Excitatory-Inhibitory Balance

[1]  M. Jouvet,et al.  Brain stem PGO-on cells projecting directly to the cat dorsal lateral geniculate nucleus , 1980, Brain Research.

[2]  Tadaharu Tsumoto,et al.  Long-term potentiation and long-term depression in the neocortex , 1992, Progress in Neurobiology.

[3]  Mark F. Bear,et al.  Neocortical long-term potentiation , 1993, Current Opinion in Neurobiology.

[4]  J. Money,et al.  Suprachiasmatic nucleus: the mind's clock , 1993 .

[5]  Y. Komatsu,et al.  Long-term modification of inhibitory synaptic transmission in developing visual cortex. , 1993, Neuroreport.

[6]  B. McNaughton,et al.  Synaptic enhancement in fascia dentata: Cooperativity among coactive afferents , 1978, Brain Research.

[7]  M. Jouvet,et al.  Single unit recordings in the nuclei raphe dorsalis and magnus during the sleep-waking cycle of semi-chronic prepared cats , 1981, Neuroscience Letters.

[8]  M. Bear,et al.  Common forms of synaptic plasticity in the hippocampus and neocortex in vitro. , 1993, Science.

[9]  J. Pettigrew,et al.  Depletion of brain catecholamines: failure of ocular dominance shift after monocular occlusion in kittens. , 1976, Science.

[10]  Y. Komatsu,et al.  GABAB Receptors, Monoamine Receptors, and Postsynaptic Inositol Trisphosphate-Induced Ca2+ Release Are Involved in the Induction of Long-Term Potentiation at Visual Cortical Inhibitory Synapses , 1996, The Journal of Neuroscience.

[11]  J. Lisman.  The CaM kinase II hypothesis for the storage of synaptic memory , 1994, Trends in Neurosciences.

[12]  Y. Yoshimura,et al.  Activity-Dependent Maintenance of Long-Term Potentiation at Visual Cortical Inhibitory Synapses , 2000, The Journal of Neuroscience.

[13]  J. Young,et al.  GABAB Receptor‐Mediated Inhibition of Histamine H1‐Receptor‐Induced Inositol Phosphate Formation in Slices of Rat Cerebral Cortex , 1988, Journal of neurochemistry.

[14]  M. Stryker,et al.  Neural plasticity without postsynaptic action potentials: less-active inputs become dominant when kitten visual cortical cells are pharmacologically inhibited. , 1988, Proceedings of the National Academy of Sciences of the United States of America.

[15]  Y. Komatsu,et al.  Age-dependent long-term potentiation of inhibitory synaptic transmission in rat visual cortex , 1994, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[16]  B. Gustafsson,et al.  Long-term potentiation in the hippocampus using depolarizing current pulses as the conditioning stimulus to single volley synaptic potentials , 1987, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[17]  T. Wiesel.  Postnatal development of the visual cortex and the influence of environment , 1982, Nature.

[18]  Hiroshi Kato,et al.  Reversal of long-term potentiation (depotentiation) induced by tetanus stimulation of the input to CA1 neurons of guinea pig hippocampal slices , 1991, Brain Research.

[19]  R. Malinow,et al.  Postsynaptic hyperpolarization during conditioning reversibly blocks induction of long-term potentiation , 1986, Nature.

[20]  J. Gaiarsa,et al.  Bidirectional plasticity expressed by GABAergic synapses in the neonatal rat hippocampus. , 1996, The Journal of physiology.

[21]  Gary Lynch,et al.  Stable depression of potentiated synaptic responses in the hippocampus with 1–5 Hz stimulation , 1990, Brain Research.

[22]  D. Johnston,et al.  NMDA-receptor-independent long-term potentiation. , 1992, Annual review of physiology.

[23]  G. Shepherd,et al.  Long-term modifications of synaptic efficacy in the human inferior and middle temporal cortex. , 1996, Proceedings of the National Academy of Sciences of the United States of America.

[24]  Y. Frégnac,et al.  Development of neuronal selectivity in primary visual cortex of cat. , 1984, Physiological reviews.

[25]  T. Tsumoto,et al.  Modification of orientation sensitivity of cat visual cortex neurons by removal of GABA-mediated inhibition , 1979, Experimental Brain Research.

[26]  R. Nicoll,et al.  NMDA-receptor-dependent synaptic plasticity: multiple forms and mechanisms , 1993, Trends in Neurosciences.

[27]  Y. Frégnac,et al.  A cellular analogue of visual cortical plasticity , 1988, Nature.

[28]  W. Singer,et al.  Different voltage-dependent thresholds for inducing long-term depression and long-term potentiation in slices of rat visual cortex , 1990, Nature.

[29]  Ronald M. Harper,et al.  Dorsal raphe neurons: depression of firing during sleep in cats , 1976, Brain Research.

[30]  A. Sillito.  The contribution of inhibitory mechanisms to the receptive field properties of neurones in the striate cortex of the cat. , 1975, The Journal of physiology.

[31]  M. Bear,et al.  Long-term depression in hippocampus. , 1996, Annual review of neuroscience.

[32]  Thanos Tzounopoulos,et al.  A Role for cAMP in Long-Term Depression at Hippocampal Mossy Fiber Synapses , 1998, Neuron.

[33]  B. Alger,et al.  Pharmacology of GABA and Glycine Neurotransmission , 2001, Handbook of Experimental Pharmacology.

[34]  W Singer,et al.  Involvement of Serotonin in Developmental Plasticity of Kitten Visual Cortex , 1995, The European journal of neuroscience.