Pentoxifylline reduces acute lung injury in chronic endotoxemia.
暂无分享,去创建一个
W. Loomis | D. Hoyt | R. Coimbra | C. Michetti | W. Junger | P. Wolf
[1] R. McIntyre,et al. TNF receptor I mediates chemokine production and neutrophil accumulation in the lung following systemic lipopolysaccharide. , 2001, The Journal of surgical research.
[2] R. Dickerson,et al. Effect of pentoxifylline on nitrogen balance and 3-methylhistidine excretion in parenterally fed endotoxemic rats. , 2001, Nutrition.
[3] T. Scalea,et al. Acute respiratory distress syndrome among trauma patients: trends in ICU mortality, risk factors, complications and resource utilization , 2001, Intensive Care Medicine.
[4] J. Hogg,et al. Interleukin-6 changes deformability of neutrophils and induces their sequestration in the lung. , 2001, American journal of respiratory and critical care medicine.
[5] H. Hashimoto,et al. Pentoxifylline induces the shedding of L-selectin on polymorphonuclear cells by stimulation via adenosine receptor as well as by the inhibition of phosphodiesterase , 2001, Modern rheumatology.
[6] M. Wilson,et al. Lazaroid and pentoxifylline suppress sepsis-induced increases in renal vascular resistance via altered arachidonic acid metabolism. , 2000, The Journal of surgical research.
[7] P. Wang,et al. Mechanism of the beneficial effects of pentoxifylline during sepsis: maintenance of adrenomedullin responsiveness and downregulation of proinflammatory cytokines. , 2000, The Journal of surgical research.
[8] T. Krakauer. Pentoxifylline inhibits ICAM-1 expression and chemokine production induced by proinflammatory cytokines in human pulmonary epithelial cells. , 2000, Immunopharmacology.
[9] B. Beck-Schimmer,et al. Lipopolysaccharide induces functional ICAM-1 expression in rat alveolar epithelial cells in vitro. , 2000, American journal of physiology. Lung cellular and molecular physiology.
[10] R. Coimbra,et al. Hypertonic saline and pentoxifylline prevent lung injury and bacterial translocation after hemorrhagic shock. , 1999, Shock.
[11] S. Tasaka,et al. CD11/CD18-dependent and -independent neutrophil emigration in the lungs: how do neutrophils know which route to take? , 2000, American journal of respiratory cell and molecular biology.
[12] G. Zibari,et al. Effects of dextran and pentoxifylline on hemorrhagic shock-induced P-selectin expression. , 1999, The Journal of surgical research.
[13] K. Ley,et al. P-selectin and ICAM-1 mediate endotoxin-induced neutrophil recruitment and injury to the lung and liver. , 1999, The American journal of physiology.
[14] R. Shenkar,et al. Mechanisms of lung neutrophil activation after hemorrhage or endotoxemia: roles of reactive oxygen intermediates, NF-kappa B, and cyclic AMP response element binding protein. , 1999, Journal of immunology.
[15] H. Repo,et al. Mechanisms and consequences of phagocyte adhesion to endothelium. , 1999, Annals of medicine.
[16] M. Yen,et al. Pentoxifylline improves circulatory failure and survival in murine models of endotoxaemia. , 1999, European journal of pharmacology.
[17] R. Urbaschek,et al. Pentoxifylline improves bacterial clearance during hemorrhage and endotoxemia. , 1999, Critical care medicine.
[18] D. Breuillé,et al. Cytokine modulation by PX differently affects specific acute phase proteins during sepsis in rats. , 1998, American journal of physiology. Regulatory, integrative and comparative physiology.
[19] C. Wiedermann,et al. Pentoxifylline Differentially Regulates Migration and Respiratory Burst Activity of the Neutrophil a , 1997, Annals of the New York Academy of Sciences.
[20] H. Redmond,et al. INTERCELLULAR ADHESION MOLECULE‐1 (ICAM‐1) IS EXPRESSED ON HUMAN NEUTROPHILS AND IS ESSENTIAL FOR NEUTROPHIL ADHERENCE AND AGGREGATION , 1997, Shock.
[21] C. Parkos. I. Neutrophil adhesive interactions with intestinal epithelium. , 1997, American journal of physiology. Gastrointestinal and liver physiology.
[22] P. Ward,et al. Expression of lung vascular and airway ICAM-1 after exposure to bacterial lipopolysaccharide. , 1997, American journal of respiratory cell and molecular biology.
[23] R. McIntyre,et al. Pentoxifylline treatment attenuates pulmonary vasomotor dysfunction in acute lung injury. , 1997, The Journal of surgical research.
[24] Y. Mándi,et al. Effects of tumor necrosis factor and pentoxifylline on ICAM-1 expression on human polymorphonuclear granulocytes. , 1997, International Archives of Allergy and Immunology.
[25] D. Hoyt,et al. Acute lung injury in endotoxemic rats is associated with sustained circulating IL-6 levels and intrapulmonary CINC activity and neutrophil recruitment--role of circulating TNF-alpha and IL-beta? , 1996, Shock.
[26] J. Hakim,et al. Inhibition of human neutrophil binding to hydrogen peroxide-treated endothelial cells by cAMP and hydroxyl radical scavengers. , 1996, Free radical biology & medicine.
[27] A. Nüssler,et al. Proinflammatory cytokines and endotoxin stimulate ICAM-1 gene expression and secretion by normal human hepatocytes. , 1994, Immunology.
[28] T. van der Poll,et al. Pentoxifylline attenuates neutrophil activation in experimental endotoxemia in chimpanzees. , 1993, Journal of immunology.
[29] A. Fowler,et al. Role of the neutrophil in adult respiratory distress syndrome , 1993, The British journal of surgery.
[30] V. Nadkarni,et al. Effects of in vivo pentoxifylline treatment on survival and ex vivo vascular contractility in a rat lipopolysaccharide shock model. , 1992, Circulatory shock.
[31] E. Chi,et al. Pulmonary effects of continuous endotoxin infusion in the rat. , 1991, Circulatory shock.
[32] R. Gerzer,et al. Cyclic nucleotides differentially regulate the synthesis of tumour necrosis factor-alpha and interleukin-1 beta by human mononuclear cells. , 1991, Immunology.
[33] R. Strieter,et al. Cellular and molecular regulation of tumor necrosis factor-alpha production by pentoxofylline , 1988 .
[34] W. J. Novick,et al. Inhibition of the inflammatory action of interleukin-1 and tumor necrosis factor (alpha) on neutrophil function by pentoxifylline , 1988, Infection and immunity.
[35] R. Vestal,et al. Polymorphonuclear leukocyte inhibition by therapeutic concentrations of theophylline is mediated by cyclic-3',5'-adenosine monophosphate. , 1988, The American review of respiratory disease.
[36] J. Michael,et al. Pharmacological modification of pulmonary vascular injury: possible role of cAMP. , 1987, Journal of applied physiology.
[37] D. Strandness,et al. Pentoxifylline efficacy in the treatment of intermittent claudication: multicenter controlled double-blind trial with objective assessment of chronic occlusive arterial disease patients. , 1982, American heart journal.