Intestinal microbial dysbiosis aggravates the progression of Alzheimer’s disease in Drosophila
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[1] Debomoy K Lahiri,et al. Targeting Tumor Necrosis Factor Alpha for Alzheimer's Disease. , 2016, Current Alzheimer research.
[2] D. Holtzman,et al. Antibiotic-induced perturbations in gut microbial diversity influences neuro-inflammation and amyloidosis in a murine model of Alzheimer’s disease , 2016, Scientific Reports.
[3] John D Lambris,et al. Microbial Endocrinology: Interkingdom Signaling in Infectious Disease and Health , 2016, Advances in Experimental Medicine and Biology.
[4] M. Deleidi,et al. Immune aging, dysmetabolism, and inflammation in neurological diseases , 2015, Front. Neurosci..
[5] S. Mazmanian,et al. Review Control of Brain Development, Function, and Behavior by the Microbiome Figure 2. Microbiome Influence on Bbb Integrity , 2022 .
[6] Theodore L. Roth,et al. Inflammation and neuroprotection in traumatic brain injury. , 2015, JAMA neurology.
[7] J. Shim,et al. Olfactory Control of Blood Progenitor Maintenance , 2013, Cell.
[8] B. Ganetzky,et al. Dnr1 mutations cause neurodegeneration in Drosophila by activating the innate immune response in the brain , 2013, Proceedings of the National Academy of Sciences.
[9] J. Brüning,et al. CNS insulin signaling in the control of energy homeostasis and glucose metabolism – from embryo to old age , 2013, Trends in Endocrinology & Metabolism.
[10] T. Dinan,et al. Mind-altering Microorganisms: the Impact of the Gut Microbiota on Brain and Behaviour , 2022 .
[11] 莊志立. Infection-induced intestinal oxidative stress triggers organ-to-organ immunological communication in Drosophila , 2012 .
[12] L. Mucke,et al. Alzheimer Mechanisms and Therapeutic Strategies , 2012, Cell.
[13] In-Hwan Jang,et al. Genetic evidence of a redox‐dependent systemic wound response via Hayan Protease‐Phenoloxidase system in Drosophila , 2012, The EMBO journal.
[14] B. Strooper,et al. The amyloid cascade hypothesis for Alzheimer's disease: an appraisal for the development of therapeutics , 2011, Nature Reviews Drug Discovery.
[15] J. C. Pastor-Pareja,et al. Shaping cells and organs in Drosophila by opposing roles of fat body-secreted Collagen IV and perlecan. , 2011, Developmental cell.
[16] N. Perrimon,et al. Drosophila as a model for interorgan communication: lessons from studies on energy homeostasis. , 2011, Developmental cell.
[17] Tallie Z. Baram,et al. The role of inflammation in epilepsy , 2011, Nature Reviews Neurology.
[18] Y. Michotte,et al. The dual role of the neuroinflammatory response after ischemic stroke: modulatory effects of hypothermia , 2010, Journal of Neuroinflammation.
[19] Eng H. Lo,et al. The Science of Stroke: Mechanisms in Search of Treatments , 2010, Neuron.
[20] G. Conductier,et al. The role of monocyte chemoattractant protein MCP1/CCL2 in neuroinflammatory diseases , 2010, Journal of Neuroimmunology.
[21] Fred H. Gage,et al. Mechanisms Underlying Inflammation in Neurodegeneration , 2010, Cell.
[22] Paul Martin,et al. Prioritization of Competing Damage and Developmental Signals by Migrating Macrophages in the Drosophila Embryo , 2010, Current Biology.
[23] Neuron. Immune Activation in Brain Aging and Neurodegeneration: Too Much or Too Little? , 2010 .
[24] J. Daly. A tissue-scale gradient of hydrogen peroxide mediates rapid wound detection in zebrafish , 2010 .
[25] F. LaFerla,et al. Alzheimer's disease. , 2010, The New England journal of medicine.
[26] Eng H. Lo,et al. The Science of Stroke: Mechanisms in Search of Treatments , 2010, Neuron.
[27] Y. Ip,et al. Tissue damage-induced intestinal stem cell division in Drosophila. , 2009, Cell stem cell.
[28] E. Hirsch,et al. Infiltration of CD4+ lymphocytes into the brain contributes to neurodegeneration in a mouse model of Parkinson disease. , 2008, The Journal of clinical investigation.
[29] Jun Tan,et al. Blocking TGF-β–Smad2/3 innate immune signaling mitigates Alzheimer-like pathology , 2008, Nature Medicine.
[30] G. Sykiotis,et al. Keap1/Nrf2 signaling regulates oxidative stress tolerance and lifespan in Drosophila. , 2008, Developmental cell.
[31] I. Andó,et al. Supplemental Experimental Procedures Drosophila Strains , 2022 .
[32] C. Geula,et al. Ccr2 deficiency impairs microglial accumulation and accelerates progression of Alzheimer-like disease , 2007, Nature Medicine.
[33] Qing Wang,et al. The inflammatory response in stroke , 2007, Journal of Neuroimmunology.
[34] B. Oh,et al. The Drosophila amidase PGRP-LB modulates the immune response to bacterial infection. , 2006, Immunity.
[35] J. Julien,et al. Bone Marrow-Derived Microglia Play a Critical Role in Restricting Senile Plaque Formation in Alzheimer's Disease , 2006, Neuron.
[36] D. Kelvin,et al. Defining the Origins and Evolution of the Chemokine/Chemokine Receptor System1 , 2006, The Journal of Immunology.
[37] Ann-Shyn Chiang,et al. Dissecting the pathological effects of human Aβ40 and Aβ42 in Drosophila: A potential model for Alzheimer's disease , 2004 .
[38] Yahong Lin,et al. A JNK-Dependent Pathway Is Required for TNFα-Induced Apoptosis , 2003, Cell.
[39] B. Lemaître,et al. A single gene that promotes interaction of a phytopathogenic bacterium with its insect vector, Drosophila melanogaster , 2003, EMBO reports.
[40] Yahong Lin,et al. A JNK-dependent pathway is required for TNFalpha-induced apoptosis. , 2003, Cell.
[41] T. Aigaki,et al. Eiger, a TNF superfamily ligand that triggers the Drosophila JNK pathway , 2002, The EMBO journal.
[42] B. Lemaître,et al. Tissue-specific inducible expression of antimicrobial peptide genes in Drosophila surface epithelia. , 2000, Immunity.
[43] Y. Koh,et al. Regulation of DLG Localization at Synapses by CaMKII-Dependent Phosphorylation , 1999, Cell.
[44] R. Goyal,et al. The enteric nervous system. , 1996, The New England journal of medicine.