Insulin sensitivity is decreased in normal women by doses of ethinyl estradiol used in oral contraceptives.

[1]  I. Godsland,et al.  The effects of different formulations of oral contraceptive agents on lipid and carbohydrate metabolism. , 1990, The New England journal of medicine.

[2]  W. Spellacy,et al.  The effects of two triphasic oral contraceptives on carbohydrate metabolism in women during 1 year of use. , 1989, Fertility and sterility.

[3]  I. Godsland,et al.  Clinical and metabolic considerations of long-term oral contraceptive use. , 1992, American journal of obstetrics and gynecology.

[4]  W. Droegemueller,et al.  Triphasic Randomized Clinical Trial: Comparison of effects on carbohydrate metabolism. , 1989, American Journal of Obstetrics and Gynecology.

[5]  R. Lobo Lipids, clotting factors, and diabetes: endogenous risk factors for cardiovascular disease. , 1988, American journal of obstetrics and gynecology.

[6]  P. Sönksen,et al.  Insulin Metabolism, Insulin Sensitivity and Hormonal Responses to Insulin Infusion in Patients Taking Oral Contraceptive Steroids , 1975, European journal of clinical investigation.

[7]  N. Saurbrey,et al.  Oral contraception and insulin sensitivity: in vivo assessment in normal women and women with previous gestational diabetes. , 1987, The Journal of clinical endocrinology and metabolism.

[8]  W. Spellacy Carbohydrate metabolism during treatment with estrogen, progestogen, and low-dose oral contraceptives. , 1982, American journal of obstetrics and gynecology.

[9]  W. Spellacy,et al.  The effects of two years of mestranol treatment on carbohydrate metabolism. , 1982, Metabolism: clinical and experimental.

[10]  R. Lobo,et al.  A possible bimodal effect of estrogen on insulin sensitivity in postmenopausal women and the attenuating effect of added progestin. , 1993, Fertility and sterility.

[11]  G. Kasdorf,et al.  Prospective studies of insulin sensitivity in normal women receiving oral contraceptive agents. , 1988, The Journal of clinical endocrinology and metabolism.

[12]  R N Bergman,et al.  Assessment of insulin sensitivity in vivo. , 1985, Endocrine reviews.

[13]  V. Herbert,et al.  Coated charcoal immunoassay of insulin. , 1965, The Journal of clinical endocrinology and metabolism.

[14]  J. Fiet,et al.  Metabolic Effect at Six and Twelve Months of Cyproterone Acetate (2 mg) Combined with Ethinyl Estradiol (35 μg) in 31 Patients , 1990 .

[15]  U. Gaspard,et al.  Effects of a 1-year treatment with a low-dose combined oral contraceptive containing ethinyl estradiol and cyproterone acetate on glucose and insulin metabolism. , 1993, Fertility and sterility.

[16]  R. Krauss,et al.  The metabolic impact of oral contraceptives. , 1992, American journal of obstetrics and gynecology.

[17]  U. Gaspard,et al.  Effects of ethinyl estradiol combined with desogestrel and cyproterone acetate on glucose tolerance and insulin response to an oral glucose load: a one-year randomized, prospective, comparative trial. , 1990, American journal of obstetrics and gynecology.

[18]  W. Spellacy,et al.  The effect of estrogens on carbohydrate metabolism: glucose, insulin, and growth hormone studies on one hundred and seventy-one women ingesting Premarin, mestranol, and ethinyl estradiol for six months. , 1972, American journal of obstetrics and gynecology.

[19]  C. Zancanaro,et al.  Estimates of in vivo insulin action in man: comparison of insulin tolerance tests with euglycemic and hyperglycemic glucose clamp studies. , 1989, The Journal of clinical endocrinology and metabolism.

[20]  I. Godsland,et al.  Insulin resistance, secretion, and metabolism in users of oral contraceptives. , 1992, The Journal of clinical endocrinology and metabolism.

[21]  P. Trinder Determination of Glucose in Blood Using Glucose Oxidase with an Alternative Oxygen Acceptor , 1969 .