Sheet migration by wounded monolayers as an emergent property of single-cell dynamics
暂无分享,去创建一个
[1] W. Carter,et al. Protein kinase C spatially and temporally regulates gap junctional communication during human wound repair via phosphorylation of connexin43 on serine368 , 2004, The Journal of cell biology.
[2] J. Southgate,et al. Agent-based computational modeling of wounded epithelial cell monolayers , 2004, IEEE Transactions on NanoBioscience.
[3] M. Abercrombie,et al. Observations on the social behaviour of cells in tissue culture. I. Speed of movement of chick heart fibroblasts in relation to their mutual contacts. , 1953, Experimental cell research.
[4] H. Othmer,et al. Models of dispersal in biological systems , 1988, Journal of mathematical biology.
[5] Y. Hegerfeldt,et al. Collective cell movement in primary melanoma explants: plasticity of cell-cell interaction, beta1-integrin function, and migration strategies. , 2002, Cancer research.
[6] H. Green,et al. QUANTITATIVE STUDIES OF THE GROWTH OF MOUSE EMBRYO CELLS IN CULTURE AND THEIR DEVELOPMENT INTO ESTABLISHED LINES , 1963, The Journal of cell biology.
[7] W. Carter,et al. Protein kinase C spatially and temporally regulates gap junctional communication during human wound repair via phosphorylation of connexin 43 on serine 368 , 2004 .
[8] P. Friedl,et al. Collective cell migration in morphogenesis and cancer. , 2004, The International journal of developmental biology.
[9] S. Kondo,et al. Effects of angiotensin II receptor signaling during skin wound healing. , 2004, The American journal of pathology.
[10] R. Anderson,et al. Mechanisms of recovery from mechanical injury of renal tubular epithelial cells. , 1994, The American journal of physiology.
[11] R. Dulbecco,et al. Conditions determining initiation of DNA synthesis in 3T3 cells. , 1970, Proceedings of the National Academy of Sciences of the United States of America.
[12] Bretschneider,et al. A model for dictyostelium slug movement , 1999, Journal of theoretical biology.
[13] Walter S. Krawczyk,et al. A PATTERN OF EPIDERMAL CELL MIGRATION DURING WOUND HEALING , 1971, The Journal of cell biology.
[14] Bing Li,et al. Inhibition of vascular endothelial growth factor-induced angiogenesis suppresses tumour growth in vivo , 1993, Nature.
[15] T. Nishio,et al. Tenascin-C regulates proliferation and migration of cultured astrocytes in a scratch wound assay , 2005, Neuroscience.
[16] H Green,et al. The initiation of cell division in a contact-inhibited mammalian cell line. , 1965, Journal of cellular physiology.
[17] P. Friedl. Prespecification and plasticity: shifting mechanisms of cell migration. , 2004, Current opinion in cell biology.
[18] T. Vicsek,et al. Proliferative and migratory responses of astrocytes to in vitro injury , 2000, Journal of neuroscience research.
[19] Rizwan U. Farooqui,et al. Multiple rows of cells behind an epithelial wound edge extend cryptic lamellipodia to collectively drive cell-sheet movement , 2005, Journal of Cell Science.
[20] Timothy J Mitchison,et al. Screening for cell migration inhibitors via automated microscopy reveals a Rho-kinase inhibitor. , 2005, Chemistry & biology.
[21] R. Isseroff,et al. β2‐Adrenergic receptor activation delays wound healing , 2006 .
[22] Paul Martin,et al. Parallels between tissue repair and embryo morphogenesis , 2004, Development.
[23] C. S. Chen,et al. Geometric control of cell life and death. , 1997, Science.
[24] M. Corada,et al. Contact inhibition of VEGF-induced proliferation requires vascular endothelial cadherin, β-catenin, and the phosphatase DEP-1/CD148 , 2003, The Journal of cell biology.
[25] J. Folkman,et al. Role of cell shape in growth control , 1978, Nature.
[26] Paul Martin,et al. Wound Healing--Aiming for Perfect Skin Regeneration , 1997, Science.
[27] L. Castor,et al. Flattening, movement and control of division of epithelial‐like cells , 1970, Journal of cellular physiology.
[28] J. Klarlund,et al. Wounding Induces Motility in Sheets of Corneal Epithelial Cells through Loss of Spatial Constraints , 2004, Journal of Biological Chemistry.
[29] J. Zieske. Extracellular matrix and wound healing , 2001, Current opinion in ophthalmology.
[30] M. Abercrombie,et al. Observations on the social behaviour of cells in tissue culture. II. Monolayering of fibroblasts. , 1954, Experimental cell research.
[31] R. Isseroff,et al. Beta2-adrenergic receptor activation delays wound healing. , 2006, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[32] J. Sherratt,et al. Intercellular adhesion and cancer invasion: a discrete simulation using the extended Potts model. , 2002, Journal of theoretical biology.
[33] P J Sammak,et al. How do injured cells communicate with the surviving cell monolayer? , 1997, Journal of cell science.
[34] J. Forrester,et al. Insulin, not leptin, promotes in vitro cell migration to heal monolayer wounds in human corneal epithelium. , 2004, Investigative ophthalmology & visual science.