfused rat hearts. , 1983, Hoppe-Seyler's Zeitschrift fur physiologische Chemie.
[21] L. Opie,et al. Effects of Substrates on Tissue Metabolic Changes in the Isolated Rat Heart during Underperfusion and on Release of Lactate Dehydrogenase and Arrhythmias during Reperfusion , 1978, Circulation research.
[22] S. Furukawa,et al. Changes in components of kinin system and hemodynamics in acute myocardial infarction. , 1978, American heart journal.
[23] S. Furukawa,et al. Changes in hemodynamics and bradykinin concentration in coronary sinus blood in experimental coronary artery occlusion. , 1977, Japanese heart journal.
[24] W. Linz,et al. Local Inhibition of Bradykinin Degradation in Ischemic Hearts , 1990, Journal of cardiovascular pharmacology.
[25] K. Shimamoto,et al. Role of Kallikrein‐Kinin System in the Hypotensive Mechanisms of Converting Enzyme Inhibitors in Essential Hypertension , 1989, Journal of cardiovascular pharmacology.
[26] P. Vanhoutte,et al. Why are converting enzyme inhibitors vasodilators? , 1989, British journal of clinical pharmacology.
[27] J. Parratt,et al. Eicosanoids and susceptibility to ventricular arrhythmias during myocardial ischaemia and reperfusion. , 1987, Journal of molecular and cellular cardiology.
[28] W. Linz,et al. Beneficial effects of the converting enzyme inhibitor, ramipril, in ischemic rat hearts. , 1986, Journal of cardiovascular pharmacology.