Concanavalin A-Induced Hepatitis and Fas Ligand in α Disparate Roles for TNF-

D. MacKay and Lyle L. MoldawerJames Norman, Woody Denham, Michael Clare-Salzler, Sally L.Carl K. Edwards III, Cynthia L. Tannahill, Carmen C. Solorzano, Riadh Ksontini, Dorothy B. Colagiovanni, Michael D. Josephs,http://www.jimmunol.org/content/160/8/4082J Immunol€1998; 160:4082-4089; ;Referenceshttp://www.jimmunol.org/content/160/8/4082.full#ref-list-1This article cites 39 articles, 11 of which you can access for free at: Subscriptionshttp://jimmunol.org/subscriptionsInformation about subscribing to The Journal of Immunology is online at: Permissionshttp://www.aai.org/ji/copyright.htmlSubmit copyright permission requests at: Email Alertshttp://jimmunol.org/cgi/alerts/etocReceive free email-alerts when new articles cite this article. Sign up at:

[1]  Y. Iwakura,et al.  Suppression of concanavalin A-induced hepatitis in IFN-gamma(-/-) mice, but not in TNF-alpha(-/-) mice: role for IFN-gamma in activating apoptosis of hepatocytes. , 1997, Journal of immunology.

[2]  L. Moldawer,et al.  A MATRIX METALLOPROTEINASE INHIBITOR PREVENTS PROCESSING OF TUMOR NECROSIS FACTOR α (TNFα) AND ABROGATES ENDOTOXIN‐INDUCED LETHALITY , 1997 .

[3]  S. Nagata,et al.  Essential roles of the Fas ligand in the development of hepatitis , 1997, Nature Medicine.

[4]  R. Kamen,et al.  Caspase-1 processes IFN-γ-inducing factor and regulates LPS-induced IFN- γ production , 1997, Nature.

[5]  J. Mountz,et al.  MACROPHAGE ACTIVATION‐INDUCED CELL DEATH IS MEDIATED BY FAS LIGAND AND TNF: 458 , 1997 .

[6]  M. Lambert,et al.  Cloning of a disintegrin metalloproteinase that processes precursor tumour-necrosis factor-α , 1997, Nature.

[7]  Nicole Nelson,et al.  A metalloproteinase disintegrin that releases tumour-necrosis factor-α from cells , 1997, Nature.

[8]  S. Nagata,et al.  Apoptosis by Death Factor , 1997, Cell.

[9]  J. Vauthey,et al.  Involvement of 26-kDa cell-associated TNF-alpha in experimental hepatitis and exacerbation of liver injury with a matrix metalloproteinase inhibitor. , 1997, Journal of immunology.

[10]  Douglas K. Miller,et al.  The interleukin‐1β converting enzyme family of cysteine proteases , 1997 .

[11]  S. Nagata,et al.  Systemic injection of a tripeptide inhibits the intracellular activation of CPP32-like proteases in vivo and fully protects mice against Fas-mediated fulminant liver destruction and death , 1996, The Journal of experimental medicine.

[12]  Junying Yuan,et al.  Human ICE/CED-3 Protease Nomenclature , 1996, Cell.

[13]  G. Gerken,et al.  Role of sinusoidal endothelial cells of the liver in concanavalin A‐induced hepatic injury in mice , 1996, Hepatology.

[14]  J. Mountz,et al.  Autoimmune disease results from multiple interactive defects in apoptosis induction molecules and signaling pathways. , 1996, Behring Institute Mitteilungen.

[15]  T. Akaike,et al.  Concanavalin A induces perforin‐mediated but not Fas‐mediated hepatic injury , 1996, Hepatology.

[16]  P. Kiener,et al.  Differential expression of Fas (CD95) and Fas ligand on normal human phagocytes: implications for the regulation of apoptosis in neutrophils , 1996, The Journal of experimental medicine.

[17]  S. Küsters,et al.  Interferon gamma plays a critical role in T cell-dependent liver injury in mice initiated by concanavalin A. , 1996, Gastroenterology.

[18]  G. Evan,et al.  A License to Kill , 1996, Cell.

[19]  A. H. Drummond,et al.  Fas ligand in human serum , 1996, Nature Medicine.

[20]  K. Yoshino,et al.  Metalloproteinase-mediated Release of Human Fas Ligand , 1995 .

[21]  D. Green,et al.  Fas Ligand-Induced Apoptosis as a Mechanism of Immune Privilege , 1995, Science.

[22]  P. Galle,et al.  Involvement of the CD95 (APO-1/Fas) receptor and ligand in liver damage , 1995, The Journal of experimental medicine.

[23]  Seamus J. Martin,et al.  Protease activation during apoptosis: Death by a thousand cuts? , 1995, Cell.

[24]  S. Lowry,et al.  PEG-BP-30 monotherapy attenuates the cytokine-mediated inflammatory cascade in baboon Escherichia coli septic shock. , 1995, The Journal of surgical research.

[25]  J. Cleveland,et al.  Contenders in FasL/TNF death signaling , 1995, Cell.

[26]  W. Fiers,et al.  Requirement of an ICE/CED-3 protease for Fas/APO-1-mediated apoptosis , 1995, Nature.

[27]  S. Nagata,et al.  Involvement of an ICE-like protease in Fas-mediated apoptosis , 1995, Nature.

[28]  T. Yokota,et al.  Expression of the Fas ligand in cells of T cell lineage. , 1995, Journal of immunology.

[29]  M. Su,et al.  Altered cytokine export and apoptosis in mice deficient in interleukin-1 beta converting enzyme. , 1995, Science.

[30]  M. Leist,et al.  Concanavalin A—induced T‐cell—mediated hepatic injury in mice: The role of tumor necrosis factor , 1995, Hepatology.

[31]  L. Steinman,et al.  Reversal of experimental autoimmune encephalomyelitis with a hydroxamate inhibitor of matrix metalloproteases. , 1994, The Journal of clinical investigation.

[32]  E. O’Neill,et al.  T cell activation-associated hepatic injury: mediation by tumor necrosis factors and protection by interleukin 6 , 1994, The Journal of experimental medicine.

[33]  P. Golstein,et al.  Fas‐based lymphocyte‐mediated cytotoxicity against syngeneic activated lymphocytes: A regulatory pathway? , 1994, European journal of immunology.

[34]  N. Jenkins,et al.  Generalized lymphoproliferative disease in mice, caused by a point mutation in the fas ligand , 1994, Cell.

[35]  Shai Shaham,et al.  The C. elegans cell death gene ced-3 encodes a protein similar to mammalian interleukin-1β-converting enzyme , 1993, Cell.

[36]  S. Nagata,et al.  Lethal effect of the anti-Fas antibody in mice , 1993, Nature.

[37]  D. Grobelny,et al.  Inhibition of human skin fibroblast collagenase, thermolysin, and Pseudomonas aeruginosa elastase by peptide hydroxamic acids. , 1992, Biochemistry.

[38]  A. Wendel,et al.  A T cell-dependent experimental liver injury in mice inducible by concanavalin A. , 1992, The Journal of clinical investigation.

[39]  L. Moldawer,et al.  Tumor necrosis factor soluble receptors circulate during experimental and clinical inflammation and can protect against excessive tumor necrosis factor alpha in vitro and in vivo. , 1992, Proceedings of the National Academy of Sciences of the United States of America.

[40]  P. Chomczyński,et al.  Single-step method of RNA isolation by acid guanidinium thiocyanate-phenol-chloroform extraction. , 1987, Analytical biochemistry.

[41]  M. Leist,et al.  The 55-kD Tumor Necrosis Factor Receptor and CD95 Independently Signal Murine Hepatocyte Apoptosis and Subsequent Liver Failure , 1996, Molecular medicine.

[42]  S. Nagata.  Apoptosis mediated by the Fas system. , 1996, Progress in molecular and subcellular biology.