Autophagy differentially regulates TNF receptor Fn14 by distinct mammalian Atg8 proteins
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[1] R. Klausen,et al. Selective , 2020, Encyclopedia of the UN Sustainable Development Goals.
[2] R. Schekman,et al. Remodeling of ER-exit sites initiates a membrane supply pathway for autophagosome biogenesis , 2017, bioRxiv.
[3] G. Ramm,et al. Atg8 family LC3/GABARAP proteins are crucial for autophagosome–lysosome fusion but not autophagosome formation during PINK1/Parkin mitophagy and starvation , 2016, The Journal of cell biology.
[4] Ah Ram Lee,et al. Hepatitis B virus–triggered autophagy targets TNFRSF10B/death receptor 5 for degradation to limit TNFSF10/TRAIL response , 2016, Autophagy.
[5] Yue Meng,et al. Gefitinib enhances human colon cancer cells to TRAIL-induced apoptosis of via autophagy- and JNK-mediated death receptors upregulation , 2016, Apoptosis.
[6] I. Dikic,et al. The integration of autophagy and cellular trafficking pathways via RAB GAPs , 2015, Autophagy.
[7] S. Carlsson,et al. Membrane dynamics in autophagosome biogenesis , 2015, Journal of Cell Science.
[8] Xiaojun Tan,et al. A Kinase-Independent Role for EGF Receptor in Autophagy Initiation , 2015, Cell.
[9] R. Schekman,et al. Phosphatidylinositol 3-kinase and COPII generate LC3 lipidation vesicles from the ER-Golgi intermediate compartment , 2014, eLife.
[10] Lin Mei,et al. The chemotherapeutic potential of PEG-b-PLGA copolymer micelles that combine chloroquine as autophagy inhibitor and docetaxel as an anti-cancer drug. , 2014, Biomaterials.
[11] John A. Tallarico,et al. Selective VPS34 inhibitor blocks autophagy and uncovers a role for NCOA4 in ferritin degradation and iron homeostasis in vivo , 2014, Nature Cell Biology.
[12] A. Ernst,et al. Cargo recognition and trafficking in selective autophagy , 2014, Nature Cell Biology.
[13] Zvulun Elazar,et al. Endocytosis and autophagy: exploitation or cooperation? , 2014, Cold Spring Harbor perspectives in biology.
[14] Doris Popovic,et al. TBC1D5 and the AP2 complex regulate ATG9 trafficking and initiation of autophagy , 2014, EMBO reports.
[15] S. Ghosh,et al. Regulation of Fibroblast Growth Factor-inducible 14 (Fn14) Expression Levels via Ligand-independent Lysosomal Degradation* , 2014, The Journal of Biological Chemistry.
[16] H. Wajant. The TWEAK‐Fn14 system as a potential drug target , 2013, British journal of pharmacology.
[17] Z. Elazar,et al. The Atg8 family: multifunctional ubiquitin-like key regulators of autophagy. , 2013, Essays in biochemistry.
[18] S. Tooze. Current views on the source of the autophagosome membrane. , 2013, Essays in biochemistry.
[19] N. Grishin,et al. EGFR-Mediated Beclin 1 Phosphorylation in Autophagy Suppression, Tumor Progression, and Tumor Chemoresistance , 2013, Cell.
[20] R. Schekman,et al. The ER–Golgi intermediate compartment is a key membrane source for the LC3 lipidation step of autophagosome biogenesis , 2013, eLife.
[21] Wei-dong Hu,et al. Application and interpretation of current autophagy inhibitors and activators , 2013, Acta Pharmacologica Sinica.
[22] W. Hittelman,et al. The TWEAK Receptor Fn14 is a Novel Therapeutic Target in Melanoma: Immunotoxins Targeting Fn14 Receptor for Malignant Melanoma Treatment , 2012, The Journal of investigative dermatology.
[23] N. Mizushima,et al. Ubiquitin-like proteins and autophagy at a glance , 2012, Journal of Cell Science.
[24] J. Harper,et al. Rab GTPase-Activating Proteins in Autophagy: Regulation of Endocytic and Autophagy Pathways by Direct Binding to Human ATG8 Modifiers , 2012, Molecular and Cellular Biology.
[25] Sankar Ghosh,et al. NF-κB, the first quarter-century: remarkable progress and outstanding questions. , 2012, Genes & development.
[26] Oh-Hyung Kwon,et al. Elevated fibroblast growth factor-inducible 14 expression promotes gastric cancer growth via nuclear factor-κB and is associated with poor patient outcome. , 2012, Cancer letters.
[27] T. Zheng,et al. TWEAK/Fn14 pathway: an immunological switch for shaping tissue responses , 2011, Immunological reviews.
[28] S. Ghosh,et al. Crosstalk in NF-κB signaling pathways , 2011, Nature Immunology.
[29] J. Ruland. Return to homeostasis: downregulation of NF-κB responses , 2011, Nature Immunology.
[30] Scott D Emr,et al. The ESCRT pathway. , 2011, Developmental cell.
[31] Z. Elazar,et al. Biogenesis and cargo selectivity of autophagosomes. , 2011, Annual review of biochemistry.
[32] T. Lamark,et al. Selective autophagy mediated by autophagic adapter proteins , 2011, Autophagy.
[33] K. Blackwell,et al. TRAF2 phosphorylation promotes NF-κB–dependent gene expression and inhibits oxidative stress-induced cell death , 2011, Molecular biology of the cell.
[34] Kay Hofmann,et al. Selective autophagy: ubiquitin-mediated recognition and beyond , 2010, Nature Cell Biology.
[35] Z. Elazar,et al. LC3 and GATE‐16/GABARAP subfamilies are both essential yet act differently in autophagosome biogenesis , 2010, The EMBO journal.
[36] S. Ybarra,et al. Antibodies to TWEAK Receptor Inhibit Human Tumor Growth through Dual Mechanisms , 2010, Clinical Cancer Research.
[37] D. Klionsky,et al. Regulation mechanisms and signaling pathways of autophagy. , 2009, Annual review of genetics.
[38] H. Wajant,et al. TNF-like weak inducer of apoptosis inhibits proinflammatory TNF receptor-1 signaling , 2009, Cell Death and Differentiation.
[39] Liying Gu,et al. TWEAK promotes ovarian cancer cell metastasis via NF-kappaB pathway activation and VEGF expression. , 2009, Cancer letters.
[40] N. Mizushima,et al. Atg14 and UVRAG: Mutually exclusive subunits of mammalian Beclin 1-PI3K complexes , 2009, Autophagy.
[41] M. Komatsu,et al. A role for NBR1 in autophagosomal degradation of ubiquitinated substrates. , 2009, Molecular cell.
[42] N. Mizushima,et al. Beclin 1 forms two distinct phosphatidylinositol 3-kinase complexes with mammalian Atg14 and UVRAG. , 2008, Molecular biology of the cell.
[43] T. Noda,et al. An Atg4B mutant hampers the lipidation of LC3 paralogues and causes defects in autophagosome closure. , 2008, Molecular biology of the cell.
[44] D. Vaux,et al. TWEAK-FN14 signaling induces lysosomal degradation of a cIAP1–TRAF2 complex to sensitize tumor cells to TNFα , 2008, The Journal of cell biology.
[45] J. Winkles. The TWEAK–Fn14 cytokine–receptor axis: discovery, biology and therapeutic targeting , 2008, Nature Reviews Drug Discovery.
[46] H. Cunliffe,et al. The Fibroblast Growth Factor–Inducible 14 Receptor Is Highly Expressed in HER2-Positive Breast Tumors and Regulates Breast Cancer Cell Invasive Capacity , 2008, Molecular Cancer Research.
[47] S. Ralston,et al. Ubiquitin Recognition by the Ubiquitin-associated Domain of p62 Involves a Novel Conformational Switch* , 2008, Journal of Biological Chemistry.
[48] A. Isaacs,et al. Functional multivesicular bodies are required for autophagic clearance of protein aggregates associated with neurodegenerative disease , 2007, The Journal of cell biology.
[49] R. Piper,et al. Biogenesis and function of multivesicular bodies. , 2007, Annual review of cell and developmental biology.
[50] M. Diaz-Meco,et al. Signal integration and diversification through the p62 scaffold protein. , 2007, Trends in biochemical sciences.
[51] G. Hostetter,et al. Increased fibroblast growth factor-inducible 14 expression levels promote glioma cell invasion via Rac1 and nuclear factor-kappaB and correlate with poor patient outcome. , 2006, Cancer research.
[52] Michael J Rust,et al. Sub-diffraction-limit imaging by stochastic optical reconstruction microscopy (STORM) , 2006, Nature Methods.
[53] C. Putterman,et al. The role of TWEAK/Fn14 in the pathogenesis of inflammation and systemic autoimmunity. , 2004, Frontiers in bioscience : a journal and virtual library.
[54] J. Winkles,et al. The Fn14 cytoplasmic tail binds tumour-necrosis-factor-receptor-associated factors 1, 2, 3 and 5 and mediates nuclear factor-kappaB activation. , 2003, The Biochemical journal.
[55] Kristen R. Ross,et al. The human Fn14 receptor gene is up-regulated in migrating glioma cells in vitro and overexpressed in advanced glial tumors. , 2003, The American journal of pathology.
[56] Yongwon Choi,et al. Regulation of the Subcellular Localization of Tumor Necrosis Factor Receptor–associated Factor (TRAF)2 by TRAF1 Reveals Mechanisms of TRAF2 Signaling , 2002, The Journal of experimental medicine.
[57] J. Pober,et al. Tumor necrosis factor receptor-associated factors (TRAFs) , 2001, Oncogene.
[58] J. Backer,et al. Vps34p differentially regulates endocytosis from the apical and basolateral domains in polarized hepatic cells , 2001, The Journal of cell biology.
[59] C. Thompson,et al. Translocation of TRAF proteins regulates apoptotic threshold of cells. , 2000, Biochemical and biophysical research communications.
[60] S. Thorgeirsson,et al. The Fn14 immediate-early response gene is induced during liver regeneration and highly expressed in both human and murine hepatocellular carcinomas. , 2000, The American journal of pathology.
[61] Hao Wu,et al. Structural basis for self-association and receptor recognition of human TRAF2 , 1999, Nature.
[62] S. Munro,et al. Retrieval of TGN proteins from the cell surface requires endosomal acidification. , 1994, The EMBO journal.