Interaction of fluorescein derivatives with glibenclamide binding sites in rat brain
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O. Feron | J. Maloteaux | J. Octave | S. Holemans | O. Féron
[1] M. Lazdunski,et al. K+ channel openers prevent global ischemia-induced expression of c-fos, c-jun, heat shock protein, and amyloid beta-protein precursor genes and neuronal death in rat hippocampus. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[2] M. Roisin,et al. Effect of potassium channel modulators on the release of glutamate induced by ischaemic-like conditions in rat hippocampal slices , 1993, Neuroscience Letters.
[3] Y. Ben-Ari,et al. Nucleotides modulate the low affinity binding sites for [3H]glibenclamide in the rat brain. , 1993, The Journal of pharmacology and experimental therapeutics.
[4] F. Ashcroft,et al. The sulfonylurea receptor. , 1992, Biochimica et biophysica acta.
[5] M. Lazdunski,et al. ATP/ADP binding sites are present in the sulfonylurea binding protein associated with brain ATP-sensitive K+ channels. , 1992, Biochemistry.
[6] M. Lazdunski,et al. Activation and inhibition of ATP-sensitive K+ channels by fluorescein derivatives. , 1992, The Journal of biological chemistry.
[7] J. Mark Treherne,et al. The regional distribution of sulphonylurea binding sites in rat brain , 1991, Neuroscience.
[8] D. Triggle,et al. Characterization of binding of the ATP-sensitive potassium channel ligand, [3H]glyburide, to neuronal and muscle preparations. , 1991, The Journal of pharmacology and experimental therapeutics.
[9] Y. Ben-Ari,et al. Two binding sites for [3H]glibenclamide in the rat brain , 1991, Brain Research.
[10] M. Lazdunski,et al. Specific hippocampal lesions indicate the presence of sulfonylurea binding sites associated to ATP-sensitive K+ channels both post-synaptically and on mossy fibers , 1991, Brain Research.
[11] Y. Ben-Ari,et al. Activators of ATP-sensitive K+ channels reduce anoxic depolarization in CA3 hippocampal neurons , 1990, Neuroscience.
[12] N. S. Cook,et al. Potassium Channels — Structure, Classification, Function and Therapeutic Potential Nigel S. Cook (ed.) Ellis Horwood, 1990. £59.95 (412 pages) ISBN 0 7458 0624 4 , 1990, Trends in Neurosciences.
[13] M. Lazdunski,et al. Brain ischemia alters the density of binding sites for glibenclamide, a specific blocker of ATP-sensitive K+ channels , 1990, Brain Research.
[14] M. Lazdunski,et al. Glucose, sulfonylureas, and neurotransmitter release: role of ATP-sensitive K+ channels. , 1990, Science.
[15] Y. Ben-Ari,et al. Galanin and Glibenclamide Modulate the Anoxic Release of Glutamate in Rat CA3 Hippocampal Neurons , 1990, The European journal of neuroscience.
[16] M. Lazdunski,et al. Characterization, purification, and affinity labeling of the brain [3H]glibenclamide-binding protein, a putative neuronal ATP-regulated K+ channel. , 1988, Proceedings of the National Academy of Sciences of the United States of America.
[17] F. Ashcroft. Adenosine 5'-triphosphate-sensitive potassium channels. , 1988, Annual review of neuroscience.
[18] M. Lazdunski,et al. The receptor for antidiabetic sulfonylureas controls the activity of the ATP-modulated K+ channel in insulin-secreting cells. , 1987, The Journal of biological chemistry.
[19] D. Hawke,et al. The amino acid sequence of a fluorescein-labeled peptide from the active site of (Na,K)-ATPase. , 1984, The Journal of biological chemistry.
[20] O. H. Lowry,et al. Protein measurement with the Folin phenol reagent. , 1951, The Journal of biological chemistry.