The adaptor Act1 is required for interleukin 17–dependent signaling associated with autoimmune and inflammatory disease
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V. Tuohy | N. Giltiay | Jianhua Xiao | T. Kordula | B. Vallance | T. Hamilton | J. Hartupee | D. Jane-wit | S. Swaidani | Xiaoxia Li | C. Z. Altuntas | Youcun Qian | Caini Liu | Yi Lu | M. Aronica | M. F. Gulen | Jinbo Liu | Qiwei Zhang | Dan Jane-wit
[1] A. Sher,et al. IL-23 plays a key role in Helicobacter hepaticus–induced T cell–dependent colitis , 2006, The Journal of experimental medicine.
[2] H. Kubo,et al. IL-17A promotes the growth of airway epithelial cells through ERK-dependent signaling pathway. , 2006, Biochemical and Biophysical Research Communications - BBRC.
[3] Zihua Hu,et al. Identification of Common Transcriptional Regulatory Elements in Interleukin-17 Target Genes* , 2006, Journal of Biological Chemistry.
[4] Hans Lassmann,et al. Understanding pathogenesis and therapy of multiple sclerosis via animal models: 70 years of merits and culprits in experimental autoimmune encephalomyelitis research. , 2006, Brain : a journal of neurology.
[5] J. Derry,et al. Cutting Edge: Interleukin 17 Signals through a Heteromeric Receptor Complex , 2006, The Journal of Immunology.
[6] B. Becher,et al. Antigen presentation in autoimmunity and CNS inflammation: how T lymphocytes recognize the brain , 2006, Journal of Molecular Medicine.
[7] H. Weiner,et al. Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells , 2006, Nature.
[8] L. Presta,et al. Anti-IL-23 therapy inhibits multiple inflammatory pathways and ameliorates autoimmune encephalomyelitis. , 2006, The Journal of clinical investigation.
[9] J. Kolls,et al. Critical role of IL‐17 receptor signaling in acute TNBS‐induced colitis , 2006, Inflammatory bowel diseases.
[10] R. D. Hatton,et al. Transforming growth factor-β induces development of the TH17 lineage , 2006, Nature.
[11] Y. Iwakura,et al. The IL-23/IL-17 axis in inflammation. , 2006, The Journal of clinical investigation.
[12] R. J. Hocking,et al. TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells. , 2006, Immunity.
[13] F. Ruemmele,et al. Interleukin-17 is a potent immuno-modulator and regulator of normal human intestinal epithelial cell growth. , 2005, Biochemical and biophysical research communications.
[14] Ki-Young Lee,et al. TAK1, but not TAB1 or TAB2, plays an essential role in multiple signaling pathways in vivo. , 2005, Genes & development.
[15] R. D. Hatton,et al. Interleukin 17–producing CD4+ effector T cells develop via a lineage distinct from the T helper type 1 and 2 lineages , 2005, Nature Immunology.
[16] Ying Wang,et al. A distinct lineage of CD4 T cells regulates tissue inflammation by producing interleukin 17 , 2005, Nature Immunology.
[17] L. Joosten,et al. Interleukin-17 receptor deficiency results in impaired synovial expression of interleukin-1 and matrix metalloproteinases 3, 9, and 13 and prevents cartilage destruction during chronic reactivated streptococcal cell wall-induced arthritis. , 2005, Arthritis and rheumatism.
[18] S. Shapiro,et al. An elevated matrix metalloproteinase (MMP) in an animal model of multiple sclerosis is protective by affecting Th1/Th2 polarization , 2005, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[19] S. Akira,et al. Essential function for the kinase TAK1 in innate and adaptive immune responses , 2005, Nature Immunology.
[20] A. Lindén,et al. Interleukin-17 family members and inflammation. , 2004, Immunity.
[21] E. C. Snow,et al. Act1, a negative regulator in CD40- and BAFF-mediated B cell survival. , 2004, Immunity.
[22] Sarah L. Gaffen,et al. Functional Cooperation between Interleukin-17 and Tumor Necrosis Factor-α Is Mediated by CCAAT/Enhancer-binding Protein Family Members* , 2004, Journal of Biological Chemistry.
[23] Zhendong Zhao,et al. IFN Regulatory Factor-1 Is Required for the Up-Regulation of the CD40-NF-κB Activator 1 Axis During Airway Inflammation1 , 2003, The Journal of Immunology.
[24] Y. Ho,et al. Stimulation of Airway Mucin Gene Expression by Interleukin (IL)-17 through IL-6 Paracrine/Autocrine Loop* , 2003, The Journal of Biological Chemistry.
[25] Frank Eisenhaber,et al. The STIR-domain superfamily in signal transduction, development and immunity. , 2003, Trends in biochemical sciences.
[26] R. Kastelein,et al. Interleukin-23 rather than interleukin-12 is the critical cytokine for autoimmune inflammation of the brain , 2003, Nature.
[27] P. Chambon,et al. Expression of conditional cre recombinase in epithelial tissues of transgenic mice , 2003, Genesis.
[28] I. Homma,et al. Antigen-specific T cell sensitization is impaired in IL-17-deficient mice, causing suppression of allergic cellular and humoral responses. , 2002, Immunity.
[29] W. Sha,et al. Constitutive Expression of the B7h Ligand for Inducible Costimulator on Naive B Cells Is Extinguished after Activation by Distinct B Cell Receptor and Interleukin 4 Receptor–mediated Pathways and Can Be Rescued by CD40 Signaling , 2002, The Journal of experimental medicine.
[30] Zhengfan Jiang,et al. Role of NFκB activator Act1 in CD40-mediated signaling in epithelial cells , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[31] E. Benveniste,et al. Immune function of astrocytes , 2001, Glia.
[32] V. Trajković,et al. Interleukin-17 stimulates inducible nitric oxide synthase activation in rodent astrocytes , 2001, Journal of Neuroimmunology.
[33] J. Shellito,et al. Requirement of Interleukin 17 Receptor Signaling for Lung Cxc Chemokine and Granulocyte Colony-Stimulating Factor Expression, Neutrophil Recruitment, and Host Defense , 2001, The Journal of experimental medicine.
[34] A. Leonardi,et al. CIKS, a connection to Ikappa B kinase and stress-activated protein kinase. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[35] X. Hua,et al. Act1, an NF-κB-activating protein , 2000 .
[36] A. Israël,et al. Complete lack of NF-kappaB activity in IKK1 and IKK2 double-deficient mice: additional defect in neurulation. , 2000, Genes & development.
[37] K. Yamaguchi,et al. Requirement of Tumor Necrosis Factor Receptor–Associated Factor (Traf)6 in Interleukin 17 Signal Transduction , 2000, The Journal of experimental medicine.
[38] R. Ransohoff,et al. TGFβ1 selectively up‐regulates CCR1 expression in primary murine astrocytes , 2000, Glia.
[39] H. Reinecker,et al. NF-kappa B-inducing kinase is a common mediator of IL-17-, TNF-alpha-, and IL-1 beta-induced chemokine promoter activation in intestinal epithelial cells. , 1999, Journal of immunology.
[40] S. Morony,et al. TRAF6 deficiency results in osteopetrosis and defective interleukin-1, CD40, and LPS signaling. , 1999, Genes & development.
[41] J. Fjell,et al. B-cell-deficient mice develop experimental allergic encephalomyelitis with demyelination after myelin oligodendrocyte glycoprotein sensitization. , 1998, Journal of immunology.
[42] P. Heinrich,et al. Oncostatin M and the Interleukin-6 and Soluble Interleukin-6 Receptor Complex Regulate α1-Antichymotrypsin Expression in Human Cortical Astrocytes* , 1998, The Journal of Biological Chemistry.
[43] J. Derry,et al. Molecular characterization of the human interleukin (IL)-17 receptor. , 1997, Cytokine.
[44] R. J. Hocking,et al. TGFb in the Context of an Inflammatory Cytokine Milieu Supports De Novo Differentiation of IL-17-Producing T Cells , 2022 .