Transcriptomic analysis reveals global and temporal transcription changes during Candida glabrata adaptation to an oxidative environment.

[1]  Kundan Kumar,et al.  Candida glabrata: A Lot More Than Meets the Eye , 2019, Microorganisms.

[2]  R. A. Crawford,et al.  Translational regulation in response to stress in Saccharomyces cerevisiae , 2018, Yeast.

[3]  K. H. Wong,et al.  RNA polymerase II ChIP-seq—a powerful and highly affordable method for studying fungal genomics and physiology , 2019, Biophysical Reviews.

[4]  Katy C. Kao,et al.  Identifying novel genetic determinants for oxidative stress tolerance in Candida glabrata via adaptive laboratory evolution , 2018, Yeast.

[5]  U. Güldener,et al.  Comparative genomic and transcriptomic analyses unveil novel features of azole resistance and adaptation to the human host in Candida glabrata , 2018, FEMS yeast research.

[6]  J. Nielsen,et al.  Elimination of the last reactions in ergosterol biosynthesis alters the resistance of Saccharomyces cerevisiae to multiple stresses , 2017, FEMS yeast research.

[7]  Zhìhóng Hú,et al.  Recent Advances in Ergosterol Biosynthesis and Regulation Mechanisms in Saccharomyces cerevisiae , 2017, Indian Journal of Microbiology.

[8]  P. D. Rogers,et al.  Azole Resistance in Candida glabrata , 2016, Current Infectious Disease Reports.

[9]  Lan Yan,et al.  The synthesis, regulation, and functions of sterols in Candida albicans: Well-known but still lots to learn , 2016, Virulence.

[10]  H. Sychrová,et al.  Changes in the Sterol Composition of the Plasma Membrane Affect Membrane Potential, Salt Tolerance and the Activity of Multidrug Resistance Pumps in Saccharomyces cerevisiae , 2015, PloS one.

[11]  B. Hube,et al.  Intracellular survival of Candida glabrata in macrophages: immune evasion and persistence. , 2015, FEMS yeast research.

[12]  A. De Las Peñas,et al.  The superoxide dismutases of Candida glabrata protect against oxidative damage and are required for lysine biosynthesis, DNA integrity and chronological life survival. , 2015, Microbiology.

[13]  D. Horn,et al.  Epidemiology and Outcomes of Invasive Candidiasis Due to Non-albicans Species of Candida in 2,496 Patients: Data from the Prospective Antifungal Therapy (PATH) Registry 2004–2008 , 2014, PloS one.

[14]  A. De Las Peñas,et al.  The oxidative stress response of the opportunistic fungal pathogen Candida glabrata. , 2014, Revista iberoamericana de micologia.

[15]  M. Erard,et al.  ROS production in phagocytes: why, when, and where? , 2013, Journal of leukocyte biology.

[16]  Manolis Kellis,et al.  Arboretum: Reconstruction and analysis of the evolutionary history of condition-specific transcriptional modules , 2013, Genome research.

[17]  A. De Las Peñas,et al.  Role of glutathione in the oxidative stress response in the fungal pathogen Candida glabrata , 2013, Current Genetics.

[18]  J. Lodge,et al.  Global Transcriptome Profile of Cryptococcus neoformans during Exposure to Hydrogen Peroxide Induced Oxidative Stress , 2013, PloS one.

[19]  Sébastien Dupont,et al.  ERGOSTEROL BIOSYNTHESIS: A FUNGAL PATHWAY FOR LIFE ON LAND? , 2012, Evolution; international journal of organic evolution.

[20]  S. Balusu,et al.  Functional Genomic Analysis of Candida glabrata-Macrophage Interaction: Role of Chromatin Remodeling in Virulence , 2012, PLoS pathogens.

[21]  Amparo Pascual-Ahuir,et al.  Repression of ergosterol biosynthesis is essential for stress resistance and is mediated by the Hog1 MAP kinase and the Mot3 and Rox1 transcription factors , 2011, Molecular microbiology.

[22]  T. Gabaldón,et al.  Regulation of Candida glabrata oxidative stress resistance is adapted to host environment , 2011, FEBS letters.

[23]  T. Gabaldón,et al.  From Saccharomyces cerevisiae to Candida glabrata in a few easy steps: important adaptations for an opportunistic pathogen , 2011, FEMS microbiology letters.

[24]  Ramin Homayouni,et al.  Genomewide Expression Profile Analysis of the Candida glabrata Pdr1 Regulon , 2010, Eukaryotic Cell.

[25]  M. Bushell,et al.  Translational regulation of gene expression during conditions of cell stress. , 2010, Molecular cell.

[26]  T. Myers,et al.  Microarray and Molecular Analyses of the Azole Resistance Mechanism in Candida glabrata Oropharyngeal Isolates , 2010, Antimicrobial Agents and Chemotherapy.

[27]  K. Yanagihara,et al.  Skn7p Is Involved in Oxidative Stress Response and Virulence of Candida glabrata , 2010, Mycopathologia.

[28]  G. Fadda,et al.  Gain of Function Mutations in CgPDR1 of Candida glabrata Not Only Mediate Antifungal Resistance but Also Enhance Virulence , 2009, PLoS pathogens.

[29]  D. Krysan,et al.  Live Candida albicans Suppresses Production of Reactive Oxygen Species in Phagocytes , 2008, Infection and Immunity.

[30]  K. Kuchler,et al.  Candida glabrata environmental stress response involves Saccharomyces cerevisiae Msn2/4 orthologous transcription factors , 2008, Molecular microbiology.

[31]  A. De Las Peñas,et al.  Is Controlled by the Transcription Factors Mediated by a Single Catalase, Cta1p, and Is Candida Glabrata Fungal Pathogen High Resistance to Oxidative Stress in The , 2008 .

[32]  C. Grant,et al.  Metabolic reconfiguration is a regulated response to oxidative stress , 2008, Journal of biology.

[33]  Axel Kowald,et al.  Dynamic rerouting of the carbohydrate flux is key to counteracting oxidative stress , 2007, Journal of biology.

[34]  B. Cormack,et al.  A family of glycosylphosphatidylinositol-linked aspartyl proteases is required for virulence of Candida glabrata , 2007, Proceedings of the National Academy of Sciences.

[35]  F. Robert,et al.  Oxidative Stress-Activated Zinc Cluster Protein Stb5 Has Dual Activator/Repressor Functions Required for Pentose Phosphate Pathway Regulation and NADPH Production , 2006, Molecular and Cellular Biology.

[36]  Peter G. Pappas,et al.  Invasive Fungal Pathogens: Current Epidemiological Trends , 2006 .

[37]  R. Homayouni,et al.  Pdr1 regulates multidrug resistance in Candida glabrata: gene disruption and genome‐wide expression studies , 2006, Molecular microbiology.

[38]  J. Bennett,et al.  Candida glabrata PDR1, a Transcriptional Regulator of a Pleiotropic Drug Resistance Network, Mediates Azole Resistance in Clinical Isolates and Petite Mutants , 2006, Antimicrobial Agents and Chemotherapy.

[39]  G. Fadda,et al.  Mechanisms of Azole Resistance in Clinical Isolates of Candida glabrata Collected during a Hospital Survey of Antifungal Resistance , 2005, Antimicrobial Agents and Chemotherapy.

[40]  T. Edlind,et al.  Azole Resistance in Candida glabrata: Coordinate Upregulation of Multidrug Transporters and Evidence for a Pdr1-Like Transcription Factor , 2004, Antimicrobial Agents and Chemotherapy.

[41]  V. Higgins,et al.  Yeast Genome-Wide Expression Analysis Identifies a Strong Ergosterol and Oxidative Stress Response during the Initial Stages of an Industrial Lager Fermentation , 2003, Applied and Environmental Microbiology.

[42]  A. Kastaniotis,et al.  The biochemistry of peroxisomal β-oxidation in the yeast Saccharomyces cerevisiae , 2003 .

[43]  R. Prasad,et al.  Drug Susceptibilities of Yeast Cells Are Affected by Membrane Lipid Composition , 2002, Antimicrobial Agents and Chemotherapy.

[44]  D. Sanglard,et al.  The ATP Binding Cassette Transporter GeneCgCDR1 from Candida glabrata Is Involved in the Resistance of Clinical Isolates to Azole Antifungal Agents , 1999, Antimicrobial Agents and Chemotherapy.