FOG‐1 recruits the NuRD repressor complex to mediate transcriptional repression by GATA‐1
暂无分享,去创建一个
G. Blobel | Ying-Yu Chen | W. Hong | C. Vakoc | M. Nakazawa | Carrie Rakowski | R. Kori
[1] E. Svensson,et al. The N Termini of Friend of GATA (FOG) Proteins Define a Novel Transcriptional Repression Motif and a Superfamily of Transcriptional Repressors* , 2004, Journal of Biological Chemistry.
[2] Hao Wang,et al. Global regulation of erythroid gene expression by transcription factor GATA-1. , 2004, Blood.
[3] David L. Jaye,et al. MTA3 and the Mi-2/NuRD Complex Regulate Cell Fate during B Lymphocyte Differentiation , 2004, Cell.
[4] J. Seavitt,et al. The chromatin remodeler Mi-2beta is required for CD4 expression and T cell development. , 2004, Immunity.
[5] Naoyuki Fujita,et al. Mi-2/NuRD: multiple complexes for many purposes. , 2004, Biochimica et biophysica acta.
[6] Kirby D. Johnson,et al. Coregulator-dependent facilitation of chromatin occupancy by GATA-1. , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[7] G. Blobel,et al. Context-dependent regulation of GATA-1 by friend of GATA-1. , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[8] R. Aebersold,et al. Dynamic changes in transcription factor complexes during erythroid differentiation revealed by quantitative proteomics , 2004, Nature Structural &Molecular Biology.
[9] T. Naoe,et al. Altered interaction of HDAC5 with GATA-1 during MEL cell differentiation , 2003, Oncogene.
[10] Ya-Li Yao,et al. The Metastasis-associated Proteins 1 and 2 Form Distinct Protein Complexes with Histone Deacetylase Activity* , 2003, Journal of Biological Chemistry.
[11] L. Chodosh,et al. GATA-1-Mediated Proliferation Arrest during Erythroid Maturation , 2003, Molecular and Cellular Biology.
[12] Jing Wu,et al. GATA-1-dependent transcriptional repression of GATA-2 via disruption of positive autoregulation and domain-wide chromatin remodeling , 2003, Proceedings of the National Academy of Sciences of the United States of America.
[13] G. Blobel,et al. Formation of a Tissue-Specific Histone Acetylation Pattern by the Hematopoietic Transcription Factor GATA-1 , 2003, Molecular and Cellular Biology.
[14] Jonathan E. Schmitz,et al. Novel Combinatorial Interactions of GATA-1, PU.1, and C/EBPε Isoforms Regulate Transcription of the Gene Encoding Eosinophil Granule Major Basic Protein* , 2002, The Journal of Biological Chemistry.
[15] G. Blobel,et al. Control of megakaryocyte‐specific gene expression by GATA‐1 and FOG‐1: role of Ets transcription factors , 2002, The EMBO journal.
[16] D. Gell,et al. Characterization of the Conserved Interaction between GATA and FOG Family Proteins* 210 , 2002, The Journal of Biological Chemistry.
[17] M. L. Beau,et al. Acquired mutations in GATA1 in the megakaryoblastic leukemia of Down syndrome , 2002, Nature Genetics.
[18] Aaron N. Chang,et al. GATA-factor dependence of the multitype zinc-finger protein FOG-1 for its essential role in megakaryopoiesis , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[19] S. Orkin,et al. Distinct Domains of the GATA-1 Cofactor FOG-1 Differentially Influence Erythroid versus Megakaryocytic Maturation , 2002, Molecular and Cellular Biology.
[20] S. Orkin,et al. Interaction between FOG-1 and the Corepressor C-Terminal Binding Protein Is Dispensable for Normal Erythropoiesis In Vivo , 2002, Molecular and Cellular Biology.
[21] M. Rauchman,et al. Murine Sall1 Represses Transcription by Recruiting a Histone Deacetylase Complex* , 2002, The Journal of Biological Chemistry.
[22] Tony Kouzarides,et al. Histone H3 Lysine 4 Methylation Disrupts Binding of Nucleosome Remodeling and Deacetylase (NuRD) Repressor Complex* , 2002, The Journal of Biological Chemistry.
[23] D. Reinberg,et al. Set9, a novel histone H3 methyltransferase that facilitates transcription by precluding histone tail modifications required for heterochromatin formation. , 2002, Genes & development.
[24] S. Bohlander,et al. SALL1, the gene mutated in Townes-Brocks syndrome, encodes a transcriptional repressor which interacts with TRF1/PIN2 and localizes to pericentromeric heterochromatin. , 2001, Human molecular genetics.
[25] G. Seydoux,et al. PIE-1 is a bifunctional protein that regulates maternal and zygotic gene expression in the embryonic germ line of Caenorhabditis elegans. , 2001, Genes & development.
[26] J. Friedman,et al. Targeting histone deacetylase complexes via KRAB-zinc finger proteins: the PHD and bromodomains of KAP-1 form a cooperative unit that recruits a novel isoform of the Mi-2alpha subunit of NuRD. , 2001, Genes & development.
[27] Q. Feng,et al. The MeCP1 complex represses transcription through preferential binding, remodeling, and deacetylating methylated nucleosomes. , 2001, Genes & development.
[28] Delin Chen,et al. Deacetylation of p53 modulates its effect on cell growth and apoptosis , 2000, Nature.
[29] H. Erdjument-Bromage,et al. An Ikaros-Containing Chromatin-Remodeling Complex in Adult-Type Erythroid Cells , 2000, Molecular and Cellular Biology.
[30] E. Svensson,et al. A Functionally Conserved N-terminal Domain of the Friend of GATA-2 (FOG-2) Protein Represses GATA4-Dependent Transcription* , 2000, The Journal of Biological Chemistry.
[31] L. Zon,et al. FOG acts as a repressor of red blood cell development in Xenopus. , 2000, Development.
[32] M. Leid,et al. Isolation of a Novel Family of C2H2 Zinc Finger Proteins Implicated in Transcriptional Repression Mediated by Chicken Ovalbumin Upstream Promoter Transcription Factor (COUP-TF) Orphan Nuclear Receptors* , 2000, The Journal of Biological Chemistry.
[33] S. Orkin,et al. Familial dyserythropoietic anaemia and thrombocytopenia due to an inherited mutation in GATA1 , 2000, Nature Genetics.
[34] M. Robinson,et al. Telomere shortening and apoptosis in telomerase-inhibited human tumor cells. , 1999, Genes & development.
[35] Paul Tempst,et al. MBD2 is a transcriptional repressor belonging to the MeCP1 histone deacetylase complex , 1999, Nature Genetics.
[36] A. Bird,et al. Analysis of the NuRD subunits reveals a histone deacetylase core complex and a connection with DNA methylation. , 1999, Genes & development.
[37] J. Mackay,et al. Transcriptional cofactors of the FOG family interact with GATA proteins by means of multiple zinc fingers , 1999, The EMBO journal.
[38] R. Kingston,et al. Ikaros DNA-binding proteins direct formation of chromatin remodeling complexes in lymphocytes. , 1999, Immunity.
[39] S. Orkin,et al. Use of altered specificity mutants to probe a specific protein-protein interaction in differentiation: the GATA-1:FOG complex. , 1999, Molecular cell.
[40] Weidong Wang,et al. NURD, a novel complex with both ATP-dependent chromatin-remodeling and histone deacetylase activities. , 1998, Molecular cell.
[41] S. Schreiber,et al. Chromatin deacetylation by an ATP-dependent nucleosome remodelling complex , 1998, Nature.
[42] D. Reinberg,et al. The Dermatomyositis-Specific Autoantigen Mi2 Is a Component of a Complex Containing Histone Deacetylase and Nucleosome Remodeling Activities , 1998, Cell.
[43] A. Wolffe,et al. A multiple subunit Mi-2 histone deacetylase from Xenopus laevis cofractionates with an associated Snf2 superfamily ATPase , 1998, Current Biology.
[44] S. Orkin,et al. Failure of megakaryopoiesis and arrested erythropoiesis in mice lacking the GATA-1 transcriptional cofactor FOG. , 1998, Genes & development.
[45] S. Orkin,et al. FOG, a Multitype Zinc Finger Protein, Acts as a Cofactor for Transcription Factor GATA-1 in Erythroid and Megakaryocytic Differentiation , 1997, Cell.
[46] R. Y. Tsai,et al. Cloning and Functional Characterization of Roaz, a Zinc Finger Protein that Interacts with O/E-1 to Regulate Gene Expression: Implications for Olfactory Neuronal Development , 1997, The Journal of Neuroscience.
[47] S. Orkin,et al. Erythroid-cell-specific properties of transcription factor GATA-1 revealed by phenotypic rescue of a gene-targeted cell line , 1997, Molecular and cellular biology.
[48] Y Fujiwara,et al. Arrested development of embryonic red cell precursors in mouse embryos lacking transcription factor GATA-1. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[49] G. Nolan,et al. Production of high-titer helper-free retroviruses by transient transfection. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[50] S. Orkin,et al. Erythroid differentiation in chimaeric mice blocked by a targeted mutation in the gene for transcription factor GATA-1 , 1991, Nature.