IL-16 rs11556218 gene polymorphism is associated with coronary artery disease in the Chinese Han population.

[1]  Tinghe Yu,et al.  Association of an interleukin-16 gene polymorphism with the risk and pain phenotype of endometriosis. , 2010, DNA and cell biology.

[2]  Weibo Liang,et al.  Genetic polymorphism of interleukin-16 and risk of nasopharyngeal carcinoma. , 2009, Clinica chimica acta; international journal of clinical chemistry.

[3]  Weibo Liang,et al.  The IL-16 gene polymorphisms and the risk of the systemic lupus erythematosus. , 2009, Clinica chimica acta; international journal of clinical chemistry.

[4]  Daniel F. Schwarz,et al.  New susceptibility locus for coronary artery disease on chromosome 3q22.3 , 2009, Nature Genetics.

[5]  A. Sharpe,et al.  T-Cell Costimulation and Coinhibition in Atherosclerosis , 2008, Circulation research.

[6]  Weibo Liang,et al.  The association of interleukin-16 polymorphisms with IL-16 serum levels and risk of colorectal and gastric cancer. , 2008, Carcinogenesis.

[7]  J. Ioannidis.  Why Most Discovered True Associations Are Inflated , 2008, Epidemiology.

[8]  G. Ning,et al.  Association of the interleukin (IL)-16 gene polymorphisms with Graves' disease. , 2008, Clinical immunology.

[9]  W. Willett,et al.  Multiple loci identified in a genome-wide association study of prostate cancer , 2008, Nature Genetics.

[10]  P. Donnelly,et al.  Replicating genotype–phenotype associations , 2007, Nature.

[11]  G. Abecasis,et al.  Human Genome Variation 2006: emerging views on structural variation and large-scale SNP analysis , 2007, Nature Genetics.

[12]  D. Duffy,et al.  A polymorphism in the promoter region of the human interleukin‐16 gene is not associated with asthma or atopy in an Australian population , 2005, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.

[13]  E. Lander,et al.  Meta-analysis of genetic association studies supports a contribution of common variants to susceptibility to common disease , 2003, Nature Genetics.

[14]  K. Sullivan,et al.  Interferon-γ polymorphisms in systemic lupus erythematosus , 2001, Genes and Immunity.

[15]  J. Bluestone,et al.  Effect of Immune Deficiency on Lipoproteins and Atherosclerosis in Male Apolipoprotein E-Deficient Mice , 2001, Arteriosclerosis, thrombosis, and vascular biology.

[16]  Xinghua Zhou,et al.  Transfer of CD4+ T Cells Aggravates Atherosclerosis in Immunodeficient Apolipoprotein E Knockout Mice , 2000, Circulation.

[17]  R. Kurth,et al.  Interleukin‐16 stimulates the expression and production of pro‐inflammatory cytokines by human monocytes , 2000, Immunology.

[18]  J. Sparks,et al.  Human B‐cell lines constitutively express and secrete interleukin‐16 , 2000, Immunology.

[19]  R. Ross,et al.  Atherosclerosis is an Inflammatory Disease , 1998 .

[20]  D. Metcalfe,et al.  Human mast cells produce the CD4+ T lymphocyte chemoattractant factor, IL-16. , 1997, Journal of immunology.

[21]  W. Cruikshank,et al.  Interleukin 16 and its function as a CD4 ligand. , 1996, Immunology today.

[22]  W. Cruikshank,et al.  Histamine-induced secretion of lymphocyte chemoattractant factor from CD8+ T cells is independent of transcription and translation. Evidence for constitutive protein synthesis and storage. , 1995, Journal of immunology.

[23]  Henry A. Erlich,et al.  PCR Technology: Principles and Applications for DNA Amplification , 1989 .

[24]  H. Colten,et al.  Molecular mechanism for feedback regulation of C4 biosynthesis in guinea pig peritoneal macrophage , 1984, The Journal of experimental medicine.

[25]  W. Cruikshank,et al.  Modulation of lymphocyte migration by human lymphokines. I. Identification and characterization of chemoattractant activity for lymphocytes from mitogen-stimulated mononuclear cells. , 1982, Journal of immunology.

[26]  W. Kimberling,et al.  Genetic–Epidemiologic Study of Early–onset Ischemic Heart Disease , 1980, Circulation.

[27]  A. Iwamoto,et al.  A new polymorphism in the promoter region of the human interleukin-16 (IL-16) gene , 2000, Genes and Immunity.

[28]  Alan D. Lopez,et al.  The global burden of disease, 1990–2020 , 1998, Nature Medicine.