Elevation of phospholipase C-β1 expression by amyloid-β facilitates calcium overload in neuronal cells
暂无分享,去创建一个
W. Song | K. Lee | Byeong C. Kim | So Hee Kim | Y. Oh | Jiyun Park | Hwan Kim | K. Y. Choi | Yeong‐Jin Kim
[1] Razi Ahmad,et al. Alzheimer's disease and its treatment by different approaches: A review. , 2021, European journal of medicinal chemistry.
[2] D. Dickson,et al. The neuropathological diagnosis of Alzheimer’s disease , 2019, Molecular Neurodegeneration.
[3] K. Myers. PLCB1 Biallelic Point Mutations Cause West Syndrome. , 2019, Pediatric neurology.
[4] K. Cheung,et al. Calcium signaling in Alzheimer's disease & therapies. , 2018, Biochimica et biophysica acta. Molecular cell research.
[5] I. Bezprozvanny,et al. Dysregulation of Intracellular Calcium Signaling in Alzheimer's Disease. , 2018, Antioxidants & redox signaling.
[6] M. Stecker,et al. Amyloid toxicity in Alzheimer’s disease , 2018, Reviews in the neurosciences.
[7] M. Mesulam,et al. The cholinergic system in the pathophysiology and treatment of Alzheimer's disease. , 2018, Brain : a journal of neurology.
[8] S. Scarlata,et al. Phospholipase Cb1 regulates proliferation of neuronal cells , 2018, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[9] Sangyun Jeong,et al. Molecular and Cellular Basis of Neurodegeneration in Alzheimer’s Disease , 2017, Molecules and cells.
[10] A. Hannan,et al. Isoform specific differences in phospholipase C beta 1 expression in the prefrontal cortex in schizophrenia and suicide , 2017, npj Schizophrenia.
[11] Josiah R. Boivin,et al. Soluble oligomeric amyloid-β induces calcium dyshomeostasis that precedes synapse loss in the living mouse brain , 2017, Molecular Neurodegeneration.
[12] Z. Khachaturian,et al. Calcium Hypothesis of Alzheimer's disease and brain aging: A framework for integrating new evidence into a comprehensive theory of pathogenesis , 2017, Alzheimer's & Dementia.
[13] Jung-Mi Hah,et al. Nec‐1 alleviates cognitive impairment with reduction of Aβ and tau abnormalities in APP/PS1 mice , 2016, EMBO molecular medicine.
[14] J. Dorszewska,et al. Molecular Basis of Familial and Sporadic Alzheimer's Disease. , 2016, Current Alzheimer research.
[15] T. Rohacs. Phosphoinositide signaling in somatosensory neurons. , 2016, Advances in biological regulation.
[16] U. Sengupta,et al. The Role of Amyloid-β Oligomers in Toxicity, Propagation, and Immunotherapy , 2016, EBioMedicine.
[17] T. H. Ferreira-Vieira,et al. Alzheimer's Disease: Targeting the Cholinergic System , 2016, Current neuropharmacology.
[18] Sukchan Lee,et al. Phospholipase C-β1 Hypofunction in the Pathogenesis of Schizophrenia , 2015, Front. Psychiatry.
[19] Sagar H. Barage,et al. Amyloid cascade hypothesis: Pathogenesis and therapeutic strategies in Alzheimer's disease , 2015, Neuropeptides.
[20] K. Stover,et al. Early detection of cognitive deficits in the 3xTg-AD mouse model of Alzheimer's disease , 2015, Behavioural Brain Research.
[21] Xinglong Wang,et al. Oxidative stress and mitochondrial dysfunction in Alzheimer's disease. , 2014, Biochimica et biophysica acta.
[22] F. Roman,et al. Onset of hippocampus‐dependent memory impairments in 5XFAD transgenic mouse model of Alzheimer's disease , 2014, Hippocampus.
[23] Arthur Christopoulos,et al. Muscarinic acetylcholine receptors: novel opportunities for drug development , 2014, Nature Reviews Drug Discovery.
[24] I. Parker,et al. Cytotoxicity of Intracellular Aβ42 Amyloid Oligomers Involves Ca2+ Release from the Endoplasmic Reticulum by Stimulated Production of Inositol Trisphosphate , 2013, The Journal of Neuroscience.
[25] L. Mucke,et al. Neurotoxicity of amyloid β-protein: synaptic and network dysfunction. , 2012, Cold Spring Harbor perspectives in medicine.
[26] J. Jhamandas,et al. Neuronal receptors as targets for the action of amyloid-beta protein (Aβ) in the brain , 2012, Expert Reviews in Molecular Medicine.
[27] V. L. Vasco,et al. Deletion of PLCB1 gene in schizophrenia-affected patients , 2011, Journal of cellular and molecular medicine.
[28] I. Scheffer,et al. Phospholipase C beta 1 deficiency is associated with early-onset epileptic encephalopathy. , 2010, Brain : a journal of neurology.
[29] T. Suuronen,et al. ER stress in Alzheimer's disease: a novel neuronal trigger for inflammation and Alzheimer's pathology , 2009, Journal of Neuroinflammation.
[30] V. Vingtdeux,et al. Calcium signaling in neurodegeneration , 2009, Molecular Neurodegeneration.
[31] M. Ohno,et al. Impairments in remote memory stabilization precede hippocampal synaptic and cognitive failures in 5XFAD Alzheimer mouse model , 2009, Neurobiology of Disease.
[32] Masahiko Watanabe,et al. Predominant expression of phospholipase Cβ1 in telencephalic principal neurons and cerebellar interneurons, and its close association with related signaling molecules in somatodendritic neuronal elements , 2008, The European journal of neuroscience.
[33] R. Resende,et al. Neurotoxic effect of oligomeric and fibrillar species of amyloid-beta peptide 1-42: Involvement of endoplasmic reticulum calcium release in oligomer-induced cell death , 2008, Neuroscience.
[34] Kim N. Green,et al. Linking Calcium to Aβ and Alzheimer's Disease , 2008, Neuron.
[35] D. Alkon,et al. Insulin, PKC signaling pathways and synaptic remodeling during memory storage and neuronal repair. , 2008, European journal of pharmacology.
[36] M. Ohno,et al. Intraneuronal β-Amyloid Aggregates, Neurodegeneration, and Neuron Loss in Transgenic Mice with Five Familial Alzheimer's Disease Mutations: Potential Factors in Amyloid Plaque Formation , 2006, The Journal of Neuroscience.
[37] S. G. Lechner,et al. Presynaptic Inhibition via a Phospholipase C- and Phosphatidylinositol Bisphosphate-Dependent Regulation of Neuronal Ca2+ Channels , 2005, Molecular Pharmacology.
[38] C. Blakemore,et al. Activity-dependent Regulation of Synapse and Dendritic Spine Morphology in Developing Barrel Cortex Requires Phospholipase C-β1 Signalling , 2005 .
[39] Wendy W. Wu,et al. Biophysical alterations of hippocampal pyramidal neurons in learning, ageing and Alzheimer's disease , 2004, Ageing Research Reviews.
[40] C. Oliveira,et al. Involvement of endoplasmic reticulum Ca2+ release through ryanodine and inositol 1,4,5‐triphosphate receptors in the neurotoxic effects induced by the amyloid‐β peptide , 2004, Journal of neuroscience research.
[41] M. Duchen,et al. β-Amyloid Peptides Induce Mitochondrial Dysfunction and Oxidative Stress in Astrocytes and Death of Neurons through Activation of NADPH Oxidase , 2004, The Journal of Neuroscience.
[42] F. LaFerla. Calcium dyshomeostasis and intracellular signalling in alzheimer's disease , 2002, Nature Reviews Neuroscience.
[43] C. Plata-salamán,et al. Inflammation and Alzheimer’s disease , 2000, Neurobiology of Aging.
[44] P. Marie,et al. Activation of Phospholipase C-β1 via Gαq/11during Calcium Mobilization by Calcitonin Gene-related Peptide* , 1998, The Journal of Biological Chemistry.
[45] S. Ryu,et al. Phospholipase C isozymes selectively couple to specific neurotransmitter receptors , 1997, Nature.
[46] H. Loh,et al. U73122 inhibits phospholipase C-dependent calcium mobilization in neuronal cells , 1994, Brain Research.
[47] K. Suzuki,et al. Tissue- and cell type-specific expression of mRNAs for four types of inositol phospholipid-specific phospholipase C. , 1989, Biochemical and biophysical research communications.
[48] S. Y. Lee,et al. Studies of inositol phospholipid-specific phospholipase C. , 1989, Science.
[49] O. Krishtal,et al. Effects of calcium and calcium‐chelating agents on the inward and outward current in the membrane of mollusc neurones , 1977, The Journal of physiology.
[50] G. García del Caño,et al. Nuclear phospholipase C-β1 and diacylglycerol LIPASE-α in brain cortical neurons. , 2014, Advances in biological regulation.
[51] W. Müller,et al. Disturbances of the neuronal calcium homeostasis in the aging nervous system. , 1994, Life sciences.