Impairment of Bone Healing by Insulin Receptor Substrate-1 Deficiency*
暂无分享,去创建一个
Kozo Nakamura | Y. Terauchi | T. Kadowaki | T. Komori | W. Ogawa | M. Matsumoto | U. Chung | H. Kawaguchi | K. Hoshi | T. Akune | H. Chikuda | T. Shimoaka | S. Kamekura | Z. Maruyama | Satoru Kamekura | Takashi Shimoaka
[1] R. Baserga,et al. IGF-I receptor signalling in transformation and differentiation , 2001, Molecular pathology : MP.
[2] M. White,et al. IRS proteins and beta-cell function. , 2001, Diabetes.
[3] E. Canalis,et al. Cortisol inhibits the synthesis of insulin-like growth factor-I in skeletal cells. , 1990, Endocrinology.
[4] D. Rao,et al. Bone Mineral Density in Diabetes , 1997, Diabetes Care.
[5] C. Krettek,et al. Adenoviral gene transfer in a rat fracture model , 2002, Laboratory animals.
[6] V. Lefebvre,et al. Accelerated Up‐Regulation of L‐Sox5, Sox6, and Sox9 by BMP‐2 Gene Transfer During Murine Fracture Healing * , 2001, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[7] B. Calabretta,et al. Multiple Signaling Pathways of the Insulin-Like Growth Factor 1 Receptor in Protection from Apoptosis , 1999, Molecular and Cellular Biology.
[8] E. Canalis. Insulin like growth factors and the local regulation of bone formation. , 1992, Bone.
[9] Kozo Nakamura,et al. Evaluation by fluorescence resonance energy transfer of the stability of nonviral gene delivery vectors under physiological conditions. , 2002, Biomacromolecules.
[10] V. Lefebvre,et al. Characterization of primary cultures of chondrocytes from type II collagen/beta-galactosidase transgenic mice. , 1994, Matrix biology : journal of the International Society for Matrix Biology.
[11] J. Bonjour,et al. Mechanism of the Mitogenic Effect of Fluoride on Osteoblast‐like Cells: Evidences for a G Protein–Dependent Tyrosine Phosphorylation Process , 1997, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[12] T. Yagi,et al. Insulin resistance and growth retardation in mice lacking insulin receptor substrate-1 , 1994, Nature.
[13] N. Kanatani,et al. Skeletal Malformations Caused by Overexpression of Cbfa1 or Its Dominant Negative Form in Chondrocytes , 2001, The Journal of cell biology.
[14] A. Sacchi,et al. Insulin Receptor Substrate-1, p70S6K, and Cell Size in Transformation and Differentiation of Hemopoietic Cells* , 2000, The Journal of Biological Chemistry.
[15] J. Baker,et al. Mice carrying null mutations of the genes encoding insulin-like growth factor I (Igf-1) and type 1 IGF receptor (Igf1r) , 1993, Cell.
[16] Z. Laron,et al. Patients with Laron Syndrome Have Osteopenia/Osteoporosis , 1999, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[17] Weiqun Li,et al. RACK1, an Insulin-Like Growth Factor I (IGF-I) Receptor-Interacting Protein, Modulates IGF-I-Dependent Integrin Signaling and Promotes Cell Spreading and Contact with Extracellular Matrix , 2002, Molecular and Cellular Biology.
[18] Kozo Nakamura,et al. Regulation of Osteoblast, Chondrocyte, and Osteoclast Functions by Fibroblast Growth Factor (FGF)-18 in Comparison with FGF-2 and FGF-10* , 2002, The Journal of Biological Chemistry.
[19] Kozo Nakamura,et al. Insulin receptor substrate-2 maintains predominance of anabolic function over catabolic function of osteoblasts , 2002, The Journal of cell biology.
[20] S. Aizawa,et al. Disruption of insulin receptor substrate 2 causes type 2 diabetes because of liver insulin resistance and lack of compensatory beta-cell hyperplasia. , 2000, Diabetes.
[21] N. Kanatani,et al. Overexpression of Cbfa1 in osteoblasts inhibits osteoblast maturation and causes osteopenia with multiple fractures , 2001, The Journal of cell biology.
[22] G. Romano,et al. The role of the insulin receptor substrate-1 in the differentiation of rat hippocampal neuronal cells , 2001, Oncogene.
[23] Kozo Nakamura,et al. Deficiency of Insulin Receptor Substrate‐1 Impairs Skeletal Growth Through Early Closure of Epiphyseal Cartilage , 2003, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[24] C. Kahn,et al. Alternative pathway of insulin signalling in mice with targeted disruption of the IRS-1 gene , 1994, Nature.
[25] N. Sonenberg,et al. Insulin signalling and insulin actions in the muscles and livers of insulin-resistant, insulin receptor substrate 1-deficient mice , 1996, Molecular and cellular biology.
[26] J. Heeger,et al. Improvement of fracture healing by systemic administration of growth hormone and local application of insulin-like growth factor-1 and transforming growth factor-beta1. , 2002, Bone.
[27] C. Kahn,et al. Differential signaling by insulin receptor substrate 1 (IRS-1) and IRS-2 in IRS-1-deficient cells , 1997, Molecular and cellular biology.
[28] D. Graves,et al. Impaired Fracture Healing in the Absence of TNF‐α Signaling: The Role of TNF‐α in Endochondral Cartilage Resorption , 2003 .
[29] L. Powell-Braxton,et al. IGF-I is required for normal embryonic growth in mice. , 1993, Genes & development.
[30] Y. Azuma,et al. Insulin receptor substrate-1 in osteoblast is indispensable for maintaining bone turnover. , 2000, The Journal of clinical investigation.
[31] R. Berger,et al. Short-term glucocorticoid treatment of prepubertal mice decreases growth and IGF-I expression in the growth plate. , 2003, The Journal of endocrinology.
[32] W. Lopaczynski. Differential regulation of signaling pathways for insulin and insulin-like growth factor I. , 1999, Acta biochimica Polonica.
[33] M. Benito,et al. IGF-I: a mitogen also involved in differentiation processes in mammalian cells. , 1996, The international journal of biochemistry & cell biology.
[34] E. Canalis,et al. Insulin-like growth factor I mediates selective anabolic effects of parathyroid hormone in bone cultures. , 1989, The Journal of clinical investigation.
[35] G. Rodan,et al. Estradiol regulation of insulin-like growth factor-I expression in osteoblastic cells: evidence for transcriptional control. , 1991, Molecular endocrinology.
[36] M. Bolander,et al. Defects of early fracture-healing in experimental diabetes. , 1989, The Journal of bone and joint surgery. American volume.
[37] Mark E. Bolander,et al. Regulation of Fracture Repair by Growth Factors , 1992, Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine.
[38] Pier Paolo Pandolfi,et al. The p66shc adaptor protein controls oxidative stress response and life span in mammals , 1999, Nature.
[39] T. Forst,et al. Bone Mineral Density and Bone Metabolism in Diabetes Mellitus , 1997, Hormone and metabolic research = Hormon- und Stoffwechselforschung = Hormones et metabolisme.
[40] Z. Laron,et al. Insulin-like growth factor 1 (IGF-1): a growth hormone , 2001, Molecular pathology : MP.
[41] D. Gospodarowicz,et al. Growth requirements of low‐density rabbit costal chondrocyte cultures maintained in serum‐free medium , 1984, Journal of cellular physiology.
[42] Y. Yazaki,et al. Signal transduction mechanism of insulin and insulin-like growth factor-1. , 1996, Endocrine journal.
[43] P. Stern,et al. Insulin‐Like Growth Factor I Production Is Essential for Anabolic Effects of Thyroid Hormone in Osteoblasts , 2000, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[44] T. Atsumi,et al. Chondrogenic differentiation of clonal mouse embryonic cell line ATDC5 in vitro: differentiation-dependent gene expression of parathyroid hormone (PTH)/PTH-related peptide receptor , 1996, The Journal of cell biology.
[45] R. Burcelin,et al. Hypoinsulinaemia, glucose intolerance and diminished β-cell size in S6K1-deficient mice , 2000, Nature.